Parasitic ectoparasitic infectious disease

Summary

Parasitic ectoparasitic infectious disease (MONDO:0002875) is a disease (an umbrella term covering 6 Mondo subtypes). A subtype of parasitic skin disorder — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

• Umbrella term: 6 Mondo subtypes

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

Field	Value
Canonical name	parasitic ectoparasitic infectious disease
Mondo ID	MONDO:0002875
MeSH	D004478
DOID	DOID:4110
ICD-10-CM	B85-B89
UMLS	C0013578
MedGen	3964
Is cancer (heuristic)	no

Also known as: ectoparasitic infestation · infestation, ectoparasitic · Infestations, ectoparasitic

Disease family

An umbrella term covering 6 Mondo subtypes.

Classification path: disease › human disease › disease by etiologic mechanism › disease of primarily extrinsic mechanism › infectious disease › parasitic infectious disease › parasitic skin disorder › parasitic ectoparasitic infectious disease

Related subtypes (5): cercarial dermatitis, cutaneous leishmaniasis, infection by Trypanosoma gambiense, Demodex folliculitis, demodicidosis of sebaceous gland

Subtypes (6): lice infestation, mite infestation, tick infestation, myiasis, tungiasis, external hirudiniasis

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.