Polyneuritis

disease
On this page

Also known as Inflammatory polyneuropathyMultiple neuritisPolyneuritides

Summary

Polyneuritis (MONDO:0021718) is a disease and 1 clinical trial. A subtype of polyneuropathy — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Clinical trials: 1

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namepolyneuritis
Mondo IDMONDO:0021718
NCITC26864
SNOMED CT76886005
UMLSC0032541
MedGen46005
Is cancer (heuristic)no

Also known as: Inflammatory polyneuropathy · Multiple neuritis · Polyneuritides · polyneuritis

Data availability: 1 HPO phenotype.

Disease family

This is a subtype of polyneuropathy. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › nervous system disorderperipheral nervous system disorderperipheral neuropathypolyneuropathypolyneuritis

Related subtypes (9): idiopathic progressive polyneuropathy, polyneuropathy in collagen vascular disease, critical illness polyneuropathy, demyelinating polyneuropathy, chronic polyneuropathy, paraneoplastic polyneuropathy, polyradiculoneuropathy, toxic polyneuropathy, organophosphate-induced delayed polyneuropathy

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 1.

Phase distribution (across all retrieved trials)

PhaseTrials
PHASE21

Top trials by phase / activity

NCTPhaseStatusTitle
NCT04153422PHASE2RECRUITINGIVIG in the Treatment of Autoimmune Small Fiber Neuropathy With TS-HDS, FGFR-3, or Plexin D1 Antibodies

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.