O'nyong'nyong fever
disease diseaseOn this page
Also known as O'nyong-nyong virus caused disease or disorderO'nyong-nyong virus disease or disorderO'nyong-nyong virus infectious disease
Summary
O’nyong’nyong fever (MONDO:0000342) is a disease. A subtype of Alphavirus infectious disease — broader associated-gene and molecular evidence is on the parent page (see Disease family below).
Clinical features
No curated clinical features (Orphanet) for this disease.
Identifiers
Disease identifiers
| Field | Value |
|---|---|
| Canonical name | O’nyong’nyong fever |
| Mondo ID | MONDO:0000342 |
| DOID | DOID:0050516 |
| ICD-10-CM | A92.1 |
| SNOMED CT | 85579005 |
| UMLS | C0276286 |
| MedGen | 547234 |
| GARD | 0027520 |
| Is cancer (heuristic) | no |
Also known as: O’nyong-nyong virus caused disease or disorder · O’nyong-nyong virus disease or disorder · O’nyong-nyong virus infectious disease
Disease family
Classification path: disease › human disease › disease by etiologic mechanism › disease of primarily extrinsic mechanism › infectious disease › viral infectious disease › primary viral infectious disease › Togaviridae infectious disease › Alphavirus infectious disease › O’nyong’nyong fever
Related subtypes (5): Barmah forest virus disease, Ross river fever, Venezuelan equine encephalitis, chikungunya, western equine encephalitis
Genetics & variants
GWAS landscape
No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.
Variant details and genetic-evidence tiers
No tiered GWAS variants or ClinVar records for this disease.
Genes & proteins
No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).
Function
No pathway enrichment — requires an associated-gene cohort.
Therapeutics
No druggable-target or therapeutic data for this disease’s cohort.
Clinical trials & evidence
Clinical trials
Clinical trials: 0.
Related Atlas pages
No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.