Trichosporonosis
disease diseaseOn this page
Also known as Trichosporon caused disease or disorderTrichosporon disease or disorderTrichosporon infectious disease
Summary
Trichosporonosis (MONDO:0000306) is a disease. A subtype of opportunistic systemic mycosis — broader associated-gene and molecular evidence is on the parent page (see Disease family below).
Clinical features
No curated clinical features (Orphanet) for this disease.
Identifiers
Disease identifiers
| Field | Value |
|---|---|
| Canonical name | trichosporonosis |
| Mondo ID | MONDO:0000306 |
| MeSH | D060586 |
| DOID | DOID:0050290 |
| SNOMED CT | 240761008 |
| UMLS | C0343939 |
| MedGen | 452453 |
| Is cancer (heuristic) | no |
Also known as: Trichosporon caused disease or disorder · Trichosporon disease or disorder · Trichosporon infectious disease
Disease family
An umbrella term covering 1 Mondo subtype.
Classification path: disease › human disease › disease by etiologic mechanism › disease of primarily extrinsic mechanism › infectious disease › fungal infectious disease › systemic mycosis › opportunistic systemic mycosis › trichosporonosis
Related subtypes (2): penicilliosis, disseminated candidiasis
Subtypes (1): white piedra
Genetics & variants
GWAS landscape
No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.
Variant details and genetic-evidence tiers
No tiered GWAS variants or ClinVar records for this disease.
Genes & proteins
No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).
Function
No pathway enrichment — requires an associated-gene cohort.
Therapeutics
No druggable-target or therapeutic data for this disease’s cohort.
Clinical trials & evidence
Clinical trials
Clinical trials: 0.
Related Atlas pages
No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.