Short stature-pituitary and cerebellar defects-small sella turcica syndrome

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Also known as CPHD4pituitary hormone deficiency, combined 4pituitary hormone deficiency, combined with or without cerebellar defectspituitary hormone deficiency, combined, 4pituitary hormone deficiency, combined, type 4short stature, pituitary and cerebellar defects and small sella turcica

Summary

Short stature-pituitary and cerebellar defects-small sella turcica syndrome (MONDO:0009880) is a disease caused by LHX4 (GenCC Definitive), with 3 cohort genes.

At a glance

  • Prevalence: <1 / 1 000 000 (Worldwide) [Orphanet-validated]
  • Causal gene: LHX4 (GenCC Definitive)
  • Cohort genes: 3
  • ClinVar variants: 76

Clinical features

Epidemiology

Prevalence records

2 prevalence record(s), Orphanet:

TypeClassValueGeographyValidation
Cases/families5WorldwideValidated
Point prevalence<1 / 1 000 000WorldwideValidated

Identifiers

Disease identifiers

FieldValue
Canonical nameshort stature-pituitary and cerebellar defects-small sella turcica syndrome
Mondo IDMONDO:0009880
MeSHC567492
OMIM262700
Orphanet85442
DOIDDOID:0061017
UMLSC2678408
MedGen394816
GARD0010604
Is cancer (heuristic)no

Also known as: CPHD4 · pituitary hormone deficiency, combined 4 · pituitary hormone deficiency, combined with or without cerebellar defects · pituitary hormone deficiency, combined, 4 · pituitary hormone deficiency, combined, type 4 · short stature, pituitary and cerebellar defects and small sella turcica

Data availability: 76 ClinVar variants · 5 GenCC gene-disease records.

Disease family

Classification path: disease › human disease › disease by body system or component › nervous system disordercongenital nervous system disordercombined pituitary hormone deficiencies, genetic formshort stature-pituitary and cerebellar defects-small sella turcica syndrome

Related subtypes (8): isolated congenital growth hormone deficiency, septooptic dysplasia, congenital isolated adrenocorticotropic hormone deficiency, non-acquired combined pituitary hormone deficiency with spine abnormalities, pituitary hormone deficiency, combined, 6, panhypopituitarism, pituitary hormone deficiency, combined, 1, pituitary hormone deficiency, combined or isolated, 8

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

76 retrieved; paginated sample, class counts are floors:

38 uncertain significance, 13 benign, 7 conflicting classifications of pathogenicity, 6 pathogenic, 6 benign/likely benign, 5 likely benign, 1 likely pathogenic

ClinVarVariant (HGVS)GeneClassificationReview
189234NM_033343.4(LHX4):c.452-2A>CACBD6Pathogeniccriteria provided, single submitter
7507NM_033343.4(LHX4):c.628G>C (p.Ala210Pro)ACBD6Pathogenicno assertion criteria provided
18444NM_033343.4(LHX4):c.293dup (p.Thr99fs)LHX4Pathogenicno assertion criteria provided
7506NM_033343.4(LHX4):c.607-1G>CLHX4-AS1Pathogenicno assertion criteria provided
7509NM_033343.4(LHX4):c.569T>G (p.Leu190Arg)LHX4-AS1Pathogenicno assertion criteria provided
7510NM_033343.4(LHX4):c.1162C>A (p.Pro388Thr)LHX4-AS1Pathogenicno assertion criteria provided
4056579NM_033343.4(LHX4):c.461_462del (p.Glu154fs)ACBD6Likely pathogeniccriteria provided, single submitter
293857NM_033343.4(LHX4):c.37G>A (p.Val13Ile)LHX4Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
293861NM_033343.4(LHX4):c.146A>G (p.His49Arg)LHX4Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
7508NM_033343.4(LHX4):c.250C>T (p.Arg84Cys)LHX4Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
801583NM_033343.4(LHX4):c.704G>A (p.Arg235Gln)LHX4Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
801584NM_033343.4(LHX4):c.776G>A (p.Arg259Gln)LHX4Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
874814NM_033343.4(LHX4):c.1007G>A (p.Ser336Asn)LHX4Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
876697NM_033343.4(LHX4):c.378G>A (p.Thr126=)LHX4Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1710224NM_033343.4(LHX4):c.822T>A (p.Tyr274Ter)ACBD6Uncertain significanceno assertion criteria provided
2433430NM_033343.4(LHX4):c.1087A>T (p.Ile363Phe)ACBD6Uncertain significancecriteria provided, single submitter
3238840NM_033343.4(LHX4):c.694A>G (p.Lys232Glu)ACBD6Uncertain significancecriteria provided, single submitter
874818NM_033343.4(LHX4):c.*17C>TACBD6Uncertain significancecriteria provided, single submitter
875752NM_033343.4(LHX4):c.*154A>GACBD6Uncertain significancecriteria provided, single submitter
875753NM_033343.4(LHX4):c.*221G>TACBD6Uncertain significancecriteria provided, single submitter
876749NM_033343.4(LHX4):c.*280T>CACBD6Uncertain significancecriteria provided, single submitter
876750NM_033343.4(LHX4):c.*460G>CACBD6Uncertain significancecriteria provided, single submitter
976050NM_033343.4(LHX4):c.602T>C (p.Val201Ala)ACBD6Uncertain significancecriteria provided, single submitter
1213718NM_033343.4(LHX4):c.464C>T (p.Ala155Val)LHX4Uncertain significancecriteria provided, single submitter
1705660NM_033343.4(LHX4):c.569T>C (p.Leu190Pro)LHX4Uncertain significancecriteria provided, single submitter
2433432NM_033343.4(LHX4):c.37_39del (p.Val13del)LHX4Uncertain significancecriteria provided, single submitter
2573040NM_033343.4(LHX4):c.451G>C (p.Asp151His)LHX4Uncertain significancecriteria provided, single submitter
2627082NM_033343.4(LHX4):c.364C>T (p.Arg122Trp)LHX4Uncertain significancecriteria provided, single submitter
293853NM_033343.4(LHX4):c.-172A>GLHX4Uncertain significancecriteria provided, single submitter
293854NM_033343.4(LHX4):c.-134G>CLHX4Uncertain significancecriteria provided, single submitter

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 8 · Orphanet: 4 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

GenCC gene–disease validity (cohort genes)

the Disease column is the GenCC-asserted condition — a cohort gene’s strongest validity may be for a related predisposition syndrome.

GeneClassificationInheritanceDiseaseRecords
LHX4DefinitiveAutosomal dominantshort stature-pituitary and cerebellar defects-small sella turcica syndrome8

Orphanet rare-disease linkage (cohort genes)

GeneOrphanet IDRare disease
LHX4Orphanet:226307Hypothyroidism due to deficient transcription factors involved in pituitary development or function
LHX4Orphanet:85442Short stature-pituitary and cerebellar defects-small sella turcica syndrome
LHX4Orphanet:95494Combined pituitary hormone deficiencies, genetic forms
LHX4Orphanet:95496Pituitary stalk interruption syndrome

Cohort genes → proteins

3 cohort genes, 2 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence3

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
LHX4HGNC:21734ENSG00000121454Q969G2LIM/homeobox protein Lhx4gencc,clinvar
ACBD6HGNC:23339ENSG00000230124Q9BR61Acyl-CoA-binding domain-containing protein 6clinvar
LHX4-AS1HGNC:40982LHX4 antisense RNA 1clinvar

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
LHX4LIM/homeobox protein Lhx4May play a critical role in the development of respiratory control mechanisms and in the normal growth and maturation of the lung.
ACBD6Acyl-CoA-binding domain-containing protein 6Binds long-chain acyl-coenzyme A molecules with a strong preference for unsaturated C18:1-CoA, lower affinity for unsaturated C20:4-CoA, and very weak affinity for saturated C16:0-CoA.

Protein-family classification

Druggable: 0 · Difficult: 2 · Unknown: 1 · Druggable fraction: 0.0

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Scaffold/PPI15.8×0.482
Transcription factor12.8×0.482
Other/Unknown10.6×0.914

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
LHX4Transcription factornoHD, Znf_LIM, Homeodomain-like_sf
ACBD6Scaffold/PPInoAcyl-CoA-binding_protein, Ankyrin_rpt, FERM/acyl-CoA-bd_prot_sf
LHX4-AS1Other/Unknownno

Expression context

Cohort genes with no expression data: 1.

1 cohort gene are a single-cell marker in ≥1 SCXA experiment.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)2
unknown1

Top tissues across cohort

TissueCohort genes
buccal mucosa cell1
pancreatic ductal cell1
sperm1
cortical plate1
ganglionic eminence1
left testis1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
LHX4166tissue_specificyesbuccal mucosa cell, sperm, pancreatic ductal cell
ACBD6231ubiquitousmarkercortical plate, ganglionic eminence, left testis
LHX4-AS1

Protein interactions among cohort

Intra-cohort edges: 0.

Hub genes (top 10 by interactor count)

SymbolInteractor count
LHX41,566
ACBD61,486
LHX4-AS10

Structural data

PDB: 2 · AlphaFold-only: 0 · No structure: 1

Cohort genes with PDB structures (top 30)

SymbolUniProtPDB entries
LHX4Q969G21
ACBD6Q9BR611

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 9. Enrichment computed across 3 evidence-associated genes (2 with Reactome annotation).

Pathways by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 2 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

PathwayCohort genesFoldFDRSample cohort genes
Mitochondrial Fatty Acid Beta-Oxidation1190.3×0.047ACBD6
Fatty acid metabolism165.6×0.048ACBD6
Signaling by ROBO receptors162.1×0.048LHX4
Regulation of expression of SLITs and ROBOs134.6×0.065LHX4
Axon guidance122.6×0.069LHX4
Nervous system development121.5×0.069LHX4
Metabolism of lipids115.8×0.080ACBD6
Developmental Biology17.2×0.150LHX4
Metabolism15.8×0.165ACBD6

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 1 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
medial motor column neuron differentiation18426.0×0.001LHX4
motor neuron axon guidance1702.2×0.006LHX4
placenta development1443.5×0.007LHX4
animal organ morphogenesis1191.5×0.012LHX4
neuron differentiation1100.3×0.018LHX4
negative regulation of apoptotic process134.8×0.043LHX4
apoptotic process128.7×0.045LHX4
positive regulation of transcription by RNA polymerase II114.9×0.076LHX4
regulation of transcription by RNA polymerase II111.7×0.086LHX4

Therapeutics

Drug target analysis

Approved (phase 4): 0 · Phase ≥3: 0 · Phased (≥1): 0 · Undrugged: 3

Druggability breadth: 0 of 3 evidence-associated genes (0%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Top cohort targets by molecule count

SymbolMoleculesMax phase
LHX400
ACBD600
LHX4-AS100

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 0.

Pharmacogenomics

Cohort genes with a PharmGKB record: 2; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

0 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)0
BPhased (≥1) drug, not yet approved0
CDruggable family + PDB, no drug0
DDruggable family + AlphaFold only, no drug0
EDifficult family or no structure, no drug3LHX4, ACBD6, LHX4-AS1

Undrugged target profiles

3 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

SymbolChEMBL assaysDrugged partners (top 3)
LHX40
ACBD60
LHX4-AS10

Clinical trials & evidence

Clinical trials

Clinical trials: 0.