neuronopathy, distal hereditary motor, type 2D

disease
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Also known as FBXO38 neuronopathy, distal hereditary motorHMN2Dneuronopathy, distal hereditary motor caused by mutation in FBXO38neuronopathy, distal hereditary motor, type IID

Summary

neuronopathy, distal hereditary motor, type 2D (MONDO:0014259) is a disease caused by FBXO38 (GenCC Strong), with 1 cohort gene.

At a glance

  • Causal gene: FBXO38 (GenCC Strong)
  • Cohort genes: 1
  • ClinVar variants: 34

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical nameneuronopathy, distal hereditary motor, type 2D
Mondo IDMONDO:0014259
OMIM615575
DOIDDOID:0111210
UMLSC3888271
MedGen854832
GARD0018265
Is cancer (heuristic)no

Also known as: FBXO38 neuronopathy, distal hereditary motor · HMN2D · neuronopathy, distal hereditary motor caused by mutation in FBXO38 · neuronopathy, distal hereditary motor, type IID

Data availability: 34 ClinVar variants · 3 GenCC gene-disease records.

Disease family

Classification path: disease › human disease › disease by etiologic mechanism › disease of genetic or genomic mechanism › hereditary disease › autosomal genetic disease › autosomal dominant disease › neuronopathy, distal hereditary motor, autosomal dominantdistal hereditary motor neuropathy type 2neuronopathy, distal hereditary motor, type 2D

Related subtypes (3): neuronopathy, distal hereditary motor, type 2A, neuronopathy, distal hereditary motor, type 2B, neuronopathy, distal hereditary motor, type 2C

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

34 retrieved; paginated sample, class counts are floors:

19 uncertain significance, 6 benign, 5 conflicting classifications of pathogenicity, 2 pathogenic, 2 likely benign

ClinVarVariant (HGVS)GeneClassificationReview
2431046NM_205836.3(FBXO38):c.2215C>A (p.Pro739Thr)FBXO38Pathogenicno assertion criteria provided
91854NM_205836.3(FBXO38):c.616T>C (p.Cys206Arg)FBXO38Pathogeniccriteria provided, single submitter
1045555NM_205836.3(FBXO38):c.91A>C (p.Asn31His)FBXO38Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
541015NM_205836.3(FBXO38):c.11G>A (p.Arg4Gln)FBXO38Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
656377NM_205836.3(FBXO38):c.1577G>A (p.Arg526Gln)FBXO38Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
665522NM_205836.3(FBXO38):c.1117G>A (p.Val373Met)FBXO38Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
836217NM_205836.3(FBXO38):c.2683A>G (p.Met895Val)FBXO38Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1008505NM_205836.3(FBXO38):c.2072T>C (p.Ile691Thr)FBXO38Uncertain significancecriteria provided, multiple submitters, no conflicts
1028903NM_205836.3(FBXO38):c.155G>A (p.Cys52Tyr)FBXO38Uncertain significancecriteria provided, single submitter
1285478NM_205836.3(FBXO38):c.1313A>G (p.His438Arg)FBXO38Uncertain significancecriteria provided, single submitter
1333471NM_205836.3(FBXO38):c.1570G>A (p.Asp524Asn)FBXO38Uncertain significancecriteria provided, single submitter
1709150NM_205836.3(FBXO38):c.801A>T (p.Leu267Phe)FBXO38Uncertain significancecriteria provided, single submitter
2441399NM_205836.3(FBXO38):c.1208A>G (p.Lys403Arg)FBXO38Uncertain significancecriteria provided, multiple submitters, no conflicts
2689060NM_205836.3(FBXO38):c.1717G>T (p.Asp573Tyr)FBXO38Uncertain significancecriteria provided, single submitter
4078637NM_205836.3(FBXO38):c.1991C>A (p.Pro664His)FBXO38Uncertain significancecriteria provided, single submitter
4277987NM_205836.3(FBXO38):c.962+1G>AFBXO38Uncertain significancecriteria provided, single submitter
541008NM_205836.3(FBXO38):c.1154G>T (p.Gly385Val)FBXO38Uncertain significancecriteria provided, single submitter
574620NM_205836.3(FBXO38):c.1858C>T (p.Arg620Trp)FBXO38Uncertain significancecriteria provided, single submitter
638363NM_205836.3(FBXO38):c.2075C>T (p.Pro692Leu)FBXO38Uncertain significancecriteria provided, single submitter
640185NM_205836.3(FBXO38):c.286A>G (p.Thr96Ala)FBXO38Uncertain significancecriteria provided, multiple submitters, no conflicts
665063NM_205836.3(FBXO38):c.679A>C (p.Lys227Gln)FBXO38Uncertain significancecriteria provided, single submitter
828150NM_205836.3(FBXO38):c.1618+1G>TFBXO38Uncertain significanceno assertion criteria provided
831074NC_000005.10:g.(?148394767)(148406404_?)dupFBXO38Uncertain significancecriteria provided, single submitter
931134NM_205836.3(FBXO38):c.3244C>G (p.Pro1082Ala)FBXO38Uncertain significancecriteria provided, single submitter
950489NM_205836.3(FBXO38):c.3277G>A (p.Val1093Ile)FBXO38Uncertain significancecriteria provided, single submitter
964250NM_205836.3(FBXO38):c.1796A>C (p.Glu599Ala)FBXO38Uncertain significancecriteria provided, multiple submitters, no conflicts
1169149NM_205836.3(FBXO38):c.1619-4G>TFBXO38Benigncriteria provided, multiple submitters, no conflicts
1169150NM_205836.3(FBXO38):c.1774T>C (p.Ser592Pro)FBXO38Benigncriteria provided, multiple submitters, no conflicts
1235574NM_205836.3(FBXO38):c.962+18A>GFBXO38Benigncriteria provided, multiple submitters, no conflicts
1249294NM_205836.3(FBXO38):c.3170+11T>CFBXO38Benigncriteria provided, multiple submitters, no conflicts

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 5 · Orphanet: 1 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

GenCC gene–disease validity (cohort genes)

the Disease column is the GenCC-asserted condition — a cohort gene’s strongest validity may be for a related predisposition syndrome.

GeneClassificationInheritanceDiseaseRecords
FBXO38StrongAutosomal dominantneuronopathy, distal hereditary motor, type 2D5

Orphanet rare-disease linkage (cohort genes)

GeneOrphanet IDRare disease
FBXO38Orphanet:139525Distal hereditary motor neuropathy type 2

Cohort genes → proteins

1 cohort genes, 1 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence1

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
FBXO38HGNC:28844ENSG00000145868Q6PIJ6F-box only protein 38gencc,clinvar

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
FBXO38F-box only protein 38Substrate recognition component of a SCF (SKP1-CUL1-F-box protein) E3 ubiquitin-protein ligase complex which mediates the ubiquitination and subsequent proteasomal degradation of PDCD1/PD-1, thereby regulating T-cells-mediated immunity.

Protein-family classification

Druggable: 0 · Difficult: 0 · Unknown: 1 · Druggable fraction: 0.0

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Other/Unknown11.8×0.558

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
FBXO38Other/UnknownnoF-box_dom, LRR_dom_sf, F-box-like_dom_sf

Expression context

Cohort genes with no expression data: 0.

1 cohort gene are a single-cell marker in ≥1 SCXA experiment.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)1
unknown0

Top tissues across cohort

TissueCohort genes
adrenal tissue1
calcaneal tendon1
endothelial cell1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
FBXO38287ubiquitousmarkercalcaneal tendon, adrenal tissue, endothelial cell

Protein interactions among cohort

Intra-cohort edges: 0.

Hub genes (top 10 by interactor count)

SymbolInteractor count
FBXO381,732

Structural data

PDB: 0 · AlphaFold-only: 1 · No structure: 0

AlphaFold-only cohort genes (top 30 by pLDDT)

SymbolUniProtpLDDT
FBXO38Q6PIJ667.97

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 0. Enrichment computed across 1 evidence-associated genes (0 with Reactome annotation).

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 1 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
positive regulation of T cell mediated immune response to tumor cell12407.4×0.002FBXO38
positive regulation of T cell activation1443.5×0.005FBXO38
SCF-dependent proteasomal ubiquitin-dependent protein catabolic process1374.5×0.005FBXO38
protein K48-linked ubiquitination1168.5×0.009FBXO38
positive regulation of neuron projection development1137.0×0.009FBXO38
adaptive immune response184.3×0.012FBXO38

Therapeutics

Drug target analysis

Approved (phase 4): 0 · Phase ≥3: 0 · Phased (≥1): 0 · Undrugged: 1

Druggability breadth: 0 of 1 evidence-associated genes (0%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Top cohort targets by molecule count

SymbolMoleculesMax phase
FBXO3800

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 0.

Pharmacogenomics

Cohort genes with a PharmGKB record: 1; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

0 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)0
BPhased (≥1) drug, not yet approved0
CDruggable family + PDB, no drug0
DDruggable family + AlphaFold only, no drug0
EDifficult family or no structure, no drug1FBXO38

Undrugged target profiles

1 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

SymbolChEMBL assaysDrugged partners (top 3)
FBXO380

Clinical trials & evidence

Clinical trials

Clinical trials: 0.