Refractory plasma cell neoplasm

disease
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Summary

Refractory plasma cell neoplasm (MONDO:0004816) is a cancer and 3 clinical trials. Top therapeutic interventions include arsenic trioxide, sargramostim, and keyhole limpet hemocyanin. A subtype of refractory hematologic cancer — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Classification: Cancer
  • Clinical trials: 3

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namerefractory plasma cell neoplasm
Mondo IDMONDO:0004816
DOIDDOID:9544
NCITC7813
UMLSC0278620
MedGen75844
GARD0024112
Is cancer (heuristic)yes

Also known as: refractory plasma cell neoplasm

Disease family

This is a subtype of refractory hematologic cancer. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by etiologic mechanism › cancer or benign tumorneoplastic disease or syndromeneoplasmhematopoietic and lymphoid system neoplasmhematopoietic and lymphoid cell neoplasmrefractory hematologic cancerrefractory plasma cell neoplasm

Related subtypes (5): refractory hairy cell leukemia, refractory precursor T-lymphoblastic lymphoma/leukemia, refractory anemia with excess blasts in transformation, myelodysplastic syndrome with multilineage dysplasia, refractory cytopenia of childhood

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 3.

Phase distribution (across all retrieved trials)

PhaseTrials
PHASE23

Top trials by phase / activity

NCTPhaseStatusTitle
NCT00019097PHASE2COMPLETEDVaccine Therapy in Treating Patients With Multiple Myeloma
NCT00085345PHASE2WITHDRAWNMelphalan, Arsenic Trioxide, and Ascorbic Acid in Treating Patients With Relapsed or Refractory Multiple Myeloma
NCT00112879PHASE2WITHDRAWNArsenic Trioxide, Ascorbic Acid, Dexamethasone, and Thalidomide in Treating Patients With Multiple Myeloma

Drugs tested across these trials (top 30)

MoleculeMax phaseTrials referencing
ARSENIC TRIOXIDE42
SARGRAMOSTIM41
KEYHOLE LIMPET HEMOCYANIN31