Venous thoracic outlet syndrome

disease
On this page

Also known as effort subclavian vein thrombosisPaget-Schrotter diseaseVenous cervical rib syndromeVenous costoclavicular syndromeVenous hyperabduction syndromeVenous scalenus anticus syndromeVenous thoracic outlet compression syndromeVenous TOSVTOS

Summary

Venous thoracic outlet syndrome (MONDO:0018165) is a disease. A subtype of thoracic outlet syndrome — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namevenous thoracic outlet syndrome
Mondo IDMONDO:0018165
Orphanet357131
SNOMED CT25981000119102
UMLSC1956396
MedGen408326
GARD0021536
Is cancer (heuristic)no

Also known as: effort subclavian vein thrombosis · Paget-Schrotter disease · Venous cervical rib syndrome · Venous costoclavicular syndrome · Venous hyperabduction syndrome · Venous scalenus anticus syndrome · Venous thoracic outlet compression syndrome · Venous TOS · VTOS

Disease family

This is a subtype of thoracic outlet syndrome. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › syndromic diseasethoracic outlet syndromevenous thoracic outlet syndrome

Related subtypes (2): neurogenic thoracic outlet syndrome, arterial thoracic outlet syndrome

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.