Cangrelor

drug
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Also known as AR-C69931XXKengrealAdenosine triphosphate derivativeARL 69931MX

Summary

Cangrelor (CHEMBL334966) is an approved small-molecule platelet aggregation inhibitor (ATC B01AC25) targeting P2RY12, P2RY13, and GPR17; indicated across 7 conditions including thrombotic disease and myocardial infarction.

At a glance

  • Status: Approved (max clinical phase 4)
  • Modality: Small molecule
  • ATC class: B01AC25
  • Targets: 3 (P2RY12, P2RY13, GPR17)
  • Indications: 7 conditions
  • Clinical trials: 30
  • Chemistry: 776.4 Da · C17H25Cl2F3N5O12P3S2

Identifiers

Drug identity and classification

FieldValue
ChEMBL IDCHEMBL334966
NameCangrelor
TypeSmall molecule
Max phase4
FDA approvedyes
PubChem CID9854012
ChEBICHEBI:90841
ATCB01AC25
Molecular formulaC17H25Cl2F3N5O12P3S2
Molecular weight776.4
InChIKeyPAEBIVWUMLRPSK-IDTAVKCVSA-N

SMILES: CSCCNC1=C2C(=NC(=N1)SCCC(F)(F)F)N(C=N2)[C@H]3[C@@H]([C@@H]([C@H](O3)COP(=O)(O)OP(=O)(C(P(=O)(O)O)(Cl)Cl)O)O)O

IUPAC name: [dichloro-[[[(2R,3S,4R,5R)-3,4-dihydroxy-5-[6-(2-methylsulfanylethylamino)-2-(3,3,3-trifluoropropylsulfanyl)purin-9-yl]oxolan-2-yl]methoxy-hydroxyphosphoryl]oxy-hydroxyphosphoryl]methyl]phosphonic acid

ChEBI definition: A nucleoside triphosphate analogue that is 5’-O-[({dichloro(phosphono)methylphosphoryl}oxy)(hydroxy)phosphoryl]adenosine carrying additional 2-(methylsulfanyl)ethyl and (3,3,3-trifluoropropyl)sulfanyl substituents at positions N6 and C2 respectively. Used (in the form of its tetrasodium salt) as an intravenous antiplatelet drug that prevents formation of harmful blood clots in the coronary arteries.

Pharmacological roles (ChEBI): platelet aggregation inhibitor, P2Y12 receptor antagonist.

Also known as: AR-C69931XX, Cangrelor, Kengreal, Adenosine triphosphate derivative, ARL 69931MX, CANGRELOR

Parent form; salt/anhydrous children: CHEMBL1097279

Patent coverage: 363 distinct patent families (900 SureChEMBL compound mentions), from 1 matched compound structure(s). Mentions count patents naming the compound (not distinct inventions), so promiscuous / reference molecules inflate the mention figure — families are the dedup metric.

Targets

Targets

Primary targets (GtoPdb curated mechanism): the Cancer dependency column is the DepMap CRISPR fitness signal (% of screened cell lines dependent on the target).

GeneTargetActionpAffinityCancer dependencyUniProt
P2RY12P2Y12 receptorAntagonist80.5%Q9H244
P2RY13P2Y13 receptorAntagonist8.30%Q9BPV8
GPR17GPR17Antagonist8.921.6%Q13304

Broader ChEMBL bioactivity targets: 2 (assay-derived). Sample: P2Y purinoceptor 12, P2Y purinoceptor 13.

Bioactivity

ChEMBL activities: 8 potent at pChembl ≥ 5 of 8 total. Top 30 by potency (10 = 0.1 nM, 6 = 1 µM):

TargetpChemblTypeValueUnitActivity ID
P2RY129.4IC500.4nMCHEMBL_ACT_16582134
P2RY129.4IC500.4nMCHEMBL_ACT_25089906
P2RY129.4IC500.4nMCHEMBL_ACT_26024767
P2RY129.4IC500.4nMCHEMBL_ACT_70413
P2RY129.35IC500.45nMCHEMBL_ACT_24689693
P2RY128.7IC502nMCHEMBL_ACT_15642381
P2RY127.74IC5018.3nMCHEMBL_ACT_16419569
P2RY135.2EC506322nMCHEMBL_ACT_27404942

Target pathways

Aggregated over 3 target gene(s): P2RY12, P2RY13, GPR17.

Top Reactome pathways

5 total, by targets touching each:

PathwayTargetsGenes
P2Y receptors3GPR17, P2RY12, P2RY13
G alpha (i) signalling events3GPR17, P2RY12, P2RY13
Leukotriene receptors1GPR17
ADP signalling through P2Y purinoceptor 121P2RY12
G alpha (q) signalling events1GPR17

Dominant GO biological processes

GO termTargets
G protein-coupled receptor signaling pathway3
signal transduction3
G protein-coupled purinergic nucleotide receptor signaling pathway2
monoatomic ion transport1
substrate-dependent cell migration, cell extension1
adenylate cyclase-inhibiting G protein-coupled receptor signaling pathway1
phospholipase C-activating G protein-coupled receptor signaling pathway1
hemostasis1
calcium-mediated signaling1
cerebral cortex radial glia-guided migration1
cell projection organization1
lamellipodium assembly1
platelet activation1
positive regulation of integrin activation by cell surface receptor linked signal transduction1
positive regulation of cell adhesion mediated by integrin1

Indications & clinical

Indications

7 indications (3 at ChEMBL trial phase 4). Phase below is the highest clinical-trial phase recorded for this drug against each disease — not the molecule’s overall approval status (that is in the Summary).

IndicationTrial phaseMONDOEFO
thrombotic disease4MONDO:0000831HP:0004419
myocardial infarction4MONDO:0005068EFO:0000612
ischemic disease3MONDO:0005053EFO:0000556
acute coronary syndrome2MONDO:0005542EFO:0005672
ST-elevation myocardial infarction2MONDO:0041656EFO:0008585
coronary artery disorder2MONDO:0005010EFO:0001645

1 further indication record had no mapped disease name (EFO/MeSH-only) or were duplicates, and are omitted.

Clinical trials

Total trials: 30.

Phase distribution

PhaseTrials
PHASE412
PHASE27
PHASE34
Not specified4
PHASE13

Top trials by phase / activity

NCTPhaseStatusTitle
NCT06089577PHASE4RECRUITINGEffects of Cangrelor on MIcRovAscular Disfunction During Elective Percutaneous CORonary Intervention
NCT07225842PHASE4RECRUITINGThe Bridging Antiplatelet Therapy With Cangrelor 2 Study
NCT02733341PHASE4COMPLETEDThe Effect of IV Cangrelor and Oral Ticagrelor Study
NCT02943369PHASE4COMPLETEDCangrelor Following Ticagrelor Loading vs Ticagrelor Loading Alone in STEMI
NCT02978040PHASE4COMPLETEDRandomized Comparison of Cangrelor, Tirofiban and Prasugrel in Patients With STEMI Referred for Primary PCI.
NCT03043274PHASE4TERMINATEDCangrelor in ST-Elevation Myocardial Infarction to Decrease Infarct Size
NCT03247738PHASE4COMPLETEDPlatelet Inhibition With Cangrelor and Crushed Ticagrelor in STEMI
NCT03273075PHASE4UNKNOWNAdd-on Cangrelor in STEMI-triggered Cardiac Arrest
NCT03551964PHASE4COMPLETEDDual Antiplatelet Therapy For Shock Patients With Acute Myocardial Infarction
NCT04005729PHASE4COMPLETEDCangrelor in Comatose Survivors of OHCA Undergoing Primary PCI
NCT04634162PHASE4COMPLETEDPD and PK Profiles of Switching Between Cangrelor and Ticagrelor Following Ticagrelor Pre-treatment
NCT04668144PHASE4COMPLETEDPharmacodynamic and Pharmacokinetic of Switching From Cangrelor to Prasugrel in ACS Patients Undergoing PCI
NCT04667078PHASE3RECRUITINGREperfusion With P2Y12 Inhibitors in Addition to mEchanical thRombectomy for perFUsion Imaging Selected Acute Stroke patiEnts
NCT00305162PHASE3TERMINATEDA Clinical Trial to Demonstrate the Efficacy of Cangrelor
NCT00385138PHASE3TERMINATEDCangrelor Versus Standard Therapy to Achieve Optimal Management of Platelet Inhibition.
NCT01156571PHASE3COMPLETEDA Clinical Trial Comparing Cangrelor to Clopidogrel Standard Therapy in Subjects Who Require Percutaneous Coronary Intervention (PCI) (CHAMPION PHOENIX)
NCT06792643PHASE2RECRUITINGCangrelor on Top of anticoagUlation in Patients With myocaRdial Infarction-related Cardiogenic Shock/Cardiac Arrest receiVIng VA-ECMO
NCT00767507PHASE2COMPLETEDMaintenance of Platelet Inhibition With Cangrelor
NCT01766466PHASE2COMPLETEDCangrelor Ticagrelor Transition Study
NCT01852019PHASE2COMPLETEDCangrelor Prasugrel Transition Study
NCT01979445PHASE2COMPLETEDCangrelor to Clopidogrel or Prasugrel Transition Study
NCT03102723PHASE2UNKNOWNPlatelet Inhibition to Target Reperfusion Injury
NCT03862651PHASE2UNKNOWNMaintenance Of aNtiplatElet Therapy in Patients With Coronary Stenting Undergoing Surgery (MONET BRIDGE)
NCT00102674PHASE1COMPLETEDPharmacokinetics/Pharmacodynamics (PK/PD) of Cangrelor
NCT00699504PHASE1COMPLETEDAssess the Effect of Cangrelor at the Therapeutic Dose and a Supratherapeutic Dose Level on the QT/QTc Interval in Healthy Volunteers
NCT02765633PHASE1COMPLETEDCangrelor Neonatal PK/PD and Safety Study
NCT03048019Not specifiedTERMINATEDAntiplatelet Effects of Tirofiban vs. Cangrelor N-STEMI Patients Undergoing Percutaneous Coronary Intervention
NCT04138641Not specifiedCOMPLETEDDutch Cangrelor Registry
NCT04790032Not specifiedCOMPLETEDPharmacodynamic Effects of Cangrelor in ACS or CCS Patients Undergoing PCI (POMPEII Registry)
NCT05505591Not specifiedUNKNOWNIntravenous CAngrelor in High-bleeding Risk Patients Undergoing percutaneouS Coronary Intervention (ICARUS) Registry

Clinical evidence (CIViC)

No CIViC predictive evidence (expected for non-precision-medicine drugs).

Pharmacology

Pharmacogenomics

No CPIC/DPWG dosing guideline, but PharmGKB curates 2 clinical and 2 variant annotation(s) for this drug (gene-keyed; see PharmGKB).

Molecules sharing ≥1 of this drug’s curated primary targets, merged from two biobtree sources and ranked by shared-target count, then clinical phase: ChEMBL clinical-stage candidates (development phase ≥2) and PubChem drug-class bioactivity (approved / known drugs acting on the target). Deduplicated by drug name; the drug’s own salt forms are excluded. Note: for a drug with few primary targets a shared-target match can reflect off-target / promiscuous binding rather than the same therapeutic mechanism — the phase ordering surfaces bona-fide therapeutics first.

20 molecules share ≥1 primary target. Top 20 by shared-target count:

MoleculeSourceStatusShared targets
TICAGRELORChEMBL + PubChemPhase 4 (approved)P2RY12, P2RY13
ANAGRELIDEChEMBL + PubChemPhase 4 (approved)P2RY12
INDOMETHACINChEMBL + PubChemPhase 4 (approved)GPR17
MONTELUKASTChEMBL + PubChemPhase 4 (approved)GPR17
CLOPIDOGRELChEMBLPhase 4 (approved)P2RY12
PRANLUKASTChEMBLPhase 4 (approved)GPR17
PRASUGRELChEMBLPhase 4 (approved)P2RY12
SELATOGRELChEMBLPhase 3P2RY12
SURAMIN HEXASODIUMChEMBLPhase 3P2RY13
ELINOGRELChEMBLPhase 2P2RY12
GAVESTINELChEMBLPhase 2GPR17
LIXAZINONEChEMBLPhase 2P2RY12
REGRELORChEMBLPhase 2P2RY12
REGRELOR DISODIUMChEMBLPhase 2P2RY12
AspirinPubChemApprovedP2RY12
DoxorubicinPubChemApprovedP2RY12
MitoxantronePubChemApprovedP2RY12
RizatriptanPubChemApprovedGPR17
tryptophanPubChemApprovedGPR17
ZileutonPubChemApprovedGPR17