Nutritional disorder

disease
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Summary

Nutritional disorder (MONDO:0005137) is a disease (an umbrella term covering 7 Mondo subtypes) and 11 clinical trials. A subtype of disease by etiologic mechanism — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Umbrella term: 7 Mondo subtypes
  • Clinical trials: 11

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namenutritional disorder
Mondo IDMONDO:0005137
EFOEFO:0001069
MeSHD009748
DOIDDOID:374
NCITC26836
SNOMED CT2492009
UMLSC3714509
MedGen811347
Is cancer (heuristic)no

Also known as: nutritional disorder

Disease family

An umbrella term covering 7 Mondo subtypes.

Classification path: disease › human disease › disease by etiologic mechanism › nutritional disorder

Related subtypes (4): cancer or benign tumor, idiopathic disease, disease of genetic or genomic mechanism, disease of primarily extrinsic mechanism

Subtypes (7): potassium deficiency disease, overnutrition, eating disorder, hemorrhagic disease of newborn, nutritional deficiency disease, lactose intolerance, refeeding syndrome

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

Drugs indicated for this disease

0 approved, 3 in late-stage (phase 3) trials. Disease-direct ChEMBL indications, not inferred from the associated-gene cohort below.

DrugDevelopment status
LidocainePhase 3 (in late-stage trials)
LiraglutidePhase 3 (in late-stage trials)
SemaglutidePhase 3 (in late-stage trials)

Earlier-phase candidates (phase 2, investigational — efficacy not yet established): OMEGA-3 FATTY ACIDS, Oxycodone.

Clinical trials & evidence

Clinical trials

Clinical trials: 11.

Phase distribution (across all retrieved trials)

PhaseTrials
Not specified9
PHASE31
EARLY_PHASE11

Top trials by phase / activity

NCTPhaseStatusTitle
NCT04012333PHASE3WITHDRAWNThe Effect of Higher Protein Dosing in Critically Ill Patients: A Multicenter Randomized Trial
NCT03898505EARLY_PHASE1COMPLETEDClinical Investigation on the Safety of Avocado Pulp Lipids
NCT01869530Not specifiedRECRUITINGTARGet Kids!: Measuring Nutrition in Young Preschool Children in the Primary Care Practice Setting
NCT02845895Not specifiedCOMPLETEDStudy to Compare Strategies to Improve Detection of Nutritional Disorders in Hospitalized Adults (Compass Project)
NCT03154502Not specifiedCOMPLETEDSodium Intake in Ecuadorian Population
NCT03465462Not specifiedCOMPLETEDThe Influence of Hypotensive Drugs on Mineral Status in Experimental and Clinical Studies
NCT04039282Not specifiedCOMPLETEDExploring the Use of myfood24 (an Online Nutritional Assessment Tool) in Clinical Dietetic Practice
NCT04301791Not specifiedUNKNOWNImpact of Nutritional Status on Clinical Outcome in PICU
NCT04554082Not specifiedCOMPLETEDEvaluation of Micronutrients in Obese Patients
NCT04709198Not specifiedCOMPLETEDNutritional Status, Muscle Wasting and Fraility in Intensive Care Patients
NCT05567211Not specifiedCOMPLETEDPrevention of Energy Deficit Syndrome in Female Athletes. Molecular Mechanisms Associated With Malnutrition.

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.