Secondary hypertrophic osteoarthropathy

disease
On this page

Also known as hypertrophic pulmonary osteoarthropathyhypertrophic pulmonary osteoarthropathy (disorder) [ambiguous]

Summary

Secondary hypertrophic osteoarthropathy (MONDO:0006965) is a disease with 6 GWAS associations across 5 studies. A subtype of arthropathy — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • GWAS associations: 6

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namesecondary hypertrophic osteoarthropathy
Mondo IDMONDO:0006965
EFOEFO:1001174
MeSHD010005
DOIDDOID:10393
ICD-111325516156
SNOMED CT203357004
UMLSC0029412
MedGen18211
Is cancer (heuristic)no

Also known as: hypertrophic pulmonary osteoarthropathy · hypertrophic pulmonary osteoarthropathy (disorder) [ambiguous]

Data availability: 6 GWAS associations (5 studies).

Disease family

This is a subtype of arthropathy. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › musculoskeletal system disorderskeletal system disorderarthropathysecondary hypertrophic osteoarthropathy

Related subtypes (23): transient arthropathy, synovial plica syndrome, hypermobility syndrome, Tietze syndrome, neurogenic arthropathy, Behcet syndrome arthropathy, ankylosis, bursitis, synovium neoplasm, hydrarthrosis, articular cartilage disorder, hemarthrosis, tenosynovitis, ganglion or cyst of synovium/tendon/bursa, spondyloarthropathy, temporomandibular joint disorder, arthritic joint disease, de Quervain disease, frozen shoulder, patellofemoral pain syndrome, shoulder impingement syndrome, crystal arthropathy, vertebral joint disorder

Genetics & variants

GWAS landscape

6 GWAS associations across 5 studies. Top hits map to 3 distinct genes (as reported by GWAS).

Top associations by p-value

rsIDp-valueGeneRisk alleleOdds ratio
rs755706046e-18FANCAG0.11
rs561582321e-14TERTG0.07
chr11:890502399e-13T0.06
rs122035921e-12IRF4C0.08

Top studies (by case count)

StudyLead authorYearCasesControlsTitle
GCST90476197Verma A202441,322357,754Diversity and scale: Genetic architecture of 2068 traits in the VA Million Veteran Program.
GCST90478806Verma A202412,12897,047Diversity and scale: Genetic architecture of 2068 traits in the VA Million Veteran Program.
GCST90480467Verma A202412,12897,047Diversity and scale: Genetic architecture of 2068 traits in the VA Million Veteran Program.
GCST90478805Verma A20244,10249,987Diversity and scale: Genetic architecture of 2068 traits in the VA Million Veteran Program.
GCST90482349Verma A20243645,943Diversity and scale: Genetic architecture of 2068 traits in the VA Million Veteran Program.

Variant details and genetic-evidence tiers

Tier distribution (top 50 variants)

TierVariants
Tier 1: coding0
Tier 2: splice/UTR0
Tier 3: regulatory0
Tier 4: intronic/intergenic4

MAF distribution

BucketVariants
common (>=0.05)4
low_freq (0.01-0.05)0
rare (<0.01)0
unknown0

Functional consequences

ConsequenceCount
intron_variant3
unknown1

Top variants

rsIDChrPosAllelesMAFConsequenceGenep-valueTier
rs755706041689780269G>C0.079intron_variantFANCA6e-18Tier 4: intronic/intergenic
rs5615823251291620G>A,C,T0.221intron_variantTERT1e-14Tier 4: intronic/intergenic
chr11:890502390.3059e-13Tier 4: intronic/intergenic
rs122035926396321C>G,T0.16intron_variantIRF41e-12Tier 4: intronic/intergenic

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.