Pili torti

disease
On this page

Also known as coarse, dry, lusterless hair which breaks off easilypili torti (disease)twisted hair

Summary

Pili torti (MONDO:0009870) is a disease. A subtype of hair anomaly — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Prevalence: Unknown (Worldwide)
  • Phenotypes (HPO): 9

Clinical features

Signs & symptoms

Clinical features (HPO)

9 HPO clinical features (Orphanet curated; top 9 by frequency):

HPO IDTermFrequency
HP:0003777Pili tortiVery frequent (80-99%)
HP:0010719Abnormality of hair textureVery frequent (80-99%)
HP:0000164Abnormality of the dentitionFrequent (30-79%)
HP:0000534Abnormal eyebrow morphologyFrequent (30-79%)
HP:0000682Abnormality of dental enamelFrequent (30-79%)
HP:0001596AlopeciaFrequent (30-79%)
HP:0001597Abnormality of the nailFrequent (30-79%)
HP:0002299Brittle hairFrequent (30-79%)
HP:0000365Hearing impairmentOccasional (5-29%)

Identifiers

Disease identifiers

FieldValue
Canonical namepili torti
Mondo IDMONDO:0009870
MeSHC562485
OMIM261900
Orphanet2889
ICD-11813238041
SNOMED CT17170005
UMLSC0263491
MedGen82670
GARD0004361
Is cancer (heuristic)no

Also known as: coarse, dry, lusterless hair which breaks off easily · pili torti · pili torti (disease) · twisted hair

Data availability: 1 HPO phenotype.

Disease family

This is a subtype of hair anomaly. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › integumentary system disorder › disorder of pilosebaceous unithair anomalypili torti

Related subtypes (7): alopecia, ringed hair disease, trichodysplasia-xeroderma syndrome, uncombable hair syndrome, isolated familial wooly hair disorder, pili bifurcati, pili gemini

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.