Senior-Loken syndrome 4

disease
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Also known as NPHP4 Senior-Loken syndromeSenior-Loken syndrome caused by mutation in NPHP4Senior-Loken syndrome type 4SLSN4

Summary

Senior-Loken syndrome 4 (MONDO:0011756) is a disease caused by NPHP4 (GenCC Definitive), with 1 cohort gene.

At a glance

  • Causal gene: NPHP4 (GenCC Definitive)
  • Cohort genes: 1
  • ClinVar variants: 563

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical nameSenior-Loken syndrome 4
Mondo IDMONDO:0011756
MeSHC537581
OMIM606996
DOIDDOID:0061278
UMLSC1846979
MedGen337697
GARD0015406
Is cancer (heuristic)no

Also known as: NPHP4 Senior-Loken syndrome · Senior-Loken syndrome 4 · Senior-Loken syndrome caused by mutation in NPHP4 · Senior-Loken syndrome type 4 · SLSN4

Data availability: 563 ClinVar variants · 2 GenCC gene-disease records.

Disease family

Classification path: disease › human disease › disease by etiologic mechanism › disease of genetic or genomic mechanism › hereditary diseaseciliopathySenior-Loken syndromeSenior-Loken syndrome 4

Related subtypes (8): Senior-Loken syndrome 1, senior-loken syndrome 3, Senior-Loken syndrome 5, Senior-Loken syndrome 6, Senior-Loken syndrome 7, nephronophthisis 15, Senior-Loken syndrome 8, Senior-Loken syndrome 9

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

563 retrieved; paginated sample, class counts are floors:

359 uncertain significance, 84 conflicting classifications of pathogenicity, 29 likely benign, 26 benign/likely benign, 21 likely pathogenic, 20 pathogenic/likely pathogenic, 16 benign, 8 pathogenic

ClinVarVariant (HGVS)GeneClassificationReview
1068634NM_015102.5(NPHP4):c.3083del (p.Gly1028fs)NPHP4Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1072607NM_015102.5(NPHP4):c.2908dup (p.Leu970fs)NPHP4Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1073618NM_015102.5(NPHP4):c.1271del (p.Lys424fs)NPHP4Pathogeniccriteria provided, multiple submitters, no conflicts
1179132NM_015102.5(NPHP4):c.3644+1G>TNPHP4Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1387867NM_015102.5(NPHP4):c.3773_3776del (p.Val1258fs)NPHP4Pathogeniccriteria provided, multiple submitters, no conflicts
1388969NM_015102.5(NPHP4):c.3368_3369del (p.Val1123fs)NPHP4Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1433031NM_015102.5(NPHP4):c.2007del (p.Phe670fs)NPHP4Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1694483NM_015102.5(NPHP4):c.3506del (p.Pro1169fs)NPHP4Pathogeniccriteria provided, multiple submitters, no conflicts
2156394NM_015102.5(NPHP4):c.3418G>T (p.Glu1140Ter)NPHP4Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
2719506NM_015102.5(NPHP4):c.3148del (p.Gln1050fs)NPHP4Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
2872668NM_015102.5(NPHP4):c.1692_1704del (p.Ala565fs)NPHP4Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
3403NM_015102.5(NPHP4):c.3272del (p.Val1091fs)NPHP4Pathogeniccriteria provided, multiple submitters, no conflicts
3404NM_015102.5(NPHP4):c.2335C>T (p.Gln779Ter)NPHP4Pathogenicno assertion criteria provided
3405NM_015102.5(NPHP4):c.1972C>T (p.Arg658Ter)NPHP4Pathogeniccriteria provided, single submitter
3586786NM_015102.5(NPHP4):c.1843C>T (p.Gln615Ter)NPHP4Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
3587242NM_015102.5(NPHP4):c.111G>A (p.Trp37Ter)NPHP4Pathogeniccriteria provided, multiple submitters, no conflicts
498434NM_015102.5(NPHP4):c.2011C>T (p.Gln671Ter)NPHP4Pathogeniccriteria provided, multiple submitters, no conflicts
503803NM_015102.5(NPHP4):c.1421_1423delinsCGTGG (p.Lys474fs)NPHP4Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
562355NM_015102.5(NPHP4):c.133C>T (p.Gln45Ter)NPHP4Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
562366NM_015102.5(NPHP4):c.1889_1892del (p.Pro630fs)NPHP4Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
595124NM_015102.5(NPHP4):c.2611+1G>ANPHP4Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
596144NM_015102.5(NPHP4):c.1357G>T (p.Glu453Ter)NPHP4Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
598365NM_015102.5(NPHP4):c.3409_3412del (p.Tyr1137fs)NPHP4Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
639470NM_015102.5(NPHP4):c.3325C>T (p.Arg1109Ter)NPHP4Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
867163NM_015102.5(NPHP4):c.3644+1G>ANPHP4Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
931168NM_015102.5(NPHP4):c.12G>A (p.Trp4Ter)NPHP4Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
943026NM_015102.5(NPHP4):c.834_841del (p.Ala279fs)NPHP4Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
956880NM_015102.5(NPHP4):c.1585_1586del (p.Gly529fs)NPHP4Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1030411NM_015102.5(NPHP4):c.3490_3491delinsA (p.Leu1164fs)NPHP4Likely pathogeniccriteria provided, single submitter
1179061NM_015102.5(NPHP4):c.1504-1G>ANPHP4Likely pathogeniccriteria provided, single submitter

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 9 · Orphanet: 2 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

GenCC gene–disease validity (cohort genes)

the Disease column is the GenCC-asserted condition — a cohort gene’s strongest validity may be for a related predisposition syndrome.

GeneClassificationInheritanceDiseaseRecords
NPHP4DefinitiveAutosomal recessiveSenior-Loken syndrome 49

Orphanet rare-disease linkage (cohort genes)

GeneOrphanet IDRare disease
NPHP4Orphanet:3156Senior-Loken syndrome
NPHP4Orphanet:93592Juvenile nephronophthisis

Cohort genes → proteins

1 cohort genes, 1 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence1

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
NPHP4HGNC:19104ENSG00000131697O75161Nephrocystin-4gencc,clinvar

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
NPHP4Nephrocystin-4Involved in the organization of apical junctions; the function is proposed to implicate a NPHP1-4-8 module.

Protein-family classification

Druggable: 0 · Difficult: 0 · Unknown: 1 · Druggable fraction: 0.0

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Other/Unknown11.8×0.558

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
NPHP4Other/UnknownnoNPHP4, Ig_NPHP4_4th, Ig_NPHP4_3rd

Expression context

Cohort genes with no expression data: 0.

1 cohort gene are a single-cell marker in ≥1 SCXA experiment.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)1
unknown0

Top tissues across cohort

TissueCohort genes
adenohypophysis1
right lobe of thyroid gland1
right uterine tube1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
NPHP4165ubiquitousmarkerright uterine tube, adenohypophysis, right lobe of thyroid gland

Protein interactions among cohort

Intra-cohort edges: 0.

Hub genes (top 10 by interactor count)

SymbolInteractor count
NPHP41,579

Structural data

PDB: 0 · AlphaFold-only: 1 · No structure: 0

AlphaFold-only cohort genes (top 30 by pLDDT)

SymbolUniProtpLDDT
NPHP4O7516172.44

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 2. Enrichment computed across 1 evidence-associated genes (1 with Reactome annotation).

Pathways by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 1 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

PathwayCohort genesFoldFDRSample cohort genes
Signaling by Hippo1543.8×0.004NPHP4
Anchoring of the basal body to the plasma membrane1113.1×0.009NPHP4

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 1 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
visual behavior12808.7×0.002NPHP4
protein localization to ciliary transition zone12407.4×0.002NPHP4
positive regulation of bicellular tight junction assembly11685.2×0.002NPHP4
photoreceptor cell outer segment organization11053.2×0.003NPHP4
photoreceptor cell maintenance1358.6×0.006NPHP4
retina development in camera-type eye1255.3×0.007NPHP4
negative regulation of canonical Wnt signaling pathway1117.8×0.012NPHP4
flagellated sperm motility1117.0×0.012NPHP4
cell-cell adhesion1101.5×0.012NPHP4
actin cytoskeleton organization179.1×0.014NPHP4
signal transduction116.1×0.062NPHP4

Therapeutics

Drug target analysis

Approved (phase 4): 0 · Phase ≥3: 0 · Phased (≥1): 0 · Undrugged: 1

Druggability breadth: 0 of 1 evidence-associated genes (0%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Top cohort targets by molecule count

SymbolMoleculesMax phase
NPHP400

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 0.

Pharmacogenomics

Cohort genes with a PharmGKB record: 1; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

0 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)0
BPhased (≥1) drug, not yet approved0
CDruggable family + PDB, no drug0
DDruggable family + AlphaFold only, no drug0
EDifficult family or no structure, no drug1NPHP4

Undrugged target profiles

1 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

SymbolChEMBL assaysDrugged partners (top 3)
NPHP40

Clinical trials & evidence

Clinical trials

Clinical trials: 0.