muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A1

disease
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Also known as MDDGA1muscle-eye-brain-POMT1 relatedmuscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies) type A, 1muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A, 1

Summary

muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A1 (MONDO:0009364) is a disease caused by POMT1 (GenCC Definitive), with 7 cohort genes. The dominant Reactome pathway is Matriglycan biosynthesis on DAG1 (5 cohort genes).

At a glance

  • Causal gene: POMT1 (GenCC Definitive)
  • Cohort genes: 7
  • ClinVar variants: 306

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namemuscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A1
Mondo IDMONDO:0009364
OMIM236670
DOIDDOID:0111237
NCITC128118
UMLSC4284790
MedGen924974
GARD0024665
Is cancer (heuristic)no

Also known as: MDDGA1 · muscle-eye-brain-POMT1 related · muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies) type A, 1 · muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A, 1

Data availability: 306 ClinVar variants · 4 GenCC gene-disease records · 1 cell line.

Disease family

Classification path: disease › human disease › disease by body system or component › nervous system disordercongenital nervous system disordercongenital muscular dystrophymuscular dystrophy-dystroglycanopathymuscular dystrophy-dystroglycanopathy, type Amuscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A1

Related subtypes (13): muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A3, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A, 4, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A2, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A5, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A6, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A, 7, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 8, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 10, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 11, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 12, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A13, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A14, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A9

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

306 retrieved; paginated sample, class counts are floors:

101 uncertain significance, 55 pathogenic/likely pathogenic, 48 conflicting classifications of pathogenicity, 41 likely pathogenic, 31 pathogenic, 12 benign, 11 benign/likely benign, 7 likely benign

ClinVarVariant (HGVS)GeneClassificationReview
183334NM_006876.3(B4GAT1):c.821_822insTT (p.Glu274fs)B4GAT1Pathogenic/Likely pathogenicno assertion criteria provided
197347NM_024301.5(FKRP):c.947C>G (p.Pro316Arg)FKRPPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
4220NM_024301.5(FKRP):c.1343C>T (p.Pro448Leu)FKRPPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
4221NM_024301.5(FKRP):c.826C>A (p.Leu276Ile)FKRPPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
4232NM_024301.5(FKRP):c.899T>C (p.Val300Ala)FKRPPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
4235NM_024301.5(FKRP):c.1387A>G (p.Asn463Asp)FKRPPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
550361NM_024301.5(FKRP):c.928G>T (p.Glu310Ter)FKRPPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
551007NM_024301.5(FKRP):c.266C>T (p.Pro89Leu)FKRPPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
554104NM_024301.5(FKRP):c.778G>T (p.Glu260Ter)FKRPPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
654883NM_024301.5(FKRP):c.1296G>A (p.Trp432Ter)FKRPPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
664273NM_024301.5(FKRP):c.948del (p.Cys317fs)FKRPPathogeniccriteria provided, multiple submitters, no conflicts
167069NM_001079802.2(FKTN):c.411C>A (p.Cys137Ter)FKTNPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
225359NM_001079802.2(FKTN):c.607C>T (p.Arg203Ter)FKTNPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
281839NM_001079802.2(FKTN):c.330dup (p.Thr111fs)FKTNPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
283622NM_001079802.2(FKTN):c.456_457del (p.Ser154fs)FKTNPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
3203NM_001079802.2(FKTN):c.1167dup (p.Phe390fs)FKTNPathogeniccriteria provided, multiple submitters, no conflicts
3204NM_001079802.2(FKTN):c.527T>C (p.Phe176Ser)FKTNPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
496331NM_001079802.2(FKTN):c.648-1243G>TFKTNPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1066221NM_001077365.2(POMT1):c.1176-2A>GPOMT1Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1120088NM_001077365.2(POMT1):c.1255C>T (p.Gln419Ter)POMT1Pathogeniccriteria provided, multiple submitters, no conflicts
1192212NM_001077365.2(POMT1):c.1272+1G>APOMT1Pathogeniccriteria provided, multiple submitters, no conflicts
1252077NM_001077365.2(POMT1):c.1175+3delPOMT1Pathogenicno assertion criteria provided
1343631NM_001077365.2(POMT1):c.2040_2050del (p.Val681fs)POMT1Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1360015NM_001077365.2(POMT1):c.270_280delAATTGGAGCAG (p.Gly92fs)POMT1Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1399708NM_001077365.2(POMT1):c.97C>T (p.Arg33Ter)POMT1Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1453137NM_001077365.2(POMT1):c.1364del (p.Lys455fs)POMT1Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1458255NM_001077365.2(POMT1):c.130G>A (p.Glu44Lys)POMT1Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1497287NM_001077365.2(POMT1):c.313C>T (p.Arg105Cys)POMT1Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1686087NM_001077365.2(POMT1):c.986+1G>APOMT1Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1696829NM_001077365.2(POMT1):c.1417G>C (p.Gly473Arg)POMT1Pathogeniccriteria provided, single submitter

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 12 · Orphanet: 31 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

GenCC gene–disease validity (cohort genes)

the Disease column is the GenCC-asserted condition — a cohort gene’s strongest validity may be for a related predisposition syndrome.

GeneClassificationInheritanceDiseaseRecords
POMT1DefinitiveAutosomal recessivemuscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A112

Orphanet rare-disease linkage (cohort genes)

GeneOrphanet IDRare disease
POMT1Orphanet:370959Congenital muscular dystrophy with cerebellar involvement
POMT1Orphanet:370968Congenital muscular dystrophy with intellectual disability
POMT1Orphanet:370980Congenital muscular dystrophy without intellectual disability
POMT1Orphanet:588Muscle-eye-brain disease
POMT1Orphanet:86812POMT1-related limb-girdle muscular dystrophy R11
POMT1Orphanet:899Walker-Warburg syndrome
B4GAT1Orphanet:899Walker-Warburg syndrome
FKRPOrphanet:34515FKRP-related limb-girdle muscular dystrophy R9
FKRPOrphanet:370959Congenital muscular dystrophy with cerebellar involvement
FKRPOrphanet:370968Congenital muscular dystrophy with intellectual disability
FKRPOrphanet:370980Congenital muscular dystrophy without intellectual disability
FKRPOrphanet:588Muscle-eye-brain disease
FKRPOrphanet:899Walker-Warburg syndrome
POMT2Orphanet:206559POMT2-related limb-girdle muscular dystrophy R14
POMT2Orphanet:370959Congenital muscular dystrophy with cerebellar involvement
POMT2Orphanet:370968Congenital muscular dystrophy with intellectual disability
POMT2Orphanet:588Muscle-eye-brain disease
POMT2Orphanet:899Walker-Warburg syndrome
DAG1Orphanet:206599Isolated asymptomatic elevation of creatine phosphokinase
DAG1Orphanet:280333Alpha-dystroglycan-related limb-girdle muscular dystrophy R16
DAG1Orphanet:370997Muscle-eye-brain disease with bilateral multicystic leucodystrophy
DAG1Orphanet:899Walker-Warburg syndrome
FKTNOrphanet:154Familial isolated dilated cardiomyopathy
FKTNOrphanet:206554Fukutin-related limb-girdle muscular dystrophy R13
FKTNOrphanet:272Congenital muscular dystrophy, Fukuyama type
FKTNOrphanet:370980Congenital muscular dystrophy without intellectual disability
FKTNOrphanet:588Muscle-eye-brain disease
FKTNOrphanet:899Walker-Warburg syndrome
LARGE1Orphanet:370968Congenital muscular dystrophy with intellectual disability
LARGE1Orphanet:588Muscle-eye-brain disease
LARGE1Orphanet:899Walker-Warburg syndrome

Cohort genes → proteins

7 cohort genes, 7 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence7

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
POMT1HGNC:9202ENSG00000130714Q9Y6A1Protein O-mannosyl-transferase 1gencc,clinvar
B4GAT1HGNC:15685ENSG00000174684O43505Beta-1,4-glucuronyltransferase 1clinvar
FKRPHGNC:17997ENSG00000181027Q9H9S5Ribitol 5-phosphate transferase FKRPclinvar
POMT2HGNC:19743ENSG00000009830Q9UKY4Protein O-mannosyl-transferase 2clinvar
DAG1HGNC:2666ENSG00000173402Q14118Dystroglycan 1clinvar
FKTNHGNC:3622ENSG00000106692O75072Ribitol-5-phosphate transferase FKTNclinvar
LARGE1HGNC:6511ENSG00000133424O95461Xylosyl- and glucuronyltransferase LARGE1clinvar

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
POMT1Protein O-mannosyl-transferase 1Transfers mannosyl residues to the hydroxyl group of serine or threonine residues.
B4GAT1Beta-1,4-glucuronyltransferase 1Beta-1,4-glucuronyltransferase involved in O-mannosylation of alpha-dystroglycan (DAG1).
FKRPRibitol 5-phosphate transferase FKRPCatalyzes the transfer of a ribitol 5-phosphate from CDP-L-ribitol to the ribitol 5-phosphate previously attached by FKTN/fukutin to the phosphorylated O-mannosyl trisaccharide (N-acetylgalactosamine-beta-3-N-acetylglucosamine-beta-4-(phos…
POMT2Protein O-mannosyl-transferase 2Transfers mannosyl residues to the hydroxyl group of serine or threonine residues.
DAG1Dystroglycan 1The dystroglycan complex is involved in a number of signaling events and processes including laminin deposition and extracellular matrix assembly, acetylcholine receptor clustering, sarcolemmal stability, cell survival, peripheral nerve my…
FKTNRibitol-5-phosphate transferase FKTNCatalyzes the transfer of a ribitol-phosphate from CDP-ribitol to the distal N-acetylgalactosamine of the phosphorylated O-mannosyl trisaccharide (N-acetylgalactosamine-beta-3-N-acetylglucosamine-beta-4-(phosphate-6-)mannose), a carbohydra…
LARGE1Xylosyl- and glucuronyltransferase LARGE1Bifunctional glycosyltransferase with both alpha-1,3-xylosyltransferase and beta-1,3-glucuronyltransferase activities involved in the maturation of alpha-dystroglycan (DAG1) by glycosylation leading to DAG1 binding to laminin G-like domain…

Protein-family classification

Druggable: 5 · Difficult: 0 · Unknown: 2 · Druggable fraction: 0.71

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Enzyme (other)46.8×0.004
Antibody/Immunoglobulin14.2×0.325
Other/Unknown20.5×0.968

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
POMT1Enzyme (other)yes2.4.1.109ArnT-like_N, MIR_motif, PMT-like
B4GAT1Enzyme (other)yes2.4.1.149B4GAT1
FKRPOther/UnknownnoLicD/FKTN/FKRP_NTP_transf, LicD_transferase, FKRP_N
POMT2Enzyme (other)yes2.4.1.109ArnT-like_N, MIR_motif, PMT-like
DAG1Antibody/ImmunoglobulinyesCadg, DAG1_C, Ig-like_fold
FKTNOther/UnknownnoLicD/FKTN/FKRP_NTP_transf, FKTN/MNN-like, FKTN_N
LARGE1Enzyme (other)yes2.4.1.B80Glyco_trans_8, Nucleotide-diphossugar_trans, Xyl/GlcA_transferase

Expression context

Cohort genes with no expression data: 0.

5 cohort genes are a single-cell marker in ≥1 SCXA experiment.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)7
unknown0

Top tissues across cohort

TissueCohort genes
cerebellar cortex1
cerebellar hemisphere1
right hemisphere of cerebellum1
endothelial cell1
middle temporal gyrus1
pons1
cardiac muscle of right atrium1
hindlimb stylopod muscle1
left ventricle myocardium1
left testis1
right testis1
testis1
dorsal root ganglion1
olfactory bulb1
trigeminal ganglion1
adrenal tissue1
calcaneal tendon1
germinal epithelium of ovary1
apex of heart1
cardiac ventricle1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
POMT1264ubiquitousmarkerright hemisphere of cerebellum, cerebellar hemisphere, cerebellar cortex
B4GAT1285ubiquitousmarkerendothelial cell, middle temporal gyrus, pons
FKRP230ubiquitousmarkerleft ventricle myocardium, cardiac muscle of right atrium, hindlimb stylopod muscle
POMT2222ubiquitousyesright testis, left testis, testis
DAG1299ubiquitousmarkerolfactory bulb, trigeminal ganglion, dorsal root ganglion
FKTN277ubiquitousyescalcaneal tendon, adrenal tissue, germinal epithelium of ovary
LARGE1233tissue_specificmarkerheart left ventricle, cardiac ventricle, apex of heart

Protein interactions among cohort

Intra-cohort edges: 21.

Hub genes (top 10 by interactor count)

SymbolInteractor count
DAG12,301
POMT11,475
FKRP1,436
POMT21,284
FKTN1,226
B4GAT1823
LARGE1551

Intra-cohort edges

ABSources
B4GAT1DAG1string_interaction
B4GAT1FKRPbiogrid_interaction, intact, string_interaction
B4GAT1FKTNbiogrid_interaction, intact, string_interaction
B4GAT1LARGE1intact
B4GAT1POMT1intact, string_interaction
B4GAT1POMT2string_interaction
DAG1FKRPstring_interaction
DAG1FKTNstring_interaction
DAG1LARGE1string_interaction
DAG1POMT1string_interaction
DAG1POMT2string_interaction
FKRPFKTNintact, string_interaction
FKRPLARGE1string_interaction
FKRPPOMT1string_interaction
FKRPPOMT2string_interaction
FKTNLARGE1string_interaction
FKTNPOMT1string_interaction
FKTNPOMT2string_interaction
LARGE1POMT1string_interaction
LARGE1POMT2string_interaction
POMT1POMT2intact, string_interaction

Structural data

PDB: 3 · AlphaFold-only: 4 · No structure: 0

Cohort genes with PDB structures (top 30)

SymbolUniProtPDB entries
FKRPQ9H9S58
DAG1Q141188
LARGE1O954614

AlphaFold-only cohort genes (top 30 by pLDDT)

SymbolUniProtpLDDT
FKTNO7507292.48
B4GAT1O4350588.46
POMT1Q9Y6A188.09
POMT2Q9UKY487.96

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 26. Enrichment computed across 7 evidence-associated genes (7 with Reactome annotation).

Pathways by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 7 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

PathwayCohort genesFoldFDRSample cohort genes
Matriglycan biosynthesis on DAG15582.6×7e-13B4GAT1, FKRP, DAG1, FKTN, LARGE1
Defective POMT2 causes MDDGA2, MDDGB2 and MDDGC231631.4×1e-09POMT1, POMT2, DAG1
Defective POMT1 causes MDDGA1, MDDGB1 and MDDGC131631.4×1e-09POMT1, POMT2, DAG1
DAG1 core M1 glycosylations31223.6×4e-09POMT1, POMT2, DAG1
DAG1 core M2 glycosylations3978.9×7e-09POMT1, POMT2, DAG1
DAG1 core M3 glycosylations3815.7×1e-08POMT1, POMT2, DAG1
Defective LARGE causes MDDGA6 and MDDGB621087.6×4e-06B4GAT1, LARGE1
Regulation of CDH1 posttranslational processing and trafficking to plasma membrane296.0×6e-04POMT1, POMT2
Diseases associated with O-glycosylation of proteins261.6×0.001B4GAT1, LARGE1
O-linked glycosylation241.3×0.003B4GAT1, LARGE1
Defective POMGNT1 causes MDDGA3, MDDGB3 and MDDGC31815.7×0.003DAG1
Diseases of glycosylation237.5×0.003B4GAT1, LARGE1
Diseases of metabolism223.0×0.006B4GAT1, LARGE1
Keratan sulfate/keratin metabolism170.9×0.026B4GAT1
Keratan sulfate biosynthesis154.4×0.031B4GAT1
EGR2 and SOX10-mediated initiation of Schwann cell myelination152.6×0.031DAG1
Formation of the dystrophin-glycoprotein complex (DGC)144.1×0.034DAG1
Glycosaminoglycan metabolism131.4×0.045B4GAT1
Non-integrin membrane-ECM interactions122.1×0.059DAG1
ECM proteoglycans121.5×0.059DAG1
Post-translational protein modification25.5×0.059B4GAT1, LARGE1
Metabolism of carbohydrates and carbohydrate derivatives117.2×0.067B4GAT1
Regulation of expression of SLITs and ROBOs19.9×0.105DAG1
Disease23.7×0.105B4GAT1, LARGE1
Metabolism of proteins23.5×0.109B4GAT1, LARGE1
Metabolism11.7×0.468B4GAT1

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 7 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
protein O-linked glycosylation via mannose6802.5×5e-16POMT1, B4GAT1, FKRP, POMT2, FKTN, LARGE1
basement membrane organization5364.8×3e-11FKRP, POMT2, DAG1, FKTN, LARGE1
skeletal muscle fiber differentiation3722.2×2e-07FKRP, FKTN, LARGE1
skeletal muscle tissue regeneration3380.1×1e-06FKRP, DAG1, LARGE1
connective tissue development21203.7×2e-05FKRP, LARGE1
localization of cell2802.5×5e-05FKRP, LARGE1
reactive gliosis2687.8×5e-05POMT2, LARGE1
protein O-linked glycosylation396.3×5e-05POMT1, FKTN, LARGE1
nerve development2267.5×3e-04DAG1, LARGE1
positive regulation of Rac protein signal transduction2185.2×6e-04DAG1, LARGE1
dentate gyrus development2178.3×6e-04POMT2, LARGE1
heart morphogenesis2107.0×0.002FKRP, DAG1
muscle attachment12407.4×0.003DAG1
pentitol metabolic process12407.4×0.003FKRP
nerve maturation12407.4×0.003DAG1
post-embryonic hindlimb morphogenesis12407.4×0.003LARGE1
filtration diaphragm assembly12407.4×0.003FKRP
calcium-dependent cell-matrix adhesion11203.7×0.005DAG1
pentose metabolic process11203.7×0.005FKRP
principal sensory nucleus of trigeminal nerve development1802.5×0.007LARGE1
synaptic assembly at neuromuscular junction1802.5×0.007LARGE1
retrograde trans-synaptic signaling by trans-synaptic protein complex1802.5×0.007DAG1
neuron migration238.2×0.007FKRP, LARGE1
extracellular matrix organization234.9×0.007POMT1, DAG1
creatine metabolic process1601.9×0.008FKRP
negative regulation of muscle cell apoptotic process1601.9×0.008LARGE1
oxygen metabolic process1601.9×0.008FKRP
maintenance of protein localization in endoplasmic reticulum1481.5×0.009FKRP
walking behavior1401.2×0.010LARGE1
axon guidance225.9×0.010B4GAT1, DAG1

Therapeutics

Drug target analysis

Approved (phase 4): 0 · Phase ≥3: 0 · Phased (≥1): 0 · Undrugged: 7

Druggability breadth: 2 of 7 evidence-associated genes (29%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Top cohort targets by molecule count

SymbolMoleculesMax phase
POMT100
B4GAT100
FKRP00
POMT200
DAG100
FKTN00
LARGE100

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 4.

Cohort genes with ChEMBL bioactivity (full, sorted by assay count)

SymbolAssaysType breakdown
DAG14Binding:4
LARGE12Functional:2

Cohort enzymes (BRENDA EC)

SymbolEC numbersNames
POMT12.4.1.109dolichyl-phosphate-mannose-protein mannosyltransferase
B4GAT12.4.1.149N-acetyllactosaminide beta-1,3-N-acetylglucosaminyltransferase
POMT22.4.1.109dolichyl-phosphate-mannose-protein mannosyltransferase
LARGE12.4.1.B80, 2.4.2.B18,

Pharmacogenomics

Cohort genes with a PharmGKB record: 7; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

0 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)0
BPhased (≥1) drug, not yet approved0
CDruggable family + PDB, no drug2DAG1, LARGE1
DDruggable family + AlphaFold only, no drug3POMT1, B4GAT1, POMT2
EDifficult family or no structure, no drug2FKRP, FKTN

Undrugged target profiles

7 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

SymbolChEMBL assaysDrugged partners (top 3)
POMT10
B4GAT10
FKRP0
POMT20
DAG14
FKTN0
LARGE12

Clinical trials & evidence

Clinical trials

Clinical trials: 0.