Prader-Willi syndrome due to maternal uniparental disomy of chromosome 15

disease
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Also known as Prader-Willi syndrome due to maternal uniparental disomy of chromosome type 15UPD(15)mat

Summary

Prader-Willi syndrome due to maternal uniparental disomy of chromosome 15 (MONDO:0020298) is a disease. A subtype of Prader-Willi syndrome — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Phenotypes (HPO): 67

Clinical features

Signs & symptoms

Clinical features (HPO)

67 HPO clinical features (Orphanet curated; top 50 by frequency):

HPO IDTermFrequency
HP:0000028CryptorchidismVery frequent (80-99%)
HP:0001513ObesityVery frequent (80-99%)
HP:0002119VentriculomegalyVery frequent (80-99%)
HP:0008872Feeding difficulties in infancyVery frequent (80-99%)
HP:0000064Hypoplastic labia minoraFrequent (30-79%)
HP:0000486StrabismusFrequent (30-79%)
HP:0000708Atypical behaviorFrequent (30-79%)
HP:0000709PsychosisFrequent (30-79%)
HP:0000717AutismFrequent (30-79%)
HP:0000729Autistic behaviorFrequent (30-79%)
HP:0000786Primary amenorrheaFrequent (30-79%)
HP:0000789InfertilityFrequent (30-79%)
HP:0000823Delayed pubertyFrequent (30-79%)
HP:0000824Decreased response to growth hormone stimulation testFrequent (30-79%)
HP:0001252HypotoniaFrequent (30-79%)
HP:0001256Intellectual disability, mildFrequent (30-79%)
HP:0001328Specific learning disabilityFrequent (30-79%)
HP:0001508Failure to thriveFrequent (30-79%)
HP:0001773Short footFrequent (30-79%)
HP:0002205Recurrent respiratory infectionsFrequent (30-79%)
HP:0002591PolyphagiaFrequent (30-79%)
HP:0002650ScoliosisFrequent (30-79%)
HP:0003241External genital hypoplasiaFrequent (30-79%)
HP:0004322Short statureFrequent (30-79%)
HP:0006889Intellectual disability, borderlineFrequent (30-79%)
HP:0008734Decreased testicular sizeFrequent (30-79%)
HP:0010627Anterior pituitary hypoplasiaFrequent (30-79%)
HP:0011734Central adrenal insufficiencyFrequent (30-79%)
HP:0012104Parietal cortical atrophyFrequent (30-79%)
HP:0012105Occipital cortical atrophyFrequent (30-79%)
HP:0012650Perisylvian polymicrogyriaFrequent (30-79%)
HP:0025160Abnormal temper tantrumsFrequent (30-79%)
HP:0030339Decreased circulating gonadotropin levelFrequent (30-79%)
HP:0031169Postterm pregnancyFrequent (30-79%)
HP:0200055Small handFrequent (30-79%)
HP:0000044Hypogonadotropic hypogonadismFrequent (30-79%)
HP:0000046Small scrotumFrequent (30-79%)
HP:0000060Clitoral hypoplasiaFrequent (30-79%)
HP:0000819Diabetes mellitusOccasional (5-29%)
HP:0000938OsteopeniaOccasional (5-29%)
HP:0000939OsteoporosisOccasional (5-29%)
HP:0001250SeizureOccasional (5-29%)
HP:0001263Global developmental delayOccasional (5-29%)
HP:0001385Hip dysplasiaOccasional (5-29%)
HP:0001518Small for gestational ageOccasional (5-29%)
HP:0001558Decreased fetal movementOccasional (5-29%)
HP:0002360Sleep abnormalityOccasional (5-29%)
HP:0002578GastroparesisOccasional (5-29%)
HP:0002714Downturned corners of mouthOccasional (5-29%)
HP:0002870Obstructive sleep apneaOccasional (5-29%)

Identifiers

Disease identifiers

FieldValue
Canonical namePrader-Willi syndrome due to maternal uniparental disomy of chromosome 15
Mondo IDMONDO:0020298
Orphanet98754
UMLSC5680343
MedGen1826079
GARD0016861
Is cancer (heuristic)no

Also known as: Prader-Willi syndrome due to maternal uniparental disomy of chromosome type 15 · UPD(15)mat

Disease family

This is a subtype of Prader-Willi syndrome. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › syndromic diseasePrader-Willi syndromePrader-Willi syndrome due to maternal uniparental disomy of chromosome 15

Related subtypes (4): Schaaf-Yang syndrome, Prader-Willi syndrome due to translocation, Prader-Willi syndrome due to imprinting mutation, Prader-Willi syndrome due to paternal 15q11q13 deletion

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.