Skin meningioma

Summary

Skin meningioma (MONDO:0004429) is a disease. A subtype of skin cancer — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

Field	Value
Canonical name	skin meningioma
Mondo ID	MONDO:0004429
DOID	DOID:8006
UMLS	C1275260
MedGen	698541
GARD	0023999
Anatomy (UBERON)	UBERON:0000014
Is cancer (heuristic)	no

Also known as: meningioma (disease) of zone of skin · primary meningioma of the skin · zone of skin meningioma (disease)

Disease family

This is a subtype of skin cancer. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › integumentary system disorder › integumentary system cancer › skin cancer › skin meningioma

Related subtypes (12): labium majus cancer, labia minora cancer, prepuce cancer, sweat gland cancer, cutaneous ganglioneuroma, skin carcinoma, malignant dermis tumor, cutaneous melanoma, primary cutaneous lymphoma, CD4+/CD56+ hematodermic neoplasm, eyelid cancer, sebaceous gland cancer

Subtypes (1): primary skin meningioma

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.