Parametritis
diseaseOn this page
Also known as inflammation of parametriumparametrium inflammation
Summary
Parametritis (MONDO:0006887) is a disease. A subtype of pelvic inflammatory disease — broader associated-gene and molecular evidence is on the parent page (see Disease family below).
Clinical features
No curated clinical features (Orphanet) for this disease.
Identifiers
Disease identifiers
| Field | Value |
|---|---|
| Canonical name | parametritis |
| Mondo ID | MONDO:0006887 |
| EFO | EFO:1001084 |
| MeSH | D010249 |
| DOID | DOID:1260 |
| SNOMED CT | 280483007 |
| UMLS | C0030455 |
| MedGen | 18295 |
| Is cancer (heuristic) | no |
Also known as: inflammation of parametrium · parametrium inflammation
Disease family
This is a subtype of pelvic inflammatory disease. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.
Classification path: disease › human disease › disease by body system or component › reproductive system disorder › female reproductive system disorder › pelvic inflammatory disease › parametritis
Related subtypes (3): salpingitis, acute female pelvic peritonitis, Fitz-Hugh-Curtis syndrome
Genetics & variants
GWAS landscape
No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.
Variant details and genetic-evidence tiers
No tiered GWAS variants or ClinVar records for this disease.
Genes & proteins
No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).
Function
No pathway enrichment — requires an associated-gene cohort.
Therapeutics
Drugs indicated for this disease
2 approved. Disease-direct ChEMBL indications, not inferred from the associated-gene cohort below.
| Drug | Development status |
|---|---|
| Ceftazidime | Approved (phase 4) |
| Clindamycin Phosphate | Approved (phase 4) |
Clinical trials & evidence
Clinical trials
Clinical trials: 0.
Related Atlas pages
No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.