Vulvar seborrheic keratosis

disease
On this page

Also known as mammalian vulva seborrheic keratosisseborrheic keratosis of mammalian vulvaseborrheic keratosis of the vulvaseborrheic keratosis of vulva

Summary

Vulvar seborrheic keratosis (MONDO:0006622) is a disease. A subtype of vulvar squamous neoplasm — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namevulvar seborrheic keratosis
Mondo IDMONDO:0006622
DOIDDOID:6944
NCITC6375
UMLSC1336981
MedGen237055
GARD0024446
Anatomy (UBERON)UBERON:0000997
Is cancer (heuristic)no

Also known as: mammalian vulva seborrheic keratosis · seborrheic keratosis of mammalian vulva · seborrheic keratosis of the vulva · seborrheic keratosis of vulva

Disease family

An umbrella term covering 1 Mondo subtype.

Classification path: disease › human disease › disease by etiologic mechanism › cancer or benign tumorneoplastic disease or syndromeneoplasm › epithelial neoplasm › squamous cell neoplasm › vulvar squamous neoplasm › vulvar seborrheic keratosis

Related subtypes (3): vestibular papilloma, vulvar intraepithelial neoplasia, vulvar squamous cell carcinoma

Subtypes (1): vulvar inverted follicular keratosis

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.