Screw worm infectious disease
disease diseaseOn this page
Also known as Cochliomyia hominivorax caused disease or disorderCochliomyia hominivorax disease or disorderCochliomyia hominivorax infectious diseaseinfection, screw worminfections, screw wormscrew worm infections
Summary
Screw worm infectious disease (MONDO:0005954) is a disease. A subtype of myiasis — broader associated-gene and molecular evidence is on the parent page (see Disease family below).
Clinical features
No curated clinical features (Orphanet) for this disease.
Identifiers
Disease identifiers
| Field | Value |
|---|---|
| Canonical name | screw worm infectious disease |
| Mondo ID | MONDO:0005954 |
| MeSH | D012610 |
| DOID | DOID:12927 |
| GARD | 0024260 |
| Is cancer (heuristic) | no |
Also known as: Cochliomyia hominivorax caused disease or disorder · Cochliomyia hominivorax disease or disorder · Cochliomyia hominivorax infectious disease · infection, screw worm · infections, screw worm · screw worm infections
Disease family
Classification path: disease › human disease › disease by etiologic mechanism › disease of primarily extrinsic mechanism › infectious disease › parasitic infectious disease › parasitic skin disorder › parasitic ectoparasitic infectious disease › myiasis › screw worm infectious disease
Related subtypes (3): hypodermyiasis, cavitary myiasis, cutaneous myiasis
Genetics & variants
GWAS landscape
No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.
Variant details and genetic-evidence tiers
No tiered GWAS variants or ClinVar records for this disease.
Genes & proteins
No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).
Function
No pathway enrichment — requires an associated-gene cohort.
Therapeutics
No druggable-target or therapeutic data for this disease’s cohort.
Clinical trials & evidence
Clinical trials
Clinical trials: 0.
Related Atlas pages
No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.