Pyridoxine deficiency anemia

disease
On this page

Also known as B6 deficiencies, vitaminB6 deficiency, vitaminB6 vitamin deficienciesB6 vitamin deficiencydeficiencies, B6 vitamindeficiencies, vitamin B6deficiency, B6 vitamindeficiency, pyridoxinedeficiency, vitamin B 6deficiency, vitamin B6pyridoxine deficiencypyridoxine Deficincyvitamin B6 deficienciesvitamin B6 deficiencyvitamin deficiencies, B6vitamin deficiency, B6

Summary

Pyridoxine deficiency anemia (MONDO:0004574) is a disease and 3 clinical trials. Top therapeutic interventions include pyridoxine and leucine. A subtype of vitamin B deficiency — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Clinical trials: 3

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namepyridoxine deficiency anemia
Mondo IDMONDO:0004574
MeSHD026681
DOIDDOID:8455
NCITC85221
SNOMED CT86448001
UMLSC0272013
MedGen543733
GARD0004616
Is cancer (heuristic)no

Also known as: B6 deficiencies, vitamin · B6 deficiency, vitamin · B6 vitamin deficiencies · B6 vitamin deficiency · deficiencies, B6 vitamin · deficiencies, vitamin B6 · deficiency, B6 vitamin · deficiency, pyridoxine · deficiency, vitamin B 6 · deficiency, vitamin B6 · pyridoxine deficiency · pyridoxine Deficincy · vitamin B6 deficiencies · vitamin B6 deficiency · vitamin deficiencies, B6 · vitamin deficiency, B6

Disease family

This is a subtype of vitamin B deficiency. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by etiologic mechanism › nutritional disordernutritional deficiency diseasevitamin deficiency disordervitamin B deficiencypyridoxine deficiency anemia

Related subtypes (2): beriberi, vitamin B12 deficiency

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 3.

Phase distribution (across all retrieved trials)

PhaseTrials
Not specified2
PHASE2/PHASE31

Top trials by phase / activity

NCTPhaseStatusTitle
NCT01128244PHASE2/PHASE3COMPLETEDVitamin B6 Effects for Women Taking Birth Control Pills
NCT00877812Not specifiedCOMPLETEDVitamin B6 Dependence of One-Carbon Metabolism
NCT03444155Not specifiedCOMPLETEDNatural Versus Synthetic Vitamin B Complexes in Human

Drugs tested across these trials (top 30)

MoleculeMax phaseTrials referencing
PYRIDOXINE41
LEUCINE32