Polyp of sphenoidal sinus

Summary

Polyp of sphenoidal sinus (MONDO:0021404) is a disease. A subtype of paranasal sinus disorder — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

Field	Value
Canonical name	polyp of sphenoidal sinus
Mondo ID	MONDO:0021404
NCIT	C3933
SNOMED CT	90685008
UMLS	C0264255
MedGen	75538
Anatomy (UBERON)	UBERON:0001724
Is cancer (heuristic)	no

Also known as: polyp of sphenoid sinus · polyp of the sphenoid sinus · polyp of the sphenoidal sinus · sphenoid sinus polyp · sphenoidal sinus polyp

Disease family

This is a subtype of paranasal sinus disorder. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › otorhinolaryngologic disease › nasal disorder › paranasal sinus disorder › polyp of sphenoidal sinus

Related subtypes (6): paranasal sinus neoplasm, sinusitis, maxillary sinus cholesteatoma, polyp of frontal sinus, polyp of maxillary sinus, polyp of ethmoidal sinus

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.