Osteogenesis imperfecta type 4

disease
On this page

Also known as common variable OI with normal scleraeOI type 4OI type IVOI4osteogenesis imperfecta type IV

Summary

Osteogenesis imperfecta type 4 (MONDO:0008148) is a disease caused by COL1A1 (GenCC Definitive), with 10 cohort genes and 1 clinical trial. The dominant Reactome pathway is Collagen biosynthesis and modifying enzymes (4 cohort genes). Top therapeutic interventions include cholecalciferol and setrusumab.

At a glance

  • Prevalence: 1-9 / 100 000 (Sweden) [Orphanet-validated]
  • Causal gene: COL1A1 (GenCC Definitive)
  • Cohort genes: 10
  • ClinVar variants: 307
  • Clinical trials: 1

Clinical features

Epidemiology

Prevalence records

1 prevalence record(s), Orphanet:

TypeClassValueGeographyValidation
Prevalence at birth1-9 / 100 0001.35SwedenValidated

Identifiers

Disease identifiers

FieldValue
Canonical nameosteogenesis imperfecta type 4
Mondo IDMONDO:0008148
MeSHC536045
OMIM166220
Orphanet216820
DOIDDOID:0110340
ICD-11829297901
NCITC98576
SNOMED CT205497004
UMLSC0268363
MedGen78665
GARD0008696
Is cancer (heuristic)no

Also known as: common variable OI with normal sclerae · OI type 4 · OI type IV · OI4 · osteogenesis imperfecta type IV

Data availability: 307 ClinVar variants · 11 GenCC gene-disease records · 57 cell lines.

Disease family

Classification path: disease › human disease › disease by body system or component › musculoskeletal system disorderskeletal system disorderbone disorderbone development diseaseosteochondrodysplasiaosteogenesis imperfectaosteogenesis imperfecta and a reduction of bone mineral density.osteogenesis imperfecta type 4

Related subtypes (32): Cole-Carpenter syndrome 1, calvarial doughnut lesions-bone fragility syndrome, osteogenesis imperfecta type 1, osteogenesis imperfecta type 2, gnathodiaphyseal dysplasia, geroderma osteodysplastica, osteogenesis imperfecta type 3, osteogenesis imperfecta type 9, osteoporosis-pseudoglioma syndrome, Wiedemann-Rautenstrauch syndrome, spondylo-ocular syndrome, Bruck syndrome 2, osteogenesis imperfecta type 7, osteogenesis imperfecta type 8, osteogenesis imperfecta type 5, osteogenesis imperfecta type 11, autosomal recessive cutis laxa type 2B, osteogenesis imperfecta type 10, osteogenesis imperfecta type 12, osteogenesis imperfecta type 6, short stature-optic atrophy-Pelger-Huët anomaly syndrome, osteogenesis imperfecta type 14, osteogenesis imperfecta type 15, osteogenesis imperfecta type 16, Cole-Carpenter syndrome 2, Singleton-Merten syndrome 2, osteogenesis imperfecta type 17, autosomal recessive cutis laxa type 2A, Ehlers-Danlos syndrome, spondylodysplastic type, 1, Singleton-Merten syndrome 1, osteogenesis imperfecta, type 18, osteogenesis imperfecta, type 19

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

307 retrieved; paginated sample, class counts are floors:

123 pathogenic, 59 pathogenic/likely pathogenic, 47 likely pathogenic, 25 conflicting classifications of pathogenicity, 21 uncertain significance, 15 benign/likely benign, 14 benign, 2 likely benign, 1 not provided

ClinVarVariant (HGVS)GeneClassificationReview
1072588NM_000088.4(COL1A1):c.958-1G>ACOL1A1Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1074344NM_000088.4(COL1A1):c.1804G>T (p.Gly602Ter)COL1A1Pathogeniccriteria provided, multiple submitters, no conflicts
1076006NM_000088.4(COL1A1):c.288del (p.Asp97fs)COL1A1Pathogeniccriteria provided, multiple submitters, no conflicts
1192753NM_000088.4(COL1A1):c.3656A>G (p.Asp1219Gly)COL1A1Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1254747NM_000088.4(COL1A1):c.1177C>T (p.Gln393Ter)COL1A1Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1342741NM_000088.4(COL1A1):c.1444G>A (p.Gly482Arg)COL1A1Pathogeniccriteria provided, multiple submitters, no conflicts
1357973NM_000088.4(COL1A1):c.432del (p.Gly145fs)COL1A1Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1382447NM_000088.4(COL1A1):c.779G>A (p.Gly260Asp)COL1A1Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1431075NM_000088.4(COL1A1):c.1876-2A>GCOL1A1Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1431093NM_000088.4(COL1A1):c.1667del (p.Pro556fs)COL1A1Pathogeniccriteria provided, multiple submitters, no conflicts
1685271NM_000088.4(COL1A1):c.608G>A (p.Gly203Asp)COL1A1Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1687324NM_000088.4(COL1A1):c.4159G>A (p.Ala1387Thr)COL1A1Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
17286NM_000088.4(COL1A1):c.1057G>T (p.Gly353Cys)COL1A1Pathogeniccriteria provided, single submitter
17288NM_000088.4(COL1A1):c.2110G>T (p.Gly704Cys)COL1A1Pathogeniccriteria provided, single submitter
17312NM_000088.4(COL1A1):c.994G>A (p.Gly332Arg)COL1A1Pathogeniccriteria provided, multiple submitters, no conflicts
17313NM_000088.4(COL1A1):c.3541G>A (p.Gly1181Ser)COL1A1Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
17322NM_000088.4(COL1A1):c.3235G>A (p.Gly1079Ser)COL1A1Pathogeniccriteria provided, multiple submitters, no conflicts
17324NM_000088.4(COL1A1):c.1588G>A (p.Gly530Ser)COL1A1Pathogeniccriteria provided, multiple submitters, no conflicts
17331NM_000088.4(COL1A1):c.2515G>A (p.Gly839Ser)COL1A1Pathogenicno assertion criteria provided
17341NM_000088.4(COL1A1):c.642+1G>ACOL1A1Pathogenicno assertion criteria provided
17343NM_000088.4(COL1A1):c.934C>T (p.Arg312Cys)COL1A1Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
17345NM_000088.4(COL1A1):c.1845_1847del (p.Glu615_Ala616delinsAsp)COL1A1Pathogenicno assertion criteria provided
17346NM_000088.4(COL1A1):c.1299+1G>CCOL1A1Pathogenicno assertion criteria provided
17347NM_000088.4(COL1A1):c.3040C>T (p.Arg1014Cys)COL1A1Pathogeniccriteria provided, multiple submitters, no conflicts
1805989NM_000088.4(COL1A1):c.3540del (p.Gly1181fs)COL1A1Pathogeniccriteria provided, multiple submitters, no conflicts
2159543NM_000088.4(COL1A1):c.3155G>C (p.Gly1052Ala)COL1A1Pathogeniccriteria provided, multiple submitters, no conflicts
236248NM_000088.4(COL1A1):c.2775del (p.Gly926fs)COL1A1Pathogeniccriteria provided, multiple submitters, no conflicts
2444032NM_000088.4(COL1A1):c.423del (p.Gly142fs)COL1A1Pathogeniccriteria provided, single submitter
2504977NM_000088.4(COL1A1):c.750+1G>ACOL1A1Pathogeniccriteria provided, multiple submitters, no conflicts
2572450NM_000088.4(COL1A1):c.1631del (p.Pro544fs)COL1A1Pathogeniccriteria provided, single submitter

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 89 · Orphanet: 38 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

GenCC gene–disease validity (cohort genes)

the Disease column is the GenCC-asserted condition — a cohort gene’s strongest validity may be for a related predisposition syndrome.

GeneClassificationInheritanceDiseaseRecords
COL1A1DefinitiveAutosomal dominantosteogenesis imperfecta type 420
COL1A2DefinitiveAutosomal dominantosteogenesis imperfecta21
WNT1DefinitiveAutosomal recessiveosteogenesis imperfecta type 158
CRTAPStrongAutosomal recessiveosteogenesis imperfecta type 75
FKBP10StrongAutosomal recessiveosteogenesis imperfecta type 1111
PPIBStrongAutosomal recessiveosteogenesis imperfecta type 96
SERPINF1StrongAutosomal recessiveosteogenesis imperfecta type 65
SP7StrongAutosomal recessiveosteogenesis imperfecta type 124
SPARCStrongAutosomal recessiveosteogenesis imperfecta type 176
TMEM38BStrongAutosomal recessiveosteogenesis imperfecta type 143

Orphanet rare-disease linkage (cohort genes)

GeneOrphanet IDRare disease
FKBP10Orphanet:1149Kuskokwim syndrome
FKBP10Orphanet:216812Osteogenesis imperfecta type 3
FKBP10Orphanet:216820Osteogenesis imperfecta type 4
FKBP10Orphanet:2771Bruck syndrome
COL1A1Orphanet:1310Caffey disease
COL1A1Orphanet:1899Arthrochalasia Ehlers-Danlos syndrome
COL1A1Orphanet:216796Osteogenesis imperfecta type 1
COL1A1Orphanet:216804Osteogenesis imperfecta type 2
COL1A1Orphanet:216812Osteogenesis imperfecta type 3
COL1A1Orphanet:216820Osteogenesis imperfecta type 4
COL1A1Orphanet:230857Ehlers-Danlos/osteogenesis imperfecta syndrome
COL1A1Orphanet:287Classical Ehlers-Danlos syndrome
COL1A1Orphanet:31112Dermatofibrosarcoma protuberans
COL1A1Orphanet:314029High bone mass osteogenesis imperfecta
COL1A2Orphanet:1899Arthrochalasia Ehlers-Danlos syndrome
COL1A2Orphanet:216796Osteogenesis imperfecta type 1
COL1A2Orphanet:216804Osteogenesis imperfecta type 2
COL1A2Orphanet:216812Osteogenesis imperfecta type 3
COL1A2Orphanet:216820Osteogenesis imperfecta type 4
COL1A2Orphanet:230851Cardiac-valvular Ehlers-Danlos syndrome
COL1A2Orphanet:230857Ehlers-Danlos/osteogenesis imperfecta syndrome
COL1A2Orphanet:314029High bone mass osteogenesis imperfecta
SPARCOrphanet:216820Osteogenesis imperfecta type 4
WNT1Orphanet:216812Osteogenesis imperfecta type 3
WNT1Orphanet:216820Osteogenesis imperfecta type 4
WNT1Orphanet:85193Idiopathic juvenile osteoporosis
SP7Orphanet:1513Craniodiaphyseal dysplasia
SP7Orphanet:216820Osteogenesis imperfecta type 4
CRTAPOrphanet:2050Cole-Carpenter syndrome
CRTAPOrphanet:216804Osteogenesis imperfecta type 2
CRTAPOrphanet:216812Osteogenesis imperfecta type 3
CRTAPOrphanet:216820Osteogenesis imperfecta type 4
TMEM38BOrphanet:216820Osteogenesis imperfecta type 4
SERPINF1Orphanet:216812Osteogenesis imperfecta type 3
SERPINF1Orphanet:216820Osteogenesis imperfecta type 4
PPIBOrphanet:216804Osteogenesis imperfecta type 2
PPIBOrphanet:216812Osteogenesis imperfecta type 3
PPIBOrphanet:216820Osteogenesis imperfecta type 4

Cohort genes → proteins

10 cohort genes, 10 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence10

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
FKBP10HGNC:18169ENSG00000141756Q96AY3Peptidyl-prolyl cis-trans isomerase FKBP10gencc,clinvar
COL1A1HGNC:2197ENSG00000108821P02452Collagen alpha-1(I) chaingencc,clinvar
COL1A2HGNC:2198ENSG00000164692P08123Collagen alpha-2(I) chaingencc,clinvar
SPARCHGNC:11219ENSG00000113140P09486SPARCgencc
WNT1HGNC:12774ENSG00000125084P04628Proto-oncogene Wnt-1gencc
SP7HGNC:17321ENSG00000170374Q8TDD2Transcription factor Sp7gencc
CRTAPHGNC:2379ENSG00000170275O75718Cartilage-associated proteingencc
TMEM38BHGNC:25535ENSG00000095209Q9NVV0Trimeric intracellular cation channel type Bgencc
SERPINF1HGNC:8824ENSG00000132386P36955Pigment epithelium-derived factorgencc
PPIBHGNC:9255ENSG00000166794P23284Peptidyl-prolyl cis-trans isomerase Bgencc

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
FKBP10Peptidyl-prolyl cis-trans isomerase FKBP10PPIases accelerate the folding of proteins during protein synthesis.
COL1A1Collagen alpha-1(I) chainType I collagen is a member of group I collagen (fibrillar forming collagen).
COL1A2Collagen alpha-2(I) chainType I collagen is a member of group I collagen (fibrillar forming collagen).
SPARCSPARCAppears to regulate cell growth through interactions with the extracellular matrix and cytokines.
WNT1Proto-oncogene Wnt-1Ligand for members of the frizzled family of seven transmembrane receptors.
SP7Transcription factor Sp7Transcriptional activator essential for osteoblast differentiation.
CRTAPCartilage-associated proteinNecessary for efficient 3-hydroxylation of fibrillar collagen prolyl residues.
TMEM38BTrimeric intracellular cation channel type BIntracellular monovalent cation channel required for maintenance of rapid intracellular calcium release.
SERPINF1Pigment epithelium-derived factorNeurotrophic protein; induces extensive neuronal differentiation in retinoblastoma cells.
PPIBPeptidyl-prolyl cis-trans isomerase BPPIase that catalyzes the cis-trans isomerization of proline imidic peptide bonds in oligopeptides and may therefore assist protein folding.

Protein-family classification

Druggable: 1 · Difficult: 1 · Unknown: 8 · Druggable fraction: 0.1

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Other/Unknown81.4×0.327
Enzyme (other)11.2×0.725
Transcription factor10.8×0.725

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
FKBP10Other/UnknownnoPPIase_FKBP_dom, EF_hand_dom, EF-hand-dom_pair
COL1A1Other/UnknownnoFib_collagen_C, VWF_dom, Collagen
COL1A2Other/UnknownnoFib_collagen_C, Collagen, Collagen_superfamily
SPARCOther/UnknownnoOsteonectin_CS, Kazal_dom, Fol_N
WNT1Other/UnknownnoWnt, Wnt1, Wnt_CS
SP7Transcription factornoZnf_C2H2_type, Znf_C2H2_sf
CRTAPOther/UnknownnoTPR-like_helical_dom_sf, Collagen_mod_leprecan, Leprecan_dom
TMEM38BOther/UnknownnoTRIC_channel
SERPINF1Other/UnknownnoSerpin_fam, Serpin_CS, Serpin_dom
PPIBEnzyme (other)yes5.2.1.8Cyclophilin-type_PPIase_dom, Cyclophilin-type_PPIase_CS, Cyclophilin-like_dom_sf

Expression context

Cohort genes with no expression data: 0.

8 cohort genes are a single-cell marker in ≥1 SCXA experiment.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)10
unknown0

Top tissues across cohort

TissueCohort genes
stromal cell of endometrium6
periodontal ligament3
skin of hip2
tibia2
endocervix2
ascending aorta1
thoracic aorta1
granulocyte1
nucleus accumbens1
superior frontal gyrus1
male germ line stem cell (sensu Vertebrata) in testis1
primordial germ cell in gonad1
tendon of biceps brachii1
biceps brachii1
skeletal muscle tissue of rectus abdominis1
sperm1
pericardium1
pigmented layer of retina1
caput epididymis1
corpus epididymis1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
FKBP10179ubiquitousmarkerstromal cell of endometrium, ascending aorta, thoracic aorta
COL1A1298ubiquitousmarkerstromal cell of endometrium, skin of hip, periodontal ligament
COL1A2295ubiquitousmarkerperiodontal ligament, stromal cell of endometrium, skin of hip
SPARC306ubiquitousmarkertibia, stromal cell of endometrium, periodontal ligament
WNT173tissue_specificyesgranulocyte, nucleus accumbens, superior frontal gyrus
SP746tissue_specificyesprimordial germ cell in gonad, tibia, male germ line stem cell (sensu Vertebrata) in testis
CRTAP288ubiquitousmarkertendon of biceps brachii, stromal cell of endometrium, endocervix
TMEM38B264ubiquitousmarkersperm, biceps brachii, skeletal muscle tissue of rectus abdominis
SERPINF1280ubiquitousmarkerpigmented layer of retina, pericardium, endocervix
PPIB295ubiquitousmarkerstromal cell of endometrium, corpus epididymis, caput epididymis

Protein interactions among cohort

Intra-cohort edges: 17.

Hub genes (top 10 by interactor count)

SymbolInteractor count
COL1A15,341
PPIB4,718
FKBP103,473
SPARC3,105
WNT12,506
SP72,310
SERPINF11,809
CRTAP951
TMEM38B839
COL1A2179

Intra-cohort edges

ABSources
COL1A1COL1A2intact
COL1A1CRTAPintact, string_interaction
COL1A1FKBP10intact
COL1A1PPIBintact, string_interaction
COL1A1SP7string_interaction
COL1A1SPARCintact
CRTAPFKBP10string_interaction
CRTAPPPIBstring_interaction
CRTAPSERPINF1string_interaction
CRTAPTMEM38Bstring_interaction
FKBP10PPIBstring_interaction
FKBP10SERPINF1string_interaction
FKBP10TMEM38Bstring_interaction
PPIBTMEM38Bstring_interaction
SERPINF1TMEM38Bstring_interaction
SP7TMEM38Bstring_interaction
TMEM38BWNT1string_interaction

Structural data

PDB: 6 · AlphaFold-only: 4 · No structure: 0

Cohort genes with PDB structures (top 30)

SymbolUniProtPDB entries
COL1A1P0245214
PPIBP232848
CRTAPO757186
COL1A2P081235
SPARCP094864
SERPINF1P369553

AlphaFold-only cohort genes (top 30 by pLDDT)

SymbolUniProtpLDDT
FKBP10Q96AY389.19
WNT1P0462886.53
TMEM38BQ9NVV080.17
SP7Q8TDD252.78

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 52. Enrichment computed across 10 evidence-associated genes (7 with Reactome annotation).

Pathways by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 7 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

PathwayCohort genesFoldFDRSample cohort genes
Collagen biosynthesis and modifying enzymes497.4×2e-06COL1A1, COL1A2, CRTAP, PPIB
Defective VWF binding to collagen type I21087.6×3e-05COL1A1, COL1A2
Enhanced cleavage of VWF variant by ADAMTS132815.7×3e-05COL1A1, COL1A2
Defective VWF cleavage by ADAMTS13 variant2815.7×3e-05COL1A1, COL1A2
Enhanced binding of GP1BA variant to VWF multimer:collagen2466.1×6e-05COL1A1, COL1A2
Defective binding of VWF variant to GPIb:IX:V2466.1×6e-05COL1A1, COL1A2
ECM proteoglycans364.4×7e-05COL1A1, COL1A2, SPARC
GP1b-IX-V activation signalling2271.9×1e-04COL1A1, COL1A2
Anchoring fibril formation2217.5×2e-04COL1A1, COL1A2
Platelet Adhesion to exposed collagen2191.9×2e-04COL1A1, COL1A2
Scavenging by Class A Receptors2171.7×2e-04COL1A1, COL1A2
Fibronectin matrix formation2163.1×2e-04COL1A1, COL1A2
Crosslinking of collagen fibrils2163.1×2e-04COL1A1, COL1A2
RUNX2 regulates osteoblast differentiation2130.5×4e-04COL1A1, SP7
Platelet Aggregation (Plug Formation)2125.5×4e-04COL1A1, COL1A2
Syndecan interactions2120.8×4e-04COL1A1, COL1A2
MET activates PTK2 signaling2108.8×4e-04COL1A1, COL1A2
GPVI-mediated activation cascade288.2×6e-04COL1A1, COL1A2
Collagen chain trimerization274.2×8e-04COL1A1, COL1A2
Developmental Lineage of Pancreatic Ductal Cells265.3×0.001COL1A1, COL1A2
Assembly of collagen fibrils and other multimeric structures257.2×0.001COL1A1, COL1A2
Collagen degradation250.2×0.002COL1A1, COL1A2
Non-integrin membrane-ECM interactions244.1×0.002COL1A1, COL1A2
Integrin cell surface interactions238.4×0.002COL1A1, COL1A2
Cell surface interactions at the vascular wall227.2×0.005COL1A1, COL1A2
Scavenging by Class H Receptors1407.9×0.005SPARC
Immunoregulatory interactions between a Lymphoid and a non-Lymphoid cell224.9×0.005COL1A1, COL1A2
RUNX2 regulates bone development1116.5×0.016SP7
Binding and Uptake of Ligands by Scavenger Receptors177.7×0.023SPARC
WNT ligand biogenesis and trafficking160.4×0.028WNT1

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 10 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
collagen fibril organization489.9×1e-05FKBP10, COL1A1, COL1A2, CRTAP
bone development382.9×4e-04WNT1, TMEM38B, PPIB
skin morphogenesis2280.9×0.001COL1A1, COL1A2
negative regulation of cell-substrate adhesion2210.7×0.001COL1A1, WNT1
extracellular matrix assembly2187.2×0.001FKBP10, COL1A2
protein folding331.0×0.003FKBP10, CRTAP, PPIB
cerebellum formation11685.2×0.006WNT1
midbrain-hindbrain boundary maturation during brain development11685.2×0.006WNT1
protein heterotrimerization11685.2×0.006COL1A2
cellular response to vitamin E11685.2×0.006COL1A1
blood vessel development274.9×0.006COL1A1, COL1A2
cellular response to amino acid stimulus261.3×0.006COL1A1, COL1A2
bone mineralization254.4×0.006COL1A2, TMEM38B
cellular response to glucose stimulus253.5×0.006COL1A1, SERPINF1
cellular response to retinoic acid246.8×0.008COL1A1, SERPINF1
response to insulin246.2×0.008COL1A1, SP7
diencephalon development1842.6×0.009WNT1
central nervous system morphogenesis1842.6×0.009WNT1
extracellular matrix constituent secretion1842.6×0.009TMEM38B
cellular response to fluoride1842.6×0.009COL1A1
tooth mineralization1561.7×0.010COL1A1
astrocyte-dopaminergic neuron signaling1561.7×0.010WNT1
Spemann organizer formation1561.7×0.010WNT1
secretion by lung epithelial cell involved in lung growth1561.7×0.010TMEM38B
positive regulation of dermatome development1561.7×0.010WNT1
cellular response to cobalt ion1561.7×0.010SERPINF1
negative regulation of angiogenesis233.7×0.010SPARC, SERPINF1
forebrain anterior/posterior pattern specification1421.3×0.012WNT1
negative regulation of post-translational protein modification1421.3×0.012CRTAP
cell proliferation in midbrain1337.0×0.013WNT1

Therapeutics

Drug target analysis

Approved (phase 4): 1 · Phase ≥3: 1 · Phased (≥1): 2 · Undrugged: 8

Druggability breadth: 5 of 10 evidence-associated genes (50%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Genes with an approved drug

The molecule shown is one approved compound that hits the gene — not necessarily a drug of choice or one indicated for this disease.

SymbolExample approved molecule
PPIBCYCLOSPORINE

Top cohort targets by molecule count

SymbolMoleculesMax phase
PPIB44
WNT111
FKBP1000
COL1A100
COL1A200
SPARC00
SP700
CRTAP00
TMEM38B00
SERPINF100

Drugs targeting cohort genes (top 30)

MoleculeMax phaseTargets in cohort
CYCLOSPORINE4PPIB
ALISPORIVIR3PPIB
SCY 6352PPIB
NIM8112PPIB
CIRTUVIVINT1WNT1

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 1.

Cohort genes with ChEMBL bioactivity (full, sorted by assay count)

SymbolAssaysType breakdown
PPIB13Binding:13
WNT110Binding:10
COL1A18Binding:8
COL1A24Functional:4
SERPINF11Binding:1

Cohort enzymes (BRENDA EC)

SymbolEC numbersNames
PPIB5.2.1.8peptidylprolyl isomerase

Pharmacogenomics

Cohort genes with a PharmGKB record: 10; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

5 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

CompoundMax phaseCohort target (bioactivity)
CYCLOSPORINE4PPIB
ALISPORIVIR3PPIB
SCY 6352PPIB
NIM8112PPIB
CIRTUVIVINT1WNT1

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)1PPIB
BPhased (≥1) drug, not yet approved1WNT1
CDruggable family + PDB, no drug0
DDruggable family + AlphaFold only, no drug0
EDifficult family or no structure, no drug8FKBP10, COL1A1, COL1A2, SPARC, SP7, CRTAP, TMEM38B, SERPINF1

Undrugged target profiles

8 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

SymbolChEMBL assaysDrugged partners (top 3)
CRTAP0PPIB
FKBP100
COL1A18
COL1A24
SPARC0
SP70
TMEM38B0
SERPINF11

Clinical trials & evidence

Clinical trials

Clinical trials: 1.

Phase distribution (across all retrieved trials)

PhaseTrials
PHASE21

Top trials by phase / activity

NCTPhaseStatusTitle
NCT03118570PHASE2COMPLETEDA Study in Adult Patients With Type I, III or IV Osteogenesis Imperfecta Treated With BPS804

Drugs tested across these trials (top 30)

MoleculeMax phaseTrials referencing
CHOLECALCIFEROL41
SETRUSUMAB31