Vein disorder

Summary

Vein disorder (MONDO:0004634) is a disease (an umbrella term covering 13 Mondo subtypes) and 6 clinical trials. A subtype of vascular disorder — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

• Umbrella term: 13 Mondo subtypes
• Clinical trials: 6

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

Field	Value
Canonical name	vein disorder
Mondo ID	MONDO:0004634
DOID	DOID:866
NCIT	C35279
SNOMED CT	90507008
UMLS	C0235522
MedGen	115992
Anatomy (UBERON)	UBERON:0001638
Is cancer (heuristic)	no

Also known as: disease of vein · disease or disorder of vein · disorder of vein · vein disease · vein disease or disorder

Disease family

This is a subtype of vascular disorder. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › cardiovascular disorder › vascular disorder › vein disorder

Related subtypes (59): arterial disorder, ischemic colitis, thrombotic disease, capillary disorder, angiodysplasia, hepatic vascular disorder, vascular hemostatic disease, ischemic disease, peripheral vascular disease, venous thromboembolism, ocular vascular disorder, cholesterol embolism, thoracic outlet syndrome, idiopathic spontaneous coronary artery dissection, cerebral arteriopathy with subcortical infarcts and leukoencephalopathy, angioosteohypertrophic syndrome, Bannayan-Riley-Ruvalcaba syndrome, arterial tortuosity syndrome, hereditary arterial and articular multiple calcification syndrome, pulmonary venoocclusive disease, multiple cutaneous and mucosal venous malformations, arterial dissection-lentiginosis syndrome, patent ductus arteriosus, multisystemic smooth muscle dysfunction syndrome, STING-associated vasculopathy with onset in infancy, capillary malformation, Ehlers-Danlos syndrome, vascular-like type, calciphylaxis, neonatal Marfan syndrome, Ehlers-Danlos syndrome, vascular type, lethal arteriopathy syndrome due to fibulin-4 deficiency, congenital portosystemic shunt, arterial calcification of infancy, vasculitis, Loeys-Dietz syndrome, skin vascular disease, lymphatic malformation, familial thoracic aortic aneurysm and aortic dissection, congenital anomaly of superior vena cava, congenital anomaly of the inferior vena cava, congenital anomaly of hepatic vein, congenital renal artery stenosis, internal carotid agenesis, coronary sinus stenosis, coronary sinus atresia, vascular occlusion disorder, vascular insufficiency disorder, blood vessel neoplasm, vascular ectasia, vascular disorder of penis, fibrocartilaginous embolism, vascular malformation, lymphatic vessel neoplasm, neurovascular disorder, superior vena cava syndrome, coronary microvascular disorder, segmental arterial mediolysis, bleeding disorder, vascular-type, arterial tortuosity-bone fragility syndrome

Subtypes (13): venous insufficiency, portal vein thrombosis, occlusion of tributary of retinal vein, central retinal vein occlusion, cavernous sinus meningioma, superior vena cava angiosarcoma, pulmonary vein leiomyosarcoma, phlebitis, malignant jugulotympanic paraganglioma, varicose disease, vein of Galen aneurysm, isolated splenic vein thrombosis, isolated mesenteric vein thrombosis

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 6.

Phase distribution (across all retrieved trials)

Phase	Trials
Not specified	6

Top trials by phase / activity

NCT	Phase	Status	Title
NCT05409976	Not specified	ACTIVE_NOT_RECRUITING	The GORE® VIABAHN® FORTEGRA Venous Stent IVC Study
NCT05489588	Not specified	RECRUITING	The GORE® VIABAHN® FORTEGRA Venous Stent Iliofemoral Study
NCT02570568	Not specified	COMPLETED	Pen Torch Transillumination: Shedding Light on Difficult Venepuncture
NCT03202238	Not specified	UNKNOWN	TenTaTorch: Venepuncture Made Easy
NCT04154761	Not specified	COMPLETED	Resection of the Inferior Vena Cava Due to Tumor Involvement
NCT06231940	Not specified	COMPLETED	Efficacy of Complex Decongestive Therapy (CDT) in Patients With Venous Insufficiency: a Experimental Study