Normophosphatemic familial tumoral calcinosis

disease
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Also known as NFTCtumoral calcinosis, familial, normophosphatemictumoral calcinosis, normophosphatemic, familial

Summary

Normophosphatemic familial tumoral calcinosis (MONDO:0012502) is a disease caused by SAMD9 (GenCC Strong), with 1 cohort gene.

At a glance

  • Causal gene: SAMD9 (GenCC Strong)
  • Cohort genes: 1
  • ClinVar variants: 44

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namenormophosphatemic familial tumoral calcinosis
Mondo IDMONDO:0012502
MeSHC566473
OMIM610455
Orphanet306658
DOIDDOID:0080170
UMLSC1864861
MedGen355311
GARD0010878
Is cancer (heuristic)no

Also known as: NFTC · normophosphatemic familial tumoral calcinosis · tumoral calcinosis, familial, normophosphatemic · tumoral calcinosis, normophosphatemic, familial

Data availability: 44 ClinVar variants · 5 GenCC gene-disease records.

Disease family

Classification path: disease › human disease › disease by body system or component › integumentary system disorder › familial tumoral calcinosisnormophosphatemic familial tumoral calcinosis

Related subtypes (4): tumoral calcinosis, hyperphosphatemic, familial, 2, tumoral calcinosis, hyperphosphatemic, familial, 3, familial hyperphosphatemic tumoral calcinosis/hyperphosphatemic hyperostosis syndrome, tumoral calcinosis, hyperphosphatemic, familial, 1

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

44 retrieved; paginated sample, class counts are floors:

21 uncertain significance, 8 benign/likely benign, 8 conflicting classifications of pathogenicity, 2 likely benign, 1 benign, 1 pathogenic, 1 not provided, 1 likely pathogenic, 1 pathogenic/likely pathogenic

ClinVarVariant (HGVS)GeneClassificationReview
1229NM_017654.4(SAMD9):c.4483A>G (p.Lys1495Glu)SAMD9Pathogeniccriteria provided, single submitter
985307NM_017654.4(SAMD9):c.2944C>T (p.Arg982Cys)SAMD9Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
556683NM_017654.4(SAMD9):c.132del (p.Val45fs)SAMD9Likely pathogenicno assertion criteria provided
1174761NM_017654.4(SAMD9):c.2686T>C (p.Tyr896His)SAMD9Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1299393NM_017654.4(SAMD9):c.1171G>T (p.Gly391Ter)SAMD9Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1648073NM_017654.4(SAMD9):c.4413G>A (p.Met1471Ile)SAMD9Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
225463NM_017654.4(SAMD9):c.460C>T (p.Gln154Ter)SAMD9Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
552976NM_017654.4(SAMD9):c.3381C>A (p.Tyr1127Ter)SAMD9Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
554815NM_017654.4(SAMD9):c.1800_1801del (p.Glu600fs)SAMD9Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
709973NM_017654.4(SAMD9):c.4724G>A (p.Gly1575Glu)SAMD9Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
992633NM_017654.4(SAMD9):c.3878G>A (p.Arg1293Gln)SAMD9Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1336034NM_017654.4(SAMD9):c.4001G>T (p.Arg1334Ile)SAMD9Uncertain significancecriteria provided, multiple submitters, no conflicts
1337200NM_017654.4(SAMD9):c.2611G>T (p.Glu871Ter)SAMD9Uncertain significancecriteria provided, multiple submitters, no conflicts
1337472NM_017654.4(SAMD9):c.3403A>G (p.Ile1135Val)SAMD9Uncertain significancecriteria provided, multiple submitters, no conflicts
1347521NM_017654.4(SAMD9):c.1460A>G (p.His487Arg)SAMD9Uncertain significancecriteria provided, multiple submitters, no conflicts
1497031NM_017654.4(SAMD9):c.83T>C (p.Ile28Thr)SAMD9Uncertain significancecriteria provided, multiple submitters, no conflicts
1506133NM_017654.4(SAMD9):c.29del (p.Asn10fs)SAMD9Uncertain significancecriteria provided, multiple submitters, no conflicts
1521693NM_017654.4(SAMD9):c.2581_2582insT (p.Glu861fs)SAMD9Uncertain significancecriteria provided, multiple submitters, no conflicts
1675124NM_017654.4(SAMD9):c.1252A>G (p.Asn418Asp)SAMD9Uncertain significancecriteria provided, single submitter
1675125NM_017654.4(SAMD9):c.2960T>C (p.Leu987Ser)SAMD9Uncertain significancecriteria provided, multiple submitters, no conflicts
225462NM_017654.4(SAMD9):c.95del (p.His32fs)SAMD9Uncertain significancecriteria provided, multiple submitters, no conflicts
2893962NM_017654.4(SAMD9):c.2641G>A (p.Asp881Asn)SAMD9Uncertain significancecriteria provided, multiple submitters, no conflicts
3687542NM_017654.4(SAMD9):c.2009del (p.Asn670fs)SAMD9Uncertain significancecriteria provided, multiple submitters, no conflicts
3780578NM_017654.4(SAMD9):c.1507del (p.Glu503fs)SAMD9Uncertain significancecriteria provided, single submitter
3892349NM_017654.4(SAMD9):c.2907A>C (p.Lys969Asn)SAMD9Uncertain significancecriteria provided, multiple submitters, no conflicts
553658NM_017654.4(SAMD9):c.2T>C (p.Met1Thr)SAMD9Uncertain significancecriteria provided, multiple submitters, no conflicts
554312NM_017654.4(SAMD9):c.4558G>T (p.Glu1520Ter)SAMD9Uncertain significancecriteria provided, multiple submitters, no conflicts
555093NM_017654.4(SAMD9):c.4331AAC[1] (p.Gln1445del)SAMD9Uncertain significancecriteria provided, multiple submitters, no conflicts
557138NM_017654.4(SAMD9):c.3505C>T (p.Gln1169Ter)SAMD9Uncertain significanceno assertion criteria provided
558150NM_017654.4(SAMD9):c.2509dup (p.Arg837fs)SAMD9Uncertain significanceno assertion criteria provided

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 14 · Orphanet: 2 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

GenCC gene–disease validity (cohort genes)

the Disease column is the GenCC-asserted condition — a cohort gene’s strongest validity may be for a related predisposition syndrome.

GeneClassificationInheritanceDiseaseRecords
SAMD9StrongAutosomal recessivenormophosphatemic familial tumoral calcinosis14

Orphanet rare-disease linkage (cohort genes)

GeneOrphanet IDRare disease
SAMD9Orphanet:306658Familial normophosphatemic tumoral calcinosis
SAMD9Orphanet:494433MIRAGE syndrome

Cohort genes → proteins

1 cohort genes, 1 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence1

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
SAMD9HGNC:1348ENSG00000205413Q5K651Sterile alpha motif domain-containing protein 9gencc,clinvar

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
SAMD9Sterile alpha motif domain-containing protein 9Double-stranded nucleic acid binding that acts as an antiviral factor by playing an essential role in the formation of cytoplasmic antiviral granules.

Protein-family classification

Druggable: 0 · Difficult: 0 · Unknown: 1 · Druggable fraction: 0.0

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Other/Unknown11.8×0.558

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
SAMD9Other/UnknownnoSAM, SAM/pointed_sf, P-loop_NTPase

Expression context

Cohort genes with no expression data: 0.

1 cohort gene are a single-cell marker in ≥1 SCXA experiment.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)1
unknown0

Top tissues across cohort

TissueCohort genes
amniotic fluid1
epithelium of esophagus1
esophagus squamous epithelium1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
SAMD9247ubiquitousmarkeresophagus squamous epithelium, amniotic fluid, epithelium of esophagus

Protein interactions among cohort

Intra-cohort edges: 0.

Hub genes (top 10 by interactor count)

SymbolInteractor count
SAMD91,316

Structural data

PDB: 1 · AlphaFold-only: 0 · No structure: 0

Cohort genes with PDB structures (top 30)

SymbolUniProtPDB entries
SAMD9Q5K6517

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 0. Enrichment computed across 1 evidence-associated genes (0 with Reactome annotation).

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 1 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
endosomal vesicle fusion11123.5×0.002SAMD9
innate immune response133.6×0.030SAMD9

Therapeutics

Drug target analysis

Approved (phase 4): 0 · Phase ≥3: 0 · Phased (≥1): 0 · Undrugged: 1

Druggability breadth: 0 of 1 evidence-associated genes (0%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Top cohort targets by molecule count

SymbolMoleculesMax phase
SAMD900

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 0.

Pharmacogenomics

Cohort genes with a PharmGKB record: 1; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

0 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)0
BPhased (≥1) drug, not yet approved0
CDruggable family + PDB, no drug0
DDruggable family + AlphaFold only, no drug0
EDifficult family or no structure, no drug1SAMD9

Undrugged target profiles

1 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

SymbolChEMBL assaysDrugged partners (top 3)
SAMD90

Clinical trials & evidence

Clinical trials

Clinical trials: 0.