Siberian tick typhus

Summary

Siberian tick typhus (MONDO:0001154) is a disease. A subtype of spotted fever — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

Field	Value
Canonical name	Siberian tick typhus
Mondo ID	MONDO:0001154
DOID	DOID:10921
ICD-10-CM	A77.2
ICD-11	490238539
SNOMED CT	186781003
UMLS	C0549160
MedGen	639822
GARD	0022894
Is cancer (heuristic)	no

Also known as: North Asian tick fever · North Asian tick fever (disorder) [ambiguous]

Disease family

This is a subtype of spotted fever. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by etiologic mechanism › disease of primarily extrinsic mechanism › infectious disease › bacterial infectious disease › Rickettsiosis › Rickettsiaceae infectious disease › spotted fever › Siberian tick typhus

Related subtypes (10): African tick-bite fever, Flinders island spotted fever, Japanese spotted fever, Rickettsia parkeri spotted fever, Rickettsia helvetica spotted fever, Queensland tick typhus, Rickettsia conorii infectious disease, Rocky mountain spotted fever, rickettsialpox, pseudotyphus of California

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.