Sporadic adult-onset ataxia of unknown etiology

disease
On this page

Also known as idiopathic late-onset cerebellar ataxiaSAOA

Summary

Sporadic adult-onset ataxia of unknown etiology (MONDO:0016591) is a disease and 1 clinical trial. A subtype of acquired ataxia — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Prevalence: 1-9 / 100 000 (Italy) [Orphanet-validated]
  • Phenotypes (HPO): 28
  • Clinical trials: 1

Clinical features

Epidemiology

Prevalence records

3 prevalence record(s), Orphanet:

TypeClassValueGeographyValidation
Point prevalence1-9 / 100 0006.9ItalyValidated
Point prevalence1-9 / 100 0008.4United KingdomValidated
Point prevalence1-9 / 100 0007.6EuropeNot yet validated

Signs & symptoms

Clinical features (HPO)

28 HPO clinical features (Orphanet curated; top 28 by frequency):

HPO IDTermFrequency
HP:0000496Abnormality of eye movementVery frequent (80-99%)
HP:0001251AtaxiaVery frequent (80-99%)
HP:0002066Gait ataxiaVery frequent (80-99%)
HP:0000572Visual lossFrequent (30-79%)
HP:0000608Macular degenerationFrequent (30-79%)
HP:0000640Gaze-evoked nystagmusFrequent (30-79%)
HP:0002354Memory impairmentFrequent (30-79%)
HP:0007670Abnormal vestibulo-ocular reflexFrequent (30-79%)
HP:0007772Impaired smooth pursuitFrequent (30-79%)
HP:0008278Cerebellar cortical atrophyFrequent (30-79%)
HP:0012332Abnormal autonomic nervous system physiologyFrequent (30-79%)
HP:0000338Hypomimic faceOccasional (5-29%)
HP:0000763Sensory neuropathyOccasional (5-29%)
HP:0001257SpasticityOccasional (5-29%)
HP:0001260DysarthriaOccasional (5-29%)
HP:0001291Abnormal cranial nerve morphologyOccasional (5-29%)
HP:0001300ParkinsonismOccasional (5-29%)
HP:0001347HyperreflexiaOccasional (5-29%)
HP:0002015DysphagiaOccasional (5-29%)
HP:0002063RigidityOccasional (5-29%)
HP:0002075DysdiadochokinesisOccasional (5-29%)
HP:0002304AkinesiaOccasional (5-29%)
HP:0002322Resting tremorOccasional (5-29%)
HP:0002362Shuffling gaitOccasional (5-29%)
HP:0003487Babinski signOccasional (5-29%)
HP:0000020Urinary incontinenceVery rare (<1-4%)
HP:0000726DementiaVery rare (<1-4%)
HP:0002080Intention tremorVery rare (<1-4%)

Identifiers

Disease identifiers

FieldValue
Canonical namesporadic adult-onset ataxia of unknown etiology
Mondo IDMONDO:0016591
Orphanet247234
SNOMED CT734023003
UMLSC4518339
MedGen1383968
GARD0020654
Is cancer (heuristic)no

Also known as: idiopathic late-onset cerebellar ataxia · SAOA

Disease family

Classification path: disease › human disease › disease by body system or component › nervous system disorder › atactic disorder › acquired ataxia › sporadic adult-onset ataxia of unknown etiology

Related subtypes (1): immune-mediated cerebellar ataxia

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 1.

Phase distribution (across all retrieved trials)

PhaseTrials
Not specified1

Top trials by phase / activity

NCTPhaseStatusTitle
NCT01793168Not specifiedRECRUITINGRare Disease Patient Registry & Natural History Study - Coordination of Rare Diseases at Sanford

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.