Secondary non-traumatic avascular necrosis

disease
On this page

Also known as secondary non-traumatic AVN

Summary

Secondary non-traumatic avascular necrosis (MONDO:0018376) is a disease. A subtype of secondary avascular necrosis — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Prevalence: Unknown (Worldwide)
  • Phenotypes (HPO): 13

Clinical features

Signs & symptoms

Clinical features (HPO)

13 HPO clinical features (Orphanet curated; top 13 by frequency):

HPO IDTermFrequency
HP:0010885Avascular necrosisVery frequent (80-99%)
HP:0001376Limitation of joint mobilityFrequent (30-79%)
HP:0002653Bone painFrequent (30-79%)
HP:0002960AutoimmunityFrequent (30-79%)
HP:0030955AlcoholismFrequent (30-79%)
HP:0001288Gait disturbanceOccasional (5-29%)
HP:0001370Rheumatoid arthritisOccasional (5-29%)
HP:0002664NeoplasmOccasional (5-29%)
HP:0002725Systemic lupus erythematosusOccasional (5-29%)
HP:0003549Abnormality of connective tissueOccasional (5-29%)
HP:0004377Hematological neoplasmOccasional (5-29%)
HP:0031520Groin painOccasional (5-29%)
HP:0100724HypercoagulabilityVery rare (<1-4%)

Identifiers

Disease identifiers

FieldValue
Canonical namesecondary non-traumatic avascular necrosis
Mondo IDMONDO:0018376
Orphanet399180
UMLSC5192430
MedGen1684106
GARD0021661
Is cancer (heuristic)no

Also known as: secondary non-traumatic AVN

Disease family

This is a subtype of secondary avascular necrosis. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › musculoskeletal system disorderskeletal system disorderbone disorderosteonecrosisavascular necrosissecondary avascular necrosissecondary non-traumatic avascular necrosis

Related subtypes (5): Gaucher disease type I, hereditary thrombophilia due to congenital histidine-rich (poly-L) glycoprotein deficiency, hereditary antithrombin deficiency, traumatic avascular necrosis, osteonecrosis of the jaw

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.