Ocular albinism with late-onset sensorineural deafness

disease
On this page

Also known as albinism ocular late onset sensorineural deafnessalbinism, ocular, with late-onset sensorineural deafnessOASD

Summary

Ocular albinism with late-onset sensorineural deafness (MONDO:0010390) is a disease. A subtype of ocular albinism — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Prevalence: <1 / 1 000 000 (Worldwide) [Orphanet-validated]
  • Phenotypes (HPO): 6

Clinical features

Epidemiology

Prevalence records

2 prevalence record(s), Orphanet:

TypeClassValueGeographyValidation
Cases/families9WorldwideValidated
Point prevalence<1 / 1 000 000WorldwideValidated

Signs & symptoms

Clinical features (HPO)

6 HPO clinical features (Orphanet curated; top 6 by frequency):

HPO IDTermFrequency
HP:0000407Sensorineural hearing impairmentVery frequent (80-99%)
HP:0000505Visual impairmentVery frequent (80-99%)
HP:0000613PhotophobiaVery frequent (80-99%)
HP:0000639NystagmusVery frequent (80-99%)
HP:0001107Ocular albinismVery frequent (80-99%)
HP:0000486StrabismusFrequent (30-79%)

Identifiers

Disease identifiers

FieldValue
Canonical nameocular albinism with late-onset sensorineural deafness
Mondo IDMONDO:0010390
OMIM300650
Orphanet1000
SNOMED CT722054007
UMLSC1845069
MedGen337149
GARD0000592
Is cancer (heuristic)no

Also known as: albinism ocular late onset sensorineural deafness · albinism, ocular, with late-onset sensorineural deafness · OASD

Disease family

This is a subtype of ocular albinism. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by etiologic mechanism › disease of genetic or genomic mechanism › hereditary diseaseinborn errors of metabolism › inborn disorder of amino acid and other organic acid metabolism › disorder of melanin metabolism › ocular albinismocular albinism with late-onset sensorineural deafness

Related subtypes (2): X-linked recessive ocular albinism, autosomal recessive ocular albinism

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.