Usher syndrome type 3B

disease
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Also known as HARS Usher syndromeUSH3BUsher syndrome caused by mutation in HARSUSHER syndrome, type IIIB

Summary

Usher syndrome type 3B (MONDO:0013788) is a disease caused by HARS1 (GenCC Strong), with 2 cohort genes.

At a glance

  • Causal gene: HARS1 (GenCC Strong)
  • Cohort genes: 2
  • ClinVar variants: 586

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical nameUsher syndrome type 3B
Mondo IDMONDO:0013788
OMIM614504
DOIDDOID:0110842
UMLSC3281066
MedGen482696
GARD0015813
Is cancer (heuristic)no

Also known as: HARS Usher syndrome · USH3B · Usher syndrome caused by mutation in HARS · USHER syndrome, type IIIB

Data availability: 586 ClinVar variants · 3 GenCC gene-disease records.

Disease family

Classification path: disease › human disease › disease by body system or component › syndromic diseaseUsher syndromeUsher syndrome type 3Usher syndrome type 3B

Related subtypes (1): Usher syndrome type 3A

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

586 retrieved; paginated sample, class counts are floors:

337 uncertain significance, 198 likely benign, 35 conflicting classifications of pathogenicity, 11 benign, 5 benign/likely benign

ClinVarVariant (HGVS)GeneClassificationReview
1039735NM_002109.6(HARS1):c.1484T>C (p.Val495Ala)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1091017NM_002109.6(HARS1):c.1445C>G (p.Thr482Arg)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1097974NM_002109.6(HARS1):c.653A>G (p.Asp218Gly)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1112786NM_002109.6(HARS1):c.615C>T (p.Gly205=)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1680306NM_002109.6(HARS1):c.1105A>G (p.Met369Val)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1680315NM_002109.6(HARS1):c.951+5G>THARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1680345NM_002109.6(HARS1):c.437_439dup (p.Asn146dup)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1680349NM_002109.6(HARS1):c.397-11T>GHARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1704893NM_002109.6(HARS1):c.192_198delinsACTG (p.Asp64_Ser66delinsGluLeu)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1705704NM_002109.6(HARS1):c.345T>A (p.Tyr115Ter)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1981675NM_002109.6(HARS1):c.58G>A (p.Gly20Ser)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
198477NM_002109.6(HARS1):c.694C>T (p.Arg232Cys)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
2528628NM_002109.6(HARS1):c.210G>A (p.Met70Ile)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
2655742NM_002109.6(HARS1):c.1163G>A (p.Arg388Gln)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
29756NM_002109.6(HARS1):c.1361A>C (p.Tyr454Ser)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
351240NM_002109.6(HARS1):c.382C>T (p.Arg128Cys)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
351242NM_002109.6(HARS1):c.103G>A (p.Val35Met)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
446299NM_002109.6(HARS1):c.464T>G (p.Val155Gly)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
472990NM_002109.6(HARS1):c.1445C>T (p.Thr482Met)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
472991NM_002109.6(HARS1):c.14C>A (p.Ala5Glu)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
540202NM_002109.6(HARS1):c.1402G>A (p.Glu468Lys)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
546982NM_002109.6(HARS1):c.90+1G>CHARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
565367NM_002109.6(HARS1):c.203G>A (p.Arg68Gln)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
576600NM_002109.6(HARS1):c.679T>G (p.Ser227Ala)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
578537NM_002109.6(HARS1):c.1372G>A (p.Ala458Thr)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
666839NM_002109.6(HARS1):c.1177G>A (p.Val393Met)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
774566NM_002109.6(HARS1):c.155A>C (p.Gln52Pro)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
800209NM_002109.6(HARS1):c.1124G>A (p.Arg375His)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
804288NM_002109.6(HARS1):c.1393A>C (p.Ile465Leu)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
846422NM_002109.6(HARS1):c.1329GAA[1] (p.Lys444del)HARS1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 8 · Orphanet: 4 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

GenCC gene–disease validity (cohort genes)

the Disease column is the GenCC-asserted condition — a cohort gene’s strongest validity may be for a related predisposition syndrome.

GeneClassificationInheritanceDiseaseRecords
HARS1StrongAutosomal recessiveUsher syndrome type 3B8

Orphanet rare-disease linkage (cohort genes)

GeneOrphanet IDRare disease
HARS1Orphanet:231183Usher syndrome type 3
HARS1Orphanet:488333Autosomal dominant Charcot-Marie-Tooth disease type 2W
HARS2Orphanet:642945Perrault syndrome type 1
HARS2Orphanet:642976Perrault syndrome type 2

Cohort genes → proteins

2 cohort genes, 2 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence2

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
HARS1HGNC:4816ENSG00000170445P12081Histidine–tRNA ligase, cytoplasmicgencc,clinvar
HARS2HGNC:4817ENSG00000112855P49590Histidine–tRNA ligase, mitochondrialclinvar

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
HARS1Histidine–tRNA ligase, cytoplasmicCatalyzes the ATP-dependent ligation of histidine to the 3’-end of its cognate tRNA, via the formation of an aminoacyl-adenylate intermediate (His-AMP).
HARS2Histidine–tRNA ligase, mitochondrialMitochondrial aminoacyl-tRNA synthetase that catalyzes the ATP-dependent ligation of histidine to the 3’-end of its cognate tRNA, via the formation of an aminoacyl-adenylate intermediate (His-AMP).

Protein-family classification

Druggable: 2 · Difficult: 0 · Unknown: 0 · Druggable fraction: 1.0

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Enzyme (other)212.0×0.007

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
HARS1Enzyme (other)yes6.1.1.21WHEP-TRS_dom, Anticodon-bd, HisRS/HisZ
HARS2Enzyme (other)yes6.1.1.21Anticodon-bd, HisRS/HisZ, aa-tRNA-synth_II

Expression context

Cohort genes with no expression data: 0.

2 cohort genes are a single-cell marker in ≥1 SCXA experiment.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)2
unknown0

Top tissues across cohort

TissueCohort genes
adenohypophysis1
lateral nuclear group of thalamus1
right frontal lobe1
cerebellar cortex1
cerebellar hemisphere1
right hemisphere of cerebellum1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
HARS1291ubiquitousmarkerlateral nuclear group of thalamus, right frontal lobe, adenohypophysis
HARS2272ubiquitousmarkercerebellar hemisphere, right hemisphere of cerebellum, cerebellar cortex

Protein interactions among cohort

Intra-cohort edges: 1.

Hub genes (top 10 by interactor count)

SymbolInteractor count
HARS22,703
HARS12,480

Intra-cohort edges

ABSources
HARS1HARS2intact

Structural data

PDB: 1 · AlphaFold-only: 1 · No structure: 0

Cohort genes with PDB structures (top 30)

SymbolUniProtPDB entries
HARS1P1208110

AlphaFold-only cohort genes (top 30 by pLDDT)

SymbolUniProtpLDDT
HARS2P4959088.44

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 5. Enrichment computed across 2 evidence-associated genes (2 with Reactome annotation).

Pathways by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 2 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

PathwayCohort genesFoldFDRSample cohort genes
Mitochondrial tRNA aminoacylation1259.6×0.011HARS2
Cytosolic tRNA aminoacylation1219.6×0.011HARS1
tRNA Aminoacylation1142.8×0.012HARS2
Translation131.0×0.040HARS2
Metabolism of proteins16.2×0.155HARS2

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 2 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
histidyl-tRNA aminoacylation28426.0×3e-08HARS1, HARS2
tRNA aminoacylation for protein translation2842.6×3e-06HARS1, HARS2
translation2102.8×1e-04HARS1, HARS2
mitochondrial translation186.9×0.011HARS2

Therapeutics

Drug target analysis

Approved (phase 4): 0 · Phase ≥3: 0 · Phased (≥1): 0 · Undrugged: 2

Druggability breadth: 1 of 2 evidence-associated genes (50%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Top cohort targets by molecule count

SymbolMoleculesMax phase
HARS100
HARS200

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 2.

Cohort genes with ChEMBL bioactivity (full, sorted by assay count)

SymbolAssaysType breakdown
HARS15Binding:5

Cohort enzymes (BRENDA EC)

SymbolEC numbersNames
HARS16.1.1.21histidine-tRNA ligase
HARS26.1.1.21histidine-tRNA ligase

Pharmacogenomics

Cohort genes with a PharmGKB record: 2; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

0 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)0
BPhased (≥1) drug, not yet approved0
CDruggable family + PDB, no drug1HARS1
DDruggable family + AlphaFold only, no drug1HARS2
EDifficult family or no structure, no drug0

Undrugged target profiles

2 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

SymbolChEMBL assaysDrugged partners (top 3)
HARS15
HARS20

Clinical trials & evidence

Clinical trials

Clinical trials: 0.