Sensory organ benign neoplasm

Summary

Sensory organ benign neoplasm (MONDO:0000633) is a cancer (an umbrella term covering 10 Mondo subtypes). A subtype of nervous system benign neoplasm — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

• Classification: Cancer
• Umbrella term: 10 Mondo subtypes

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

Field	Value
Canonical name	sensory organ benign neoplasm
Mondo ID	MONDO:0000633
DOID	DOID:0060096
Anatomy (UBERON)	UBERON:0000020
Is cancer (heuristic)	yes

Also known as: sense organ benign neoplasm

Disease family

This is a subtype of nervous system benign neoplasm. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by etiologic mechanism › cancer or benign tumor › neoplastic disease or syndrome › neoplasm › benign neoplasm › nervous system benign neoplasm › sensory organ benign neoplasm

Related subtypes (4): central nervous system organ benign neoplasm, neurocutaneous melanocytosis, hemangioblastoma, phakomatosis pigmentokeratotica

Subtypes (10): paranasal sinus Schneiderian papilloma, benign neoplasm of eye, benign neoplasm of ear, benign neoplasm of nasal cavity, benign neoplasm of tongue, benign neoplasm of sphenoidal sinus, benign neoplasm of frontal sinus, benign neoplasm of maxillary sinus, benign neoplasm of ethmoidal sinus, auditory system benign neoplasm

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.