Perrault syndrome 4

disease
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Also known as LARS2 Perrault syndromePerrault syndrome caused by mutation in LARS2Perrault syndrome type 4PRLTS4

Summary

Perrault syndrome 4 (MONDO:0014126) is a disease caused by LARS2 (GenCC Strong), with 2 cohort genes.

At a glance

  • Causal gene: LARS2 (GenCC Strong)
  • Cohort genes: 2
  • ClinVar variants: 55

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namePerrault syndrome 4
Mondo IDMONDO:0014126
OMIM615300
UMLSC3809105
MedGen815435
GARD0015943
Is cancer (heuristic)no

Also known as: LARS2 Perrault syndrome · Perrault syndrome 4 · Perrault syndrome caused by mutation in LARS2 · Perrault syndrome type 4 · PRLTS4

Data availability: 55 ClinVar variants · 2 GenCC gene-disease records.

Disease family

Classification path: disease › human disease › disease by etiologic mechanism › disease of genetic or genomic mechanism › hereditary disease › autosomal genetic disease › autosomal recessive diseasePerrault syndromePerrault syndrome 4

Related subtypes (6): Perrault syndrome 1, Perrault syndrome 3, Perrault syndrome 2, Perrault syndrome 5, Perrault syndrome 6, Perrault syndrome 7

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

55 retrieved; paginated sample, class counts are floors:

12 pathogenic, 12 uncertain significance, 11 likely pathogenic, 7 benign, 6 pathogenic/likely pathogenic, 6 conflicting classifications of pathogenicity, 1 benign/likely benign

ClinVarVariant (HGVS)GeneClassificationReview
427986NM_015340.3(LARS2):c.[1912G>A];[899C>T]Pathogeniccriteria provided, single submitter
1185630NM_015340.4(LARS2):c.1783del (p.Ala595fs)LARS2Pathogenicno assertion criteria provided
1185680NM_015340.4(LARS2):c.41_42del (p.Leu14fs)LARS2Pathogenicno assertion criteria provided
1342701NM_015340.4(LARS2):c.1670A>G (p.Tyr557Cys)LARS2Pathogenicno assertion criteria provided
191173NM_015340.4(LARS2):c.457A>C (p.Asn153His)LARS2Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
203991NM_015340.4(LARS2):c.1912G>A (p.Glu638Lys)LARS2Pathogeniccriteria provided, single submitter
2720074NM_015340.4(LARS2):c.1249A>G (p.Met417Val)LARS2Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
3601183NM_015340.4(LARS2):c.1239+1G>ALARS2Pathogeniccriteria provided, single submitter
3601186NM_015340.4(LARS2):c.1498G>T (p.Glu500Ter)LARS2Pathogeniccriteria provided, single submitter
3601187NM_015340.4(LARS2):c.1623-1G>TLARS2Pathogeniccriteria provided, single submitter
3601188NM_015340.4(LARS2):c.2218del (p.Thr740fs)LARS2Pathogeniccriteria provided, single submitter
3601190NM_015340.4(LARS2):c.2585_2589dup (p.Asp864fs)LARS2Pathogeniccriteria provided, single submitter
431124NM_015340.4(LARS2):c.1987C>T (p.Arg663Trp)LARS2Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
431125NM_015340.4(LARS2):c.371A>T (p.Asn124Ile)LARS2Pathogeniccriteria provided, single submitter
55871NM_015340.4(LARS2):c.1565C>A (p.Thr522Asn)LARS2Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
55872NM_015340.4(LARS2):c.1886C>T (p.Thr629Met)LARS2Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
55873NM_015340.4(LARS2):c.1077del (p.Ile360fs)LARS2Pathogeniccriteria provided, single submitter
992952NM_015340.4(LARS2):c.880G>A (p.Glu294Lys)LARS2Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1708965NM_015340.4(LARS2):c.2072G>A (p.Trp691Ter)LARS2Likely pathogeniccriteria provided, single submitter
2446359NM_015340.4(LARS2):c.1079T>C (p.Ile360Thr)LARS2Likely pathogeniccriteria provided, single submitter
3242248NM_015340.4(LARS2):c.795A>G (p.Ile265Met)LARS2Likely pathogeniccriteria provided, single submitter
3382175NM_015340.4(LARS2):c.620G>A (p.Trp207Ter)LARS2Likely pathogeniccriteria provided, single submitter
3601182NM_015340.4(LARS2):c.1193T>C (p.Ile398Thr)LARS2Likely pathogeniccriteria provided, single submitter
3601184NM_015340.4(LARS2):c.1334_1335del (p.Lys445fs)LARS2Likely pathogeniccriteria provided, single submitter
3601189NM_015340.4(LARS2):c.235-2A>GLARS2Likely pathogeniccriteria provided, multiple submitters, no conflicts
623147NM_015340.4(LARS2):c.1115C>G (p.Ser372Ter)LARS2Likely pathogeniccriteria provided, single submitter
691519NM_015340.4(LARS2):c.683G>A (p.Arg228His)LARS2Likely pathogeniccriteria provided, multiple submitters, no conflicts
691523NM_015340.4(LARS2):c.440A>C (p.Gln147Pro)LARS2Likely pathogeniccriteria provided, multiple submitters, no conflicts
691524NM_015340.4(LARS2):c.1607C>T (p.Pro536Leu)LARS2Likely pathogeniccriteria provided, multiple submitters, no conflicts
1916104NM_015340.4(LARS2):c.764C>T (p.Ala255Val)LARS2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 6 · Orphanet: 3 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

GenCC gene–disease validity (cohort genes)

the Disease column is the GenCC-asserted condition — a cohort gene’s strongest validity may be for a related predisposition syndrome.

GeneClassificationInheritanceDiseaseRecords
LARS2StrongAutosomal recessivePerrault syndrome 46

Orphanet rare-disease linkage (cohort genes)

GeneOrphanet IDRare disease
LARS2Orphanet:528091Hydrops-lactic acidosis-sideroblastic anemia-multisystemic failure syndrome
LARS2Orphanet:642945Perrault syndrome type 1
LARS2Orphanet:642976Perrault syndrome type 2

Cohort genes → proteins

2 cohort genes, 1 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence2

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
LARS2HGNC:17095ENSG00000011376Q15031Leucine–tRNA ligase, mitochondrialgencc,clinvar
LARS2-AS1HGNC:40796ENSG00000232455LARS2 antisense RNA 1clinvar

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
LARS2Leucine–tRNA ligase, mitochondrialCatalyzes the attachment of leucine to its cognate tRNA.

Protein-family classification

Druggable: 1 · Difficult: 0 · Unknown: 1 · Druggable fraction: 0.5

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Enzyme (other)16.0×0.320
Other/Unknown10.9×0.805

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
LARS2Enzyme (other)yes6.1.1.4aa-tRNA-synth_I_CS, aa-tRNA-synth_Ia, Leu-tRNA-ligase
LARS2-AS1Other/Unknownno

Expression context

Cohort genes with no expression data: 0.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)2
unknown0

Top tissues across cohort

TissueCohort genes
adrenal tissue1
oocyte1
ventricular zone1
hindlimb stylopod muscle1
male germ line stem cell (sensu Vertebrata) in testis1
primordial germ cell in gonad1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
LARS2285ubiquitousyesventricular zone, oocyte, adrenal tissue
LARS2-AS1108tissue_specificyesmale germ line stem cell (sensu Vertebrata) in testis, hindlimb stylopod muscle, primordial germ cell in gonad

Protein interactions among cohort

Intra-cohort edges: 0.

Hub genes (top 10 by interactor count)

SymbolInteractor count
LARS23,406
LARS2-AS10

Structural data

PDB: 0 · AlphaFold-only: 1 · No structure: 1

AlphaFold-only cohort genes (top 30 by pLDDT)

SymbolUniProtpLDDT
LARS2Q1503190.57

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 4. Enrichment computed across 2 evidence-associated genes (1 with Reactome annotation).

Pathways by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 1 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

PathwayCohort genesFoldFDRSample cohort genes
Mitochondrial tRNA aminoacylation1519.1×0.007LARS2
tRNA Aminoacylation1285.5×0.007LARS2
Translation162.1×0.021LARS2
Metabolism of proteins112.4×0.081LARS2

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 1 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
leucyl-tRNA aminoacylation18426.0×4e-04LARS2
tRNA aminoacylation for protein translation1842.6×0.002LARS2
mitochondrial translation1173.7×0.006LARS2

Therapeutics

Drug target analysis

Approved (phase 4): 0 · Phase ≥3: 0 · Phased (≥1): 0 · Undrugged: 2

Druggability breadth: 1 of 2 evidence-associated genes (50%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Top cohort targets by molecule count

SymbolMoleculesMax phase
LARS200
LARS2-AS100

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 1.

Cohort genes with ChEMBL bioactivity (full, sorted by assay count)

SymbolAssaysType breakdown
LARS21ADMET:1

Cohort enzymes (BRENDA EC)

SymbolEC numbersNames
LARS26.1.1.4leucine-tRNA ligase

Pharmacogenomics

Cohort genes with a PharmGKB record: 1; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

0 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)0
BPhased (≥1) drug, not yet approved0
CDruggable family + PDB, no drug0
DDruggable family + AlphaFold only, no drug1LARS2
EDifficult family or no structure, no drug1LARS2-AS1

Undrugged target profiles

2 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

SymbolChEMBL assaysDrugged partners (top 3)
LARS21
LARS2-AS10

Clinical trials & evidence

Clinical trials

Clinical trials: 0.