Spontaneous intracranial hypotension

disease
On this page

Also known as spontaneous cerebrospinal fluid leak

Summary

Spontaneous intracranial hypotension (MONDO:0018624) is a disease and 9 clinical trials. A subtype of headache disorder — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Prevalence: 1-9 / 100 000 (Worldwide) [Orphanet-validated]
  • Clinical trials: 9

Clinical features

Epidemiology

Prevalence records

1 prevalence record(s), Orphanet:

TypeClassValueGeographyValidation
Annual incidence1-9 / 100 0005WorldwideValidated

Identifiers

Disease identifiers

FieldValue
Canonical namespontaneous intracranial hypotension
Mondo IDMONDO:0018624
Orphanet443180
ICD-111304151002
UMLSC0751731
MedGen155914
GARD0021854
NORD1901
Is cancer (heuristic)no

Also known as: spontaneous cerebrospinal fluid leak

Disease family

This is a subtype of headache disorder. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › nervous system disorderneurological pain disorderheadache disorderspontaneous intracranial hypotension

Related subtypes (3): hypnic headache, reversible cerebral vasoconstriction syndrome, exertional headache

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 9.

Phase distribution (across all retrieved trials)

PhaseTrials
Not specified9

Top trials by phase / activity

NCTPhaseStatusTitle
NCT05922514Not specifiedRECRUITINGA Clinical Registry of Spontaneous Intracranial Hypotension
NCT06374524Not specifiedRECRUITINGGreater Occipital Nerve Block for Spontaneous Intracranial Hypotension
NCT06805591Not specifiedENROLLING_BY_INVITATIONPathophysiology and Treatment of Cerebrospinal Hypotension Exploration Study
NCT07085260Not specifiedNOT_YET_RECRUITINGONSD Changes and Treatment Response After Epidural Blood Patch in Spontaneous Intracranial Hypotension
NCT07582744Not specifiedNOT_YET_RECRUITINGCVFs in SIH: EID vs. PCD CTM
NCT07602556Not specifiedNOT_YET_RECRUITINGAutologous Platelet-rich Plasma Versus Whole Blood for Epidural Patch in Spontaneous Intracranial Hypotension
NCT02261792Not specifiedCOMPLETEDSpontaneous Intracranial Hypotension Treatment SIHT
NCT02603549Not specifiedSUSPENDEDPituitary Function and Spontaneous Intracranial Hypotension
NCT07283536Not specifiedWITHDRAWNBlind Versus Targeted Epidural Patch With Autologous Platelet-rich Plasma for Spontaneous Intracranial Hypotension

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.