Pancreatic cholera

disease
On this page

Also known as islet cell WDHA syndromepancreatic WDHA syndromeVerner Morrison syndromewatery diarrhea syndromewatery diarrhea with hypokalemic alkalosiswatery diarrhea, hypokalemia, and achlorhydria syndromewatery diarrhoea syndromewatery diarrhoea with hypokalemic alkalosisWDHA syndromeWDHH

Summary

Pancreatic cholera (MONDO:0004058) is a disease and 3 clinical trials. Top therapeutic interventions include gefitinib, sorafenib, and edodekin alfa. A subtype of endocrine pancreas disorder — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Clinical trials: 3

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namepancreatic cholera
Mondo IDMONDO:0004058
DOIDDOID:6977
NCITC3488
SNOMED CT39998009
UMLSC0086768
MedGen39692
Is cancer (heuristic)no

Also known as: islet cell WDHA syndrome · pancreatic WDHA syndrome · Verner Morrison syndrome · watery diarrhea syndrome · watery diarrhea with hypokalemic alkalosis · watery diarrhea, hypokalemia, and achlorhydria syndrome · watery diarrhoea syndrome · watery diarrhoea with hypokalemic alkalosis · WDHA syndrome · WDHH

Disease family

This is a subtype of endocrine pancreas disorder. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › digestive system disorderpancreas disorderendocrine pancreas disorderpancreatic cholera

Related subtypes (11): gastrin secretion abnormality, abnormality of glucagon secretion, hyperinsulinism, post-surgical hypoinsulinemia, diabetes mellitus, aggressive insulitis, benign insulitis, pancreatic neuroendocrine neoplasm, islet cell adenomatosis, insulin-resistance syndrome type A, insulin-resistance syndrome type B

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 3.

Phase distribution (across all retrieved trials)

PhaseTrials
PHASE22
PHASE11

Top trials by phase / activity

NCTPhaseStatusTitle
NCT00075439PHASE2COMPLETEDGefitinib in Treating Patients With Progressive Metastatic Neuroendocrine Tumors
NCT00131911PHASE2COMPLETEDSorafenib Tosylate in Treating Patients With Progressive Metastatic Neuroendocrine Tumors
NCT00004074PHASE1COMPLETEDInterleukin-12 and Trastuzumab in Treating Patients With Cancer That Has High Levels of HER2/Neu

Drugs tested across these trials (top 30)

MoleculeMax phaseTrials referencing
GEFITINIB41
SORAFENIB41
EDODEKIN ALFA21