Secondary adrenal insufficiency

disease
On this page

Also known as central adrenal insufficiencycentral Hypoadrenalismhypocortisolism secondary to another disorder

Summary

Secondary adrenal insufficiency (MONDO:0043370) is a disease and 6 clinical trials. Top therapeutic interventions include fludrocortisone acetate, hydrocortisone, and mifepristone. A subtype of adrenocortical insufficiency — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Clinical trials: 6

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namesecondary adrenal insufficiency
Mondo IDMONDO:0043370
NCITC62602
SNOMED CT16685009
UMLSC0948387
MedGen184925
Is cancer (heuristic)no

Also known as: central adrenal insufficiency · central Hypoadrenalism · hypocortisolism secondary to another disorder · secondary adrenal insufficiency

Disease family

This is a subtype of adrenocortical insufficiency. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › endocrine system disorderadrenal gland disorderadrenal cortex disorderadrenocortical insufficiencysecondary adrenal insufficiency

Related subtypes (1): primary adrenal insufficiency

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 6.

Phase distribution (across all retrieved trials)

PhaseTrials
Not specified3
PHASE42
PHASE21

Top trials by phase / activity

NCTPhaseStatusTitle
NCT06832566PHASE4NOT_YET_RECRUITINGRamadan Fasting Outcomes in Patients With Secondary Adrenal Insufficiency Before and After the Treatment of Hypotension.
NCT02277587PHASE4COMPLETEDDual RElease Hydrocortisone Versus conventionAl Glucocorticoid replaceMent Therapy in Hypocortisolism (DREAM)
NCT04588688PHASE2TERMINATEDMifepristone Dynamic Testing for Diagnosis for Central Adrenal Insufficiency
NCT00561236Not specifiedCOMPLETEDDoes Intravitreal Injection of Triamcinolone Acetonide Impairs the Adrenal Function
NCT04700644Not specifiedCOMPLETEDAssessment of CAI in Adults With PWS.
NCT05827965Not specifiedCOMPLETEDRamadan Fasting in Secondary Adrenal Insufficiency Patients

Drugs tested across these trials (top 30)

MoleculeMax phaseTrials referencing
FLUDROCORTISONE ACETATE41
HYDROCORTISONE41
MIFEPRISTONE41
FLUDROCORTISONE21
CHEMBL474045201