Primary unilateral adrenal hyperplasia

disease
On this page

Also known as PUAH

Summary

Primary unilateral adrenal hyperplasia (MONDO:0016504) is a disease. A subtype of primary aldosteronism — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Prevalence: Unknown (Worldwide)
  • Phenotypes (HPO): 15

Clinical features

Signs & symptoms

Clinical features (HPO)

15 HPO clinical features (Orphanet curated; top 15 by frequency):

HPO IDTermFrequency
HP:0000822HypertensionObligate (100%)
HP:0003351Decreased circulating renin concentrationObligate (100%)
HP:0011740Glucocortocoid-insensitive primary hyperaldosteronismObligate (100%)
HP:0002900HypokalemiaFrequent (30-79%)
HP:0003081Increased urinary potassiumFrequent (30-79%)
HP:0008221Adrenal hyperplasiaFrequent (30-79%)
HP:0200114Metabolic alkalosisFrequent (30-79%)
HP:0000360TinnitusOccasional (5-29%)
HP:0000421EpistaxisOccasional (5-29%)
HP:0001324Muscle weaknessOccasional (5-29%)
HP:0001959PolydipsiaOccasional (5-29%)
HP:0001962PalpitationsOccasional (5-29%)
HP:0002018NauseaOccasional (5-29%)
HP:0002315HeadacheOccasional (5-29%)
HP:0003394Muscle spasmOccasional (5-29%)

Identifiers

Disease identifiers

FieldValue
Canonical nameprimary unilateral adrenal hyperplasia
Mondo IDMONDO:0016504
Orphanet231580
ICD-111653668765
SNOMED CT715868005
UMLSC4274967
MedGen905007
GARD0020620
Is cancer (heuristic)no

Also known as: PUAH

Disease family

This is a subtype of primary aldosteronism. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › endocrine system disorderadrenal gland disorderadrenal cortex disorderadrenal gland hyperfunctionhyperaldosteronismprimary aldosteronismprimary unilateral adrenal hyperplasia

Related subtypes (3): aldosterone-producing adrenal cortex adenoma, ectopic aldosterone-producing tumor, familial hyperaldosteronism

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.