Necrotizing ulcerative gingivitis

disease
On this page

Also known as acute membranous gingivitisacute necrotising ulcerative gingivitis [ambiguous]acute necrotising ulcerative gingivostomatitis [ambiguous]ANUGFusospirillary gingivitisFusospirillosisphagedenic gingivitisVincent anginaVincent's anginaVincent's gingivitisVincent's infectionVincent's stomatitis

Summary

Necrotizing ulcerative gingivitis (MONDO:0006865) is a disease. A subtype of gingivitis — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namenecrotizing ulcerative gingivitis
Mondo IDMONDO:0006865
EFOEFO:1001058
MeSHD005892
DOIDDOID:13924
NCITC34637
SNOMED CT186963008
UMLSC0017575
MedGen42219
Is cancer (heuristic)no

Also known as: acute membranous gingivitis · acute necrotising ulcerative gingivitis [ambiguous] · acute necrotising ulcerative gingivostomatitis [ambiguous] · ANUG · Fusospirillary gingivitis · Fusospirillosis · phagedenic gingivitis · Vincent angina · Vincent’s angina · Vincent’s gingivitis · Vincent’s infection · Vincent’s stomatitis

Disease family

This is a subtype of gingivitis. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › musculoskeletal system disorderskeletal system disorderperiodontal disordergingival disordergingivitisnecrotizing ulcerative gingivitis

Related subtypes (1): chronic gingivitis

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.