Salpingitis
diseaseOn this page
Also known as fallopian tube inflammationinflammation of fallopian tube
Summary
Salpingitis (MONDO:0003619) is a disease (an umbrella term covering 6 Mondo subtypes) and 1 clinical trial. Top therapeutic interventions include gonadorelin acetate and triptorelin. A subtype of pelvic inflammatory disease — broader associated-gene and molecular evidence is on the parent page (see Disease family below).
At a glance
- Umbrella term: 6 Mondo subtypes
- Clinical trials: 1
Clinical features
No curated clinical features (Orphanet) for this disease.
Identifiers
Disease identifiers
| Field | Value |
|---|---|
| Canonical name | salpingitis |
| Mondo ID | MONDO:0003619 |
| MeSH | D012488 |
| DOID | DOID:5733 |
| NCIT | C26880 |
| SNOMED CT | 88157006 |
| UMLS | C0036130 |
| MedGen | 20646 |
| Is cancer (heuristic) | no |
Also known as: fallopian tube inflammation · inflammation of fallopian tube
Disease family
This is a subtype of pelvic inflammatory disease. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.
Classification path: disease › human disease › disease by body system or component › reproductive system disorder › female reproductive system disorder › pelvic inflammatory disease › salpingitis
Related subtypes (3): acute female pelvic peritonitis, parametritis, Fitz-Hugh-Curtis syndrome
Subtypes (6): tuberculous salpingitis, salpingo-oophoritis, acute salpingitis, chronic salpingitis, pyosalpinx, gonococcal salpingitis
Genetics & variants
GWAS landscape
No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.
Variant details and genetic-evidence tiers
No tiered GWAS variants or ClinVar records for this disease.
Genes & proteins
No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).
Function
No pathway enrichment — requires an associated-gene cohort.
Therapeutics
No druggable-target or therapeutic data for this disease’s cohort.
Clinical trials & evidence
Clinical trials
Clinical trials: 1.
Phase distribution (across all retrieved trials)
| Phase | Trials |
|---|---|
| PHASE1/PHASE2 | 1 |
Top trials by phase / activity
| NCT | Phase | Status | Title |
|---|---|---|---|
| NCT04071574 | PHASE1/PHASE2 | COMPLETED | Comparative Study on the Efficacy of Ovarian Stimulation Protocols on the Success Rate of ICSI in Female Infertility |
Drugs tested across these trials (top 30)
| Molecule | Max phase | Trials referencing |
|---|---|---|
| GONADORELIN ACETATE | 4 | 1 |
| TRIPTORELIN | 4 | 1 |
| CHEMBL2370644 | 0 | 1 |
Related Atlas pages
- Drugs: Gonadorelin Acetate, Triptorelin