Steroid-induced glaucoma - borderline
diseaseOn this page
Also known as steroid responders borderline glaucoma
Summary
Steroid-induced glaucoma - borderline (MONDO:0001996) is a disease and 1 clinical trial. Top therapeutic interventions include ranibizumab. A subtype of borderline glaucoma — broader associated-gene and molecular evidence is on the parent page (see Disease family below).
At a glance
- Clinical trials: 1
Clinical features
No curated clinical features (Orphanet) for this disease.
Identifiers
Disease identifiers
| Field | Value |
|---|---|
| Canonical name | steroid-induced glaucoma - borderline |
| Mondo ID | MONDO:0001996 |
| DOID | DOID:14548 |
| SNOMED CT | 302895007 |
| UMLS | C0339572 |
| MedGen | 573233 |
| Is cancer (heuristic) | no |
Also known as: steroid responders borderline glaucoma
Disease family
This is a subtype of borderline glaucoma. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.
Classification path: disease › human disease › disease by body system or component › disorder of orbital region › eye disorder › glaucoma › borderline glaucoma › steroid-induced glaucoma - borderline
Related subtypes (1): anatomical narrow angle borderline glaucoma
Genetics & variants
GWAS landscape
No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.
Variant details and genetic-evidence tiers
No tiered GWAS variants or ClinVar records for this disease.
Genes & proteins
No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).
Function
No pathway enrichment — requires an associated-gene cohort.
Therapeutics
No druggable-target or therapeutic data for this disease’s cohort.
Clinical trials & evidence
Clinical trials
Clinical trials: 1.
Phase distribution (across all retrieved trials)
| Phase | Trials |
|---|---|
| PHASE2 | 1 |
Top trials by phase / activity
| NCT | Phase | Status | Title |
|---|---|---|---|
| NCT01564108 | PHASE2 | COMPLETED | The LIMO Study, Lucentis for Treatment of Uveitic Patients With Refractory Cystoid Macular Oedema |
Drugs tested across these trials (top 30)
| Molecule | Max phase | Trials referencing |
|---|---|---|
| RANIBIZUMAB | 4 | 1 |
Related Atlas pages
- Drugs: Ranibizumab