Sucralose

drug
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Also known as 1',4',6'-trichloro-galactosucrose4,1',6'-trichlorogalactosucroseAspasvitE-955INS NO.955INS-955NSC-759272San sweet sa 8020Sansweet su 100SplendaSucrazitTrichlorogalactosucroseSID144208950SID170466823

Summary

Sucralose (CHEMBL3185084) is an approved small-molecule sweetening agent; indicated across 1 condition including type 2 diabetes mellitus.

At a glance

  • Status: Approved (max clinical phase 4)
  • Modality: Small molecule
  • Indications: 1 condition
  • Clinical trials: 31
  • Chemistry: 397.6 Da · C12H19Cl3O8

Identifiers

Drug identity and classification

FieldValue
ChEMBL IDCHEMBL3185084
NameSucralose
TypeSmall molecule
Max phase3
FDA approvedyes
PubChem CID71485
ChEBICHEBI:32159
Molecular formulaC12H19Cl3O8
Molecular weight397.6
InChIKeyBAQAVOSOZGMPRM-QBMZZYIRSA-N

SMILES: C([C@@H]1[C@@H]([C@@H]([C@H]([C@H](O1)O[C@]2([C@H]([C@@H]([C@H](O2)CCl)O)O)CCl)O)O)Cl)O

IUPAC name: (2R,3R,4R,5R,6R)-2-[(2R,3S,4S,5S)-2,5-bis(chloromethyl)-3,4-dihydroxyoxolan-2-yl]oxy-5-chloro-6-(hydroxymethyl)oxane-3,4-diol

ChEBI definition: A disaccharide derivative consisting of 4-chloro-4-deoxy-α-D-galactopyranose and 1,6-dichloro-1,6-dideoxy-β-D-fructofuranose units linked by a glycosidic bond.

Pharmacological roles (ChEBI): sweetening agent.

Other ChEBI roles (chemical / environmental): environmental contaminant, xenobiotic.

Also known as: 1’,4’,6’-trichloro-galactosucrose, 4,1’,6’-trichlorogalactosucrose, Aspasvit, E-955, INS NO.955, INS-955, NSC-759272, San sweet sa 8020, Sansweet su 100, Splenda, Sucralose, Sucrazit

Patent coverage: 29,485 distinct patent families (77,857 SureChEMBL compound mentions), from 1 matched compound structure(s). Mentions count patents naming the compound (not distinct inventions), so promiscuous / reference molecules inflate the mention figure — families are the dedup metric.

Targets

Targets

Broader ChEMBL bioactivity targets: 2 (assay-derived). Sample: Carbonic anhydrase 2, Carbonic anhydrase 9.

Bioactivity

ChEMBL activities: 2 potent at pChembl ≥ 5 of 2 total. Top 30 by potency (10 = 0.1 nM, 6 = 1 µM):

TargetpChemblTypeValueUnitActivity ID
CA26.52Ki300nMCHEMBL_ACT_18407146
CA95.66Ki2200nMCHEMBL_ACT_18407150

Target pathways

No target-pathway data for this drug (no mapped target genes).

Indications & clinical

Indications

1 indication (0 at ChEMBL trial phase 4). Phase below is the highest clinical-trial phase recorded for this drug against each disease — not the molecule’s overall approval status (that is in the Summary).

IndicationTrial phaseMONDOEFO
type 2 diabetes mellitus3MONDO:0005148MONDO:0005148

Clinical trials

Total trials: 31.

Phase distribution

PhaseTrials
Not specified27
PHASE22
PHASE1/PHASE21
PHASE11

Top trials by phase / activity

NCTPhaseStatusTitle
NCT03407079PHASE2RECRUITINGEffects of Sucralose on Drug Absorption and Metabolism (The SweetMeds Study)
NCT01200940PHASE1/PHASE2COMPLETEDMetabolic Effects of Non-Nutritive Sweeteners
NCT03436277PHASE2UNKNOWNEffect of the Administration of L-Carnitine on Body Weight in Women in Crossfit Training
NCT03242434PHASE1TERMINATEDInvestigation of Thermal Injury on Intestinal Permeability in Both Thermal Injury and Healthy Participants
NCT06997133Not specifiedNOT_YET_RECRUITINGSucralose as a Way to Enhance Regulatory T Cells
NCT07124585Not specifiedNOT_YET_RECRUITINGShort-Term Effects of Sucralose and Saccharin on Blood Sugar and Gut Microbiota in Type 2 Diabetes
NCT07377097Not specifiedNOT_YET_RECRUITINGEffects of Sweetener Consumption on Risk Factors for Heart Disease in Prediabetic Subjects
NCT01128829Not specifiedCOMPLETEDSugar-replacement Sweeteners, and Blood Sugar Control
NCT02335008Not specifiedCOMPLETEDThe Effect of Artificial Sweeteners (AFS) on Sweetness Sensitivity and Preference - Pilot
NCT02335021Not specifiedCOMPLETEDThe Effect of Artificial Sweeteners (AFS) on Sweetness Sensitivity, Preference and Brain Response in Adults
NCT02413424Not specifiedCOMPLETEDMetabolic Effects of Non-nutritive Sweeteners
NCT02459535Not specifiedCOMPLETEDImmediate and Long-term Induction of Incretin Release by Artificial Sweeteners 1
NCT02499705Not specifiedTERMINATEDThe Effect of Artificial Sweeteners (AFS) on Sweetness Sensitivity, Preference and Brain Response in Adolescents
NCT02589002Not specifiedCOMPLETEDEffects of Sucralose on Glucose Metabolism
NCT02650947Not specifiedCOMPLETEDSucralose Effects on Glucose Metabolism and Gut Microbiota
NCT02745730Not specifiedCOMPLETEDRS-fMRI to Nutrient Shakes
NCT02800707Not specifiedCOMPLETEDSucralose, Stevia, Gut Microbiome and Glucose Metabolism
NCT02813759Not specifiedCOMPLETEDSucralose in Subjects With Diabetes Mellitus Insulin Requesting
NCT03247114Not specifiedCOMPLETEDfMRI of Hypothalamic Responses to Glucose, Fructose, Sucrose and Sucralose
NCT03680482Not specifiedCOMPLETEDTo Compare the Effects of Non-nutritive Sweeteners Intake in Subjects With T2DM
NCT03708939Not specifiedCOMPLETEDMicrobiome and Non-caloric Sweeteners in Humans
NCT04182464Not specifiedCOMPLETEDEffects of Sucralose in Insulin Sensitivity
NCT04185662Not specifiedUNKNOWNEffects of Artificial Sweeteners on Glucose Metabolism in Patients With Type 2 Diabetes
NCT04578431Not specifiedCOMPLETEDArtificial Sweeteners in Breast Milk
NCT04713137Not specifiedCOMPLETEDEffects of Oral Pre-loads on Subsequent Energy Intake
NCT05279183Not specifiedCOMPLETEDAssessment of Reward Responses to Erythritol Using Flavor Preference Learning
NCT05575687Not specifiedCOMPLETEDBrain and Glycemic Responses to Sweet Soft Drinks
NCT05810337Not specifiedCOMPLETEDSplenda: Effects on Blood Glucose Concentration, Appetite Scores and Subsequent Energy Intake
NCT06094894Not specifiedCOMPLETEDEffects of Sucralose in Gut Intestinal Microbiota and Postprandial GLP-1
NCT06387316Not specifiedUNKNOWNCephalic Phase Responses to Nutritive and Non-Nutritive Sweeteners
NCT07231133Not specifiedCOMPLETEDEffect of Within-Meal Consumption of Allulose on Diet-induced Thermogenesis

Clinical evidence (CIViC)

No CIViC predictive evidence (expected for non-precision-medicine drugs).

Pharmacology

Pharmacogenomics

No PharmGKB pharmacogenomic data curated for this drug.

No competitor molecules sharing a primary target (ChEMBL phase ≥2 or PubChem drug-class).