polyneuropathy associated with IgM monoclonal gammapathy with anti-MAG

disease
On this page

Also known as anti-MAG neuropathyneuropathy associated with monoclonal IgM antibodies to myelin-associated glycoprotein

Summary

polyneuropathy associated with IgM monoclonal gammapathy with anti-MAG (MONDO:0018977) is a disease and 5 clinical trials. Top therapeutic interventions include rituximab. A subtype of acquired peripheral neuropathy — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Clinical trials: 5

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namepolyneuropathy associated with IgM monoclonal gammapathy with anti-MAG
Mondo IDMONDO:0018977
Orphanet639
UMLSC1736154
MedGen1434346
GARD0013173
Is cancer (heuristic)no

Also known as: anti-MAG neuropathy · neuropathy associated with monoclonal IgM antibodies to myelin-associated glycoprotein

Disease family

Classification path: disease › human disease › disease by body system or component › nervous system disorderperipheral nervous system disorderperipheral neuropathy › acquired peripheral neuropathy › polyneuropathy associated with IgM monoclonal gammapathy with anti-MAG

Related subtypes (11): axonal polyneuropathy associated with IgG/IgM/IgA monoclonal gammopathy, cranial neuralgia, neuralgic amyotrophy, POEMS syndrome, non-recovering obstetric brachial plexus lesion, anterior cutaneous nerve entrapment syndrome, pudendal neuralgia, multifocal motor neuropathy, CANOMAD syndrome, simple cryoglobulinemia, radiation-induced plexopathy

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 5.

Phase distribution (across all retrieved trials)

PhaseTrials
Not specified2
PHASE31
PHASE21
PHASE1/PHASE21

Top trials by phase / activity

NCTPhaseStatusTitle
NCT05136976PHASE3RECRUITINGRituximab Therapy in Anti-Myelin Associated Glycoprotein Patients With Characteristics of Good Responders
NCT02967679PHASE2COMPLETEDSERENDEM : MD1003 in Patients Suffering From Demyelinating Neuropathies, an Open Label Pilot Study
NCT04568174PHASE1/PHASE2TERMINATEDFirst in Human Study to Test the Safety and Preliminary Efficacy of PPSGG in Patients With Anti-MAG Neuropathy
NCT07461896Not specifiedRECRUITINGStudying Nerve Function and Structure in Charcot-Marie-Tooth Disease, Anti-MAG Neuropathy and CIDP
NCT03397303Not specifiedCOMPLETEDQuantification of Nerve Stiffness in Neuropathies

Drugs tested across these trials (top 30)

MoleculeMax phaseTrials referencing
RITUXIMAB41