Tropical endomyocardial fibrosis

disease
On this page

Also known as Davies diseaseTEMF

Summary

Tropical endomyocardial fibrosis (MONDO:0019158) is a disease. A subtype of non-familial restrictive cardiomyopathy — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Prevalence: Unknown (Worldwide) [Orphanet-validated]
  • Phenotypes (HPO): 48

Clinical features

Signs & symptoms

Clinical features (HPO)

48 HPO clinical features (Orphanet curated; top 48 by frequency):

HPO IDTermFrequency
HP:0006685Endocardial fibrosisObligate (100%)
HP:0001723Restrictive cardiomyopathyVery frequent (80-99%)
HP:0002094DyspneaVery frequent (80-99%)
HP:0003115Abnormal EKGVery frequent (80-99%)
HP:0012378FatigueVery frequent (80-99%)
HP:0001541AscitesFrequent (30-79%)
HP:0001640CardiomegalyFrequent (30-79%)
HP:0001678Atrioventricular blockFrequent (30-79%)
HP:0001945FeverFrequent (30-79%)
HP:0002240HepatomegalyFrequent (30-79%)
HP:0003073HypoalbuminemiaFrequent (30-79%)
HP:0004326CachexiaFrequent (30-79%)
HP:0004395MalnutritionFrequent (30-79%)
HP:0006677Prolonged QRS complexFrequent (30-79%)
HP:0011675ArrhythmiaFrequent (30-79%)
HP:0011712Right bundle branch blockFrequent (30-79%)
HP:0011713Left bundle branch blockFrequent (30-79%)
HP:0012249Abnormal ST segmentFrequent (30-79%)
HP:0012398Peripheral edemaFrequent (30-79%)
HP:0012764OrthopneaFrequent (30-79%)
HP:0025077Decreased QRS voltageFrequent (30-79%)
HP:0025168Left ventricular diastolic dysfunctionFrequent (30-79%)
HP:0030950Pulmonary venous hypertensionFrequent (30-79%)
HP:0031295Left atrial enlargementFrequent (30-79%)
HP:0031664Systolic heart murmurFrequent (30-79%)
HP:0033114Quadruple gallop rhythmFrequent (30-79%)
HP:0000520ProptosisOccasional (5-29%)
HP:0001653Mitral regurgitationOccasional (5-29%)
HP:0001744SplenomegalyOccasional (5-29%)
HP:0001880EosinophiliaOccasional (5-29%)
HP:0001907ThromboembolismOccasional (5-29%)
HP:0004749Atrial flutterOccasional (5-29%)
HP:0004755Supraventricular tachycardiaOccasional (5-29%)
HP:0005110Atrial fibrillationOccasional (5-29%)
HP:0005135Abnormal T-waveOccasional (5-29%)
HP:0005180Tricuspid regurgitationOccasional (5-29%)
HP:0006690Myocardial calcificationOccasional (5-29%)
HP:0010741Pedal edemaOccasional (5-29%)
HP:0011663Right ventricular cardiomyopathyOccasional (5-29%)
HP:0012664Reduced left ventricular ejection fractionOccasional (5-29%)
HP:0030057Autoimmune antibody positivityOccasional (5-29%)
HP:0030149Cardiogenic shockOccasional (5-29%)
HP:0030783Increased circulating interleukin 6 concentrationOccasional (5-29%)
HP:0030848Elevated jugular venous pressureOccasional (5-29%)
HP:0031595Abnormal P waveOccasional (5-29%)
HP:0031650Abnormal atrioventricular valve physiologyOccasional (5-29%)
HP:0005145Coronary artery stenosisVery rare (<1-4%)
HP:0031601P pulmonaleVery rare (<1-4%)

Identifiers

Disease identifiers

FieldValue
Canonical nametropical endomyocardial fibrosis
Mondo IDMONDO:0019158
Orphanet75565
SNOMED CT715626008
UMLSC2882252
MedGen909153
GARD0018928
Is cancer (heuristic)no

Also known as: Davies disease · TEMF

Disease family

Classification path: disease › human disease › disease by body system or component › musculoskeletal system disordermuscle tissue disordercardiomyopathyintrinsic cardiomyopathyrestrictive cardiomyopathy › non-familial restrictive cardiomyopathy › tropical endomyocardial fibrosis

Related subtypes (6): cardiac sarcoidosis, hypereosinophilic syndrome, wild type ATTR amyloidosis, Loeffler endocarditis, AL amyloidosis, AA amyloidosis

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.