Vertebral artery occlusion

disease
On this page

Also known as occlusion and stenosis of vertebral artery

Summary

Vertebral artery occlusion (MONDO:0001258) is a disease. A subtype of occlusion precerebral artery — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namevertebral artery occlusion
Mondo IDMONDO:0001258
DOIDDOID:11299
ICD-10-CMI65.0
SNOMED CT195182007
UMLSC0265104
MedGen539080
Anatomy (UBERON)UBERON:0001535
Is cancer (heuristic)no

Also known as: occlusion and stenosis of vertebral artery

Disease family

This is a subtype of occlusion precerebral artery. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › nervous system disordercentral nervous system disorderbrain disordercerebrovascular disorderocclusion precerebral arteryvertebral artery occlusion

Related subtypes (2): basilar artery occlusion, carotid artery occlusion

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.