Salpingitis

disease
On this page

Also known as fallopian tube inflammationinflammation of fallopian tube

Summary

Salpingitis (MONDO:0003619) is a disease (an umbrella term covering 6 Mondo subtypes) and 1 clinical trial. Top therapeutic interventions include gonadorelin acetate and triptorelin. A subtype of pelvic inflammatory disease — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Umbrella term: 6 Mondo subtypes
  • Clinical trials: 1

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namesalpingitis
Mondo IDMONDO:0003619
MeSHD012488
DOIDDOID:5733
NCITC26880
SNOMED CT88157006
UMLSC0036130
MedGen20646
Is cancer (heuristic)no

Also known as: fallopian tube inflammation · inflammation of fallopian tube

Disease family

This is a subtype of pelvic inflammatory disease. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › reproductive system disorderfemale reproductive system disorderpelvic inflammatory diseasesalpingitis

Related subtypes (3): acute female pelvic peritonitis, parametritis, Fitz-Hugh-Curtis syndrome

Subtypes (6): tuberculous salpingitis, salpingo-oophoritis, acute salpingitis, chronic salpingitis, pyosalpinx, gonococcal salpingitis

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 1.

Phase distribution (across all retrieved trials)

PhaseTrials
PHASE1/PHASE21

Top trials by phase / activity

NCTPhaseStatusTitle
NCT04071574PHASE1/PHASE2COMPLETEDComparative Study on the Efficacy of Ovarian Stimulation Protocols on the Success Rate of ICSI in Female Infertility

Drugs tested across these trials (top 30)

MoleculeMax phaseTrials referencing
GONADORELIN ACETATE41
TRIPTORELIN41
CHEMBL237064401