Talo-patello-scaphoid osteolysis

disease
On this page

Also known as singh-Williams-McAlister syndrome

Summary

Talo-patello-scaphoid osteolysis (MONDO:0012330) is a disease. A subtype of primary osteolysis — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Prevalence: <1 / 1 000 000 (Worldwide) [Orphanet-validated]
  • Phenotypes (HPO): 6

Clinical features

Epidemiology

Prevalence records

2 prevalence record(s), Orphanet:

TypeClassValueGeographyValidation
Cases/families2WorldwideValidated
Point prevalence<1 / 1 000 000WorldwideValidated

Signs & symptoms

Clinical features (HPO)

6 HPO clinical features (Orphanet curated; top 6 by frequency):

HPO IDTermFrequency
HP:0003037Enlarged jointsVery frequent (80-99%)
HP:0006202Osteolysis of scaphoidsVery frequent (80-99%)
HP:0006378Osteolysis of patellaeVery frequent (80-99%)
HP:0008095Osteolysis of talusVery frequent (80-99%)
HP:0010044Short 4th metacarpalVery frequent (80-99%)
HP:0100769SynovitisVery frequent (80-99%)

Identifiers

Disease identifiers

FieldValue
Canonical nametalo-patello-scaphoid osteolysis
Mondo IDMONDO:0012330
MeSHC536894
OMIM609655
Orphanet50809
ICD-111276091756
UMLSC1864784
MedGen400611
GARD0010061
Is cancer (heuristic)no

Also known as: singh-Williams-McAlister syndrome

Disease family

This is a subtype of primary osteolysis. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by etiologic mechanism › disease of genetic or genomic mechanism › hereditary diseaseskeletal dysplasiaprimary osteolysistalo-patello-scaphoid osteolysis

Related subtypes (13): acroosteolysis, multicentric carpo-tarsal osteolysis with or without nephropathy, pacman dysplasia, familial expansile osteolysis, Hutchinson-Gilford progeria syndrome, polycystic lipomembranous osteodysplasia with sclerosing leukoencephaly, hyaline fibromatosis syndrome, autosomal recessive distal osteolysis syndrome, Paget disease of bone 2, early-onset, Nestor-Guillermo progeria syndrome, mandibuloacral dysplasia, phalangeal microgeodic syndrome, multicentric osteolysis-nodulosis-arthropathy spectrum

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.