KEAP1

gene
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Also known as KIAA0132MGC10630MGC1114MGC20887MGC4407MGC9454INrf2KLHL19

Summary

KEAP1 (kelch like ECH associated protein 1, HGNC:23177) is a protein-coding gene on chromosome 19p13.2, encoding Kelch-like ECH-associated protein 1 (Q14145). Substrate-specific adapter of a BCR (BTB-CUL3-RBX1) E3 ubiquitin ligase complex that regulates the response to oxidative stress by targeting NFE2L2/NRF2 for ubiquitination. In precision oncology, KEAP1 Mutation confers sensitivity to Durvalumab Regimen + Chemotherapy + Tremelimumab in Lung Non-small Cell Carcinoma (CIViC Level A); 3 further curated variant–drug associations are listed below. It is a selective cancer dependency (DepMap: 19.4% of cell lines).

This gene encodes a cytoplasmic regulatory protein that plays a role in the cellular response to oxidative and electrophilic stress. The encoded protein regulates NRF2, a transcription factor that controls the expression of antioxidant and cytoprotective genes. Under non-stress conditions, KEAP1 binds NRF2 and promotes its ubiquitination and proteasomal degradation, thereby maintaining low NRF2 levels. During oxidative or electrophilic stress, conformational changes in KEAP1 impair its ability to promote NRF2 degradation, allowing NRF2 to activate genes that protect cells from oxidative damage and toxic insults.

Source: NCBI Gene 9817 — RefSeq curated summary.

At a glance

  • Gene–disease (curated): goiter, multinodular 1, with or without Sertoli-Leydig cell tumors (Supportive, GenCC)
  • GWAS associations: 6
  • Clinical variants (ClinVar): 80 total
  • Phenotypes (HPO): 14
  • Druggable target: yes — 9 molecules with ChEMBL bioactivity
  • Precision-oncology evidence (CIViC): 4 curated variant–drug associations
  • Cancer driver (intOGen): activating (oncogene-like) across 6 cancer types
  • Cancer dependency (DepMap): dependent in 19.4% of screened cell lines
  • MANE Select transcript: NM_203500

Identifiers

Gene identifiers

FieldValue
HGNC IDHGNC:23177
Approved symbolKEAP1
Namekelch like ECH associated protein 1
Location19p13.2
Locus typegene with protein product
StatusApproved
AliasesKIAA0132, MGC10630, MGC1114, MGC20887, MGC4407, MGC9454, INrf2, KLHL19
Ensembl geneENSG00000079999
Ensembl biotypeprotein_coding
OMIM606016
Entrez9817

Gene structure

Transcript identifiers

Ensembl transcripts: 19 — 15 protein_coding, 2 nonsense_mediated_decay, 1 protein_coding_CDS_not_defined, 1 retained_intron

ENST00000171111, ENST00000393623, ENST00000585845, ENST00000588024, ENST00000590237, ENST00000590593, ENST00000591039, ENST00000591419, ENST00000592055, ENST00000592478, ENST00000910164, ENST00000910165, ENST00000910166, ENST00000910167, ENST00000953683, ENST00000953684, ENST00000953685, ENST00000953686, ENST00000953687

RefSeq mRNA: 2 — MANE Select: NM_203500 NM_012289, NM_203500

CCDS: CCDS12239

Canonical transcript exons

ENST00000171111 — 6 exons

ExonStartEnd
ENSE000006792991048964810489853
ENSE000012172621049157710492262
ENSE000027371911050324110503356
ENSE000028670491048612510486818
ENSE000036786451048919210489368
ENSE000037991001049939510500080

Expression profiles

Bgee: expression breadth ubiquitous, 294 present calls, max score 97.75.

FANTOM5 (CAGE): breadth ubiquitous, TPM avg 13.5754 / max 140.1177, expressed in 1798 samples.

FANTOM5 promoters (4 alternative TSS)

Promoter IDTPM avgSamples expressed
1791447.09901707
1791423.29781649
1791433.09711593
1791450.081632

Top tissues by expression

300 total, by Bgee expression score (0-100, higher = more expressed):

TissueAnatomy IDExpression scoreQuality
hindlimb stylopod muscleUBERON:000425297.75gold quality
gastrocnemiusUBERON:000138897.45gold quality
muscle of legUBERON:000138397.02gold quality
apex of heartUBERON:000209896.82gold quality
muscle organUBERON:000163096.50gold quality
skeletal muscle organUBERON:001489296.50gold quality
stromal cell of endometriumCL:000225595.92gold quality
skeletal muscle tissue of rectus abdominisUBERON:000451195.92gold quality
lower esophagus mucosaUBERON:003583495.68gold quality
skeletal muscle tissueUBERON:000113495.60gold quality
quadriceps femorisUBERON:000137795.11gold quality
vastus lateralisUBERON:000137995.06gold quality
C1 segment of cervical spinal cordUBERON:000646994.70gold quality
gluteal muscleUBERON:000200094.55gold quality
skeletal muscle tissue of biceps brachiiUBERON:000450294.41gold quality
mucosa of transverse colonUBERON:000499194.17gold quality
right lobe of liverUBERON:000111494.08gold quality
esophagus mucosaUBERON:000246994.01gold quality
spinal cordUBERON:000224094.00gold quality
prefrontal cortexUBERON:000045193.98gold quality
muscle tissueUBERON:000238593.97gold quality
triceps brachiiUBERON:000150993.94gold quality
body of tongueUBERON:001187693.88gold quality
heart left ventricleUBERON:000208493.76gold quality
ventricular zoneUBERON:000305393.73gold quality
ganglionic eminenceUBERON:000402393.73gold quality
deltoidUBERON:000147693.68gold quality
olfactory segment of nasal mucosaUBERON:000538693.68gold quality
cardiac ventricleUBERON:000208293.53gold quality
esophagusUBERON:000104393.50gold quality

Single-cell (SCXA)

Detected in 1 experiment(s), a significant marker in 1.

ExperimentMarker?Max mean expression
E-ANND-3yes11.12

Regulation

Is transcription factor: no

Upstream regulators (CollecTRI, top): DNMT3A, DNMT3B, NFE2L2, NFKB, RELA, SP1, SPI1, TET1, TP53

miRNA regulators (miRDB)

30 targeting KEAP1, top 30 by miRDB confidence (max_score; target_count = how many genes the miRNA targets in total — lower means more specific):

miRNAMax scoreAvg scoremiRNA target_count
HSA-MIR-4481100.0066.421669
HSA-MIR-3065-5P99.9771.563281
HSA-LET-7C-3P99.9573.422862
HSA-MIR-141-3P99.9472.792421
HSA-MIR-200A-3P99.9472.682420
HSA-MIR-627-3P99.9071.423316
HSA-MIR-7159-5P99.5372.122472
HSA-MIR-10399-5P99.1769.872610
HSA-MIR-6504-3P99.1769.312891
HSA-MIR-429299.1665.571767
HSA-MIR-6791-5P99.1665.921844
HSA-MIR-6815-3P99.1368.981530
HSA-MIR-3117-5P99.0467.93618
HSA-MIR-7151-3P99.0469.722370
HSA-MIR-6876-3P98.9765.69765
HSA-MIR-62698.8966.21762
HSA-MIR-26B-3P98.7167.491102
HSA-MIR-423-5P98.6967.481522
HSA-MIR-3184-5P98.5667.131491
HSA-MIR-4726-3P98.4963.891385
HSA-MIR-1292-5P96.7462.14238
HSA-MIR-4529-3P96.4066.46582
HSA-MIR-6823-5P96.2665.69919
HSA-MIR-447195.1166.84755
HSA-MIR-805995.1166.30646
HSA-MIR-5009-5P94.8263.89775
HSA-MIR-805894.7663.41632
HSA-MIR-1269A92.7564.61542
HSA-MIR-1269B92.7564.73538
HSA-MIR-444988.8964.5948

Functional genomics

DepMap (CRISPR cell-line fitness): dependent in 19.4% of screened cell lines.

Literature-anchored findings (GeneRIF, showing 40)

  • In epithelial cells derived from retina and inner ear, Keap1 is a component of focal adhesions and zipper junctions. (PMID:11921171)
  • Keap1 dimerization causes Nrf2 sequestration (PMID:12145307)
  • phosphorylation of serine 40 is necessary for nuclear factor((erythroid-derived 2)like 2(Nrf2) release from Inhibitor of Nrf2(INrf2) (PMID:12947090)
  • Data show that Keap1 associates with the N-terminal region of Cullin 3 through the IVR domain and promotes the ubiquitination of Nrf2 in cooperation with the Cullin 3-Roc1 complex. (PMID:15282312)
  • hKeap1 regulates fetal Alz-50 reactive clone 1 in addition to its known role in control of Nrf2 (PMID:15379550)
  • structure of the Kelch domain of Keap1 protein represents a high quality model for the superfamily of eukaryotic kelch repeat proteins (PMID:15475350)
  • KEAP1 is a redox-regulated substrate adaptor protein for a Cul3-dependent ubiquitin ligase complex. (PMID:15572695)
  • crystallographic analysis of the Kelch domain from human Keap1 (PMID:15583386)
  • Keap1 negatively regulates Nrf2 function in part by targeting Nrf2 for ubiquitination by the CUL3-ROC1 ligase and subsequent degradation by the proteasome (PMID:15601839)
  • A downstream product of COX-2, 15-deoxy-Delta(12,14)-prostaglandin J2 (15d-PGJ2), activated the Nrf2 regulatory pathway in HAECs through binding to the cysteines of Keap1 (PMID:15917255)
  • Keap1 is a BTB-Kelch substrate adaptor protein for Cul3, which targets it for degradation by a proteasome-independent pathway (PMID:15983046)
  • stabilization of Nrf2 in cells under stress represents the central regulatory response mediated by mechanisms that interfere with its interaction with Keap1, leading to the induction of antioxidant enzymes important to maintain cellular redox homeostasis (PMID:16000310)
  • Modifying specific cysteines of the electrophile-sensing human Keap1 protein is insufficient to disrupt binding to the Nrf2 domain Neh2. (PMID:16006525)
  • These findings suggest a role for FAC1 in apoptosis following release of Nrf2 from Keap1 in response to oxidative stress. (PMID:16137655)
  • x-ray crystallographic analysis of solvent and side-chain interactions in the Kelch domain of Keap1 (PMID:16204884)
  • Somatic mutation and a gene variation in human lung cancer cells change glycine to cysteine in the KEAP1 DGR domain, introducing local conformational changes that reduce Keap1’s affinity for Nrf2. (PMID:16507366)
  • A recombinant protein containing both the Kelch/DGR domain and the C-terminal region of mouse Keap1 was expressed in Escherichia coli, purified to near-homogeneity and crystallized by the sitting-drop vapour-diffusion method. (PMID:16508120)
  • Loss of KEAP1 function leading to constitutive activation of NRF2-mediated gene expression in cancer suggests that tumor cells manipulate the NRF2 pathway for their survival against chemotherapeutic agents (PMID:17020408)
  • The N terminus of the PGAM5 protein contains a conserved NXESGE motif that binds to the substrate binding pocket in the Kelch domain of Keap1. (PMID:17046835)
  • review will discuss recent progress in the field of the Nrf2-Keap1 signaling pathway, with emphasis on the mechanistic studies of Nrf2 regulation by Keap1–{REVIEW} (PMID:17145701)
  • Nrf2-Keap1-dependent UGT1A1 induction by prooxidants might represent a key adaptive response to cellular oxidative stress (PMID:17259171)
  • In 84 Japanese type 2 diabetes subjects, 18 genetic variations were detected in the KEAP1 gene, including 13 novel ones. (PMID:17603223)
  • KEAP1 controls postinduction repression of the NRF2-mediated antioxidant response by escorting nuclear export of NRF2. (PMID:17636022)
  • Importantly, our findings suggest that a paradox exists whereby Nrf2 activity is beneficial in non-malignant cells but in cancer cells it may provide a selective advantage for clonal expansion. (PMID:17822677)
  • endogenous Keap1 remains mostly in the cytoplasm, and electrophiles promote nuclear accumulation of Nrf2 without altering the subcellular localization of Keap1 (PMID:17903176)
  • Nrf2 regulates INrf2 by controlling its transcription, and INrf2 controls Nrf2 by degrading it. (PMID:17925401)
  • C151 is the most important site of alkylation on Keap1 by chemoprevention agents that function by activating the antioxidant response element through Nrf2. (PMID:17980616)
  • in response to CSNO Keap1 accumulates in the nucleus with a time course similar to that of Nrf2. (PMID:18062931)
  • ProTalpha and Nrf2 compete for interaction with Keap1, therefore ProTalpha is able to liberate Nrf2 from complex with Keap1 and hence contribute to Nrf2-dependent transcription. (PMID:18240569)
  • Cys151 adduction confers a critical alkylation sensor function upon Keap1 (PMID:18251510)
  • Keap1 prevents activation of Nrf2 and lung cancer cell growth (PMID:18316592)
  • ap1-dependent signaling pathway for the induction of the constitutive GST A1 expression during epithelial cell differentiati in Caco-2 cells. (PMID:18476723)
  • Keap1 expression is regulated by an epigenetic mechanism in lung cancer. (PMID:18555005)
  • Aberrant Nrf2 activation provoked by Keap1 alteration is one of the molecular mechanisms for chemotherapeutic resistance in gallbladder cancer (PMID:18692501)
  • provides in vivo validation of the two-site substrate recognition model for Nrf2 activation by the Keap1-Cul3-based E3 ligase (PMID:18757741)
  • Targeting Nrf2 activity in lung cancers, particularly those with Keap1 mutations, could be a promising strategy to inhibit tumor growth and circumvent chemoresistance. (PMID:18829555)
  • KEAP1 mutations; activation of an adaptive response in cancer (Review) (PMID:19321346)
  • In renal cell carcinoma, Keap1 is associated with a more advanced disease, a higher grade and a poorer prognosis. (PMID:19424632)
  • Modification of Cys(151) of human Keap1, by mutation to a tryptophan, relieves the repression by Keap1 and allows activation of the antioxidant response element by Nrf2. (PMID:19489739)
  • Activating Nrf2 pathways in tissue-specific Keap1 knockout mice and human epithelial cells represents an important genetic approach against oxidant-induced lung damage. (PMID:19520915)

Cross-species orthologs

3 orthologs

OrganismSymbolGene ID
danio_reriokeap1bENSDARG00000074634
mus_musculusKeap1ENSMUSG00000003308
rattus_norvegicusKeap1ENSRNOG00000020878

Paralogs (54): KLHL13 (ENSG00000003096), KLHL20 (ENSG00000076321), KLHL42 (ENSG00000087448), KLHL22 (ENSG00000099910), KLHL4 (ENSG00000102271), KLHL2 (ENSG00000109466), KLHL5 (ENSG00000109790), BACH2 (ENSG00000112182), KLHL18 (ENSG00000114648), KLHL24 (ENSG00000114796), IVNS1ABP (ENSG00000116679), KLHL12 (ENSG00000117153), KLHL29 (ENSG00000119771), KBTBD7 (ENSG00000120696), KLHL7 (ENSG00000122550), KLHL31 (ENSG00000124743), KLHDC7B (ENSG00000130487), KLHL36 (ENSG00000135686), KLHL8 (ENSG00000145332), KLHL3 (ENSG00000146021), KLHL35 (ENSG00000149243), KLHL1 (ENSG00000150361), BACH1 (ENSG00000156273), KLHL40 (ENSG00000157119), KLHL10 (ENSG00000161594), KLHL21 (ENSG00000162413), KLHDC8A (ENSG00000162873), KBTBD8 (ENSG00000163376), KBTBD6 (ENSG00000165572), KLHL26 (ENSG00000167487), KLHL30 (ENSG00000168427), KBTBD2 (ENSG00000170852), KLHL6 (ENSG00000172578), KLHL15 (ENSG00000174010), KLHL38 (ENSG00000175946), KBTBD11 (ENSG00000176595), KLHDC7A (ENSG00000179023), KLHL28 (ENSG00000179454), KBTBD3 (ENSG00000182359), KLHL33 (ENSG00000185271)

Protein

Protein identifiers

Kelch-like ECH-associated protein 1Q14145 (reviewed: Q14145)

Alternative names: Cytosolic inhibitor of Nrf2, Kelch-like protein 19

All UniProt accessions (7): Q14145, K7EJ49, K7EJD8, K7EMY1, K7EPZ3, K7EQX2, K7ESE0

UniProt curated annotations — full annotation on UniProt →

Function. Substrate-specific adapter of a BCR (BTB-CUL3-RBX1) E3 ubiquitin ligase complex that regulates the response to oxidative stress by targeting NFE2L2/NRF2 for ubiquitination. KEAP1 acts as a key sensor of oxidative and electrophilic stress: in normal conditions, the BCR(KEAP1) complex mediates ubiquitination and degradation of NFE2L2/NRF2, a transcription factor regulating expression of many cytoprotective genes. In response to oxidative stress, different electrophile metabolites trigger non-enzymatic covalent modifications of highly reactive cysteine residues in KEAP1, leading to inactivate the ubiquitin ligase activity of the BCR(KEAP1) complex, promoting NFE2L2/NRF2 nuclear accumulation and expression of phase II detoxifying enzymes. In response to selective autophagy, KEAP1 is sequestered in inclusion bodies following its interaction with SQSTM1/p62, leading to inactivation of the BCR(KEAP1) complex and activation of NFE2L2/NRF2. The BCR(KEAP1) complex also mediates ubiquitination of SQSTM1/p62, increasing SQSTM1/p62 sequestering activity and degradation. The BCR(KEAP1) complex also targets BPTF and PGAM5 for ubiquitination and degradation by the proteasome.

Subunit / interactions. Component of the BCR(KEAP1) E3 ubiquitin ligase complex, at least composed of 2 molecules of CUL3, 2 molecules of KEAP1, and RBX1. Interacts with NFE2L2/NRF2; the interaction is direct. Forms a ternary complex with NFE2L2/NRF2 and PGAM5. Interacts with (phosphorylated) SQSTM1/p62; the interaction is direct and inactivates the BCR(KEAP1) complex by sequestering it in inclusion bodies, promoting its degradation. Interacts with NFE2L1. Interacts with BPTF and PTMA. Interacts with MAP1LC3B. Interacts indirectly with ENC1. Interacts with SESN1 and SESN2. Interacts with HSP90AA1 and HSP90AB1. Interacts with PGCKA1; this interaction prevents the ubiquitination of KEAP1 by TRIM25, thus protecting KEAP1 from degradation. (Microbial infection) Interacts with ebolavirus protein VP24; this interaction activates transcription factor NFE2L2/NRF2 by blocking its interaction with KEAP1.

Subcellular location. Cytoplasm. Nucleus.

Tissue specificity. Broadly expressed, with highest levels in skeletal muscle.

Post-translational modifications. Non-enzymatic covalent modifications of reactive cysteines by electrophile metabolites inactivate the BCR(KEAP1) complex. Accumulation of fumarate promotes the formation of cysteine S-succination (S-(2-succinyl)cysteine), leading to inactivate the BCR(KEAP1) complex and promote NFE2L2/NRF2 nuclear accumulation and activation. Nitric oxide-dependent 8-Nitro-cGMP formation promotes cysteine guanylation (S-cGMP-cysteine), leading to NFE2L2/NRF2 nuclear accumulation and activation. Itaconate, an anti-inflammatory metabolite generated in response to lipopolysaccharide, alkylates cysteines, activating NFE2L2/NRF2. Methylglyoxal, a reactive metabolite that accumulates when the glycolytic enzyme PGK1 is inhibited, promotes formation of a methylimidazole cross-link between proximal Cys-151 and Arg-135 on another KEAP1 molecule, resulting in an inactive dimer that inactivates the BCR(KEAP1) complex. Degraded via a proteasomal-independent process during selective autophagy: interaction with phosphorylated SQSTM1/p62 sequesters KEAP1 in inclusion bodies, leading to its degradation. Auto-ubiquitinated by the BCR(KEAP1) complex. Quinone-induced oxidative stress, but not sulforaphane, increases its ubiquitination. Ubiquitination and subsequent degradation is most pronounced following prolonged exposure of cells to oxidative stress, particularly in glutathione-deficient cells that are highly susceptible to oxidative stress. Deubiquitinated by USP25; leading to stabilization. Ubiquitinated by TRIM25; leading to degradation upon ER stress.

Activity regulation. Ubiquitin ligase activity of the BCR(KEAP1) complex is inhibited by oxidative stress and electrophile metabolites such as sulforaphane. Electrophile metabolites react with reactive cysteine residues in KEAP1 and trigger non-enzymatic covalent modifications of these cysteine residues, leading to inactivate the ubiquitin ligase activity of the BCR(KEAP1) complex. Selective autophagy also inactivates the BCR(KEAP1) complex via interaction between KEAP1 and SQSTM1/p62, which sequesters the complex in inclusion bodies and promotes its degradation.

Domain organisation. KEAP1 contains reactive cysteine residues that act as sensors for endogenously produced and exogenously encountered small molecules, which react with sulfhydryl groups and modify the cysteine sensors, leading to impair ability of the BCR(KEAP1) complex to ubiquitinate target proteins. The Kelch repeats mediate interaction with NFE2L2/NRF2, BPTF and PGAM5.

Pathway. Protein modification; protein ubiquitination.

Similarity. Belongs to the KEAP1 family.

RefSeq proteins (2): NP_036421, NP_987096* (*=MANE)

Domains & families (InterPro)

IDNameType
IPR000210BTB/POZ_domDomain
IPR006652Kelch_1Repeat
IPR011333SKP1/BTB/POZ_sfHomologous_superfamily
IPR011705BACKDomain
IPR015915Kelch-typ_b-propellerHomologous_superfamily
IPR017096BTB-kelch_proteinFamily
IPR030563KEAP1_BTB_POZ_domDomain
IPR047098KEAP1_BACKDomain

Pfam: PF00651, PF01344, PF07707, PF24681

UniProt features (116 total): strand 39, mutagenesis site 22, modified residue 12, sequence variant 10, helix 9, turn 7, repeat 6, site 5, domain 2, cross-link 2, chain 1, sequence conflict 1

Structure

Experimental structures (PDB)

122 structures, top 30 by resolution.

PDBMethodResolution (Å)
8XGKX-RAY DIFFRACTION1.32
1ZGKX-RAY DIFFRACTION1.35
8XGVX-RAY DIFFRACTION1.42
6TYMX-RAY DIFFRACTION1.42
9KVWX-RAY DIFFRACTION1.44
8IXSX-RAY DIFFRACTION1.48
2FLUX-RAY DIFFRACTION1.5
8IVRX-RAY DIFFRACTION1.5
9R1ZX-RAY DIFFRACTION1.5
8PKWX-RAY DIFFRACTION1.54
6LRZX-RAY DIFFRACTION1.54
8PKVX-RAY DIFFRACTION1.55
9M24X-RAY DIFFRACTION1.57
6V6ZX-RAY DIFFRACTION1.6
9R1CX-RAY DIFFRACTION1.69
9VD2X-RAY DIFFRACTION1.7
9IH9X-RAY DIFFRACTION1.7
8PKUX-RAY DIFFRACTION1.73
6HWSX-RAY DIFFRACTION1.75
8PKXX-RAY DIFFRACTION1.79
4IFJX-RAY DIFFRACTION1.8
4IFLX-RAY DIFFRACTION1.8
8IVGX-RAY DIFFRACTION1.8
9PHRX-RAY DIFFRACTION1.8
7EXIX-RAY DIFFRACTION1.82
1U6DX-RAY DIFFRACTION1.85
5F72X-RAY DIFFRACTION1.85
9DU7X-RAY DIFFRACTION1.87
9ETYX-RAY DIFFRACTION1.87
7K2AX-RAY DIFFRACTION1.9

Predicted structure (AlphaFold)

ModelpLDDTFraction very-high
AF-Q14145-F190.440.83

Antibody-complex structures (SAbDab): 15F72

Functional residue map

Curated UniProt residues grouped by drug-discovery relevance — catalytic, ligand-binding, modification, and mutation-validated positions. Source: UniProtKB sequence features.

Catalytic / active sites (5): 257 (sensor for electrophilic agents); 273 (sensor for electrophilic agents); 288 (sensor for electrophilic agents); 434 (sensor for electrophilic agents); 151 (sensor for electrophilic agents)

Post-translational modifications (14): 38, 151, 151, 151, 241, 257, 273, 288, 288, 319, 434, 613, 135, 151

Mutagenesis-validated functional residues (22):

PositionPhenotype
15reduced formation of a high-molecular mass keap1 molecule when methylglyoxal accumulates.
123–127abolished interaction with nfe2l2/nrf2; when associated with 161-a-a-162.
125–127increases ubiquitination and proteolytic degradation.
135reduced formation of a high-molecular mass keap1 molecule when methylglyoxal accumulates.
151substitution with a small side chain that prevents covalent modification by an electrophile; promotes constitutive ubiqu
151substitution with a bulky side chain that mimicks covalent modification by an electrophile; prevents ubiquitination and
161–162abolished interaction with nfe2l2/nrf2; when associated with 123-a–a-127.
162–164increases ubiquitination and proteolytic degradation.
273abolishes repression of nfe2l2/nrf2-dependent gene expression. slows down degradation of nfe2l2/nrf2.
288abolishes repression of nfe2l2/nrf2-dependent gene expression. slows down degradation of nfe2l2/nrf2.
308loss of export from nucleus; when associated with a-310.
310loss of export from nucleus; when associated with a-308.
334loss of interaction with nfe2l2/nrf2. strongly reduces repression of nfe2l2/nrf2-dependent gene expression. loss of inte
380loss of interaction with nfe2l2/nrf2. abolishes repression of nfe2l2/nrf2-dependent gene expression. impaired interactio
382loss of interaction with nfe2l2/nrf2. strongly reduces repression of nfe2l2/nrf2-dependent gene expression. impaired int
415loss of interaction with nfe2l2/nrf2. abolishes repression of nfe2l2/nrf2-dependent gene expression. loss of interaction
436loss of interaction with nfe2l2/nrf2. abolishes repression of nfe2l2/nrf2-dependent gene expression. does not affect int
478abolishes repression of nfe2l2/nrf2-dependent gene expression.
483loss of interaction with nfe2l2/nrf2. abolishes repression of nfe2l2/nrf2-dependent gene expression. loss of interaction
525loss of interaction with nfe2l2/nrf2. strongly reduces repression of nfe2l2/nrf2-dependent gene expression. abolishes in
572loss of interaction with nfe2l2/nrf2. strongly reduces repression of nfe2l2/nrf2-dependent gene expression. loss of inte
615decreases binding to pgcka1. increases protein half-life.

Function

Pathways and Gene Ontology

Reactome pathways

19 pathways

IDPathway
R-HSA-5689880Ub-specific processing proteases
R-HSA-8951664Neddylation
R-HSA-9679191Potential therapeutics for SARS
R-HSA-9755511KEAP1-NFE2L2 pathway
R-HSA-9759194Nuclear events mediated by NFE2L2
R-HSA-983168Antigen processing: Ubiquitination & Proteasome degradation
R-HSA-1280218Adaptive Immune System
R-HSA-1643685Disease
R-HSA-168256Immune System
R-HSA-2262752Cellular responses to stress
R-HSA-392499Metabolism of proteins
R-HSA-5663205Infectious disease
R-HSA-5688426Deubiquitination
R-HSA-597592Post-translational protein modification
R-HSA-8953897Cellular responses to stimuli
R-HSA-9679506SARS-CoV Infections
R-HSA-9711123Cellular response to chemical stress
R-HSA-9824446Viral Infection Pathways
R-HSA-983169Class I MHC mediated antigen processing & presentation

MSigDB gene sets: 0 (showing top):

GO Biological Process (14): negative regulation of transcription by RNA polymerase II (GO:0000122), in utero embryonic development (GO:0001701), ubiquitin-dependent protein catabolic process (GO:0006511), regulation of autophagy (GO:0010506), protein ubiquitination (GO:0016567), positive regulation of proteasomal ubiquitin-dependent protein catabolic process (GO:0032436), cellular response to oxidative stress (GO:0034599), proteasome-mediated ubiquitin-dependent protein catabolic process (GO:0043161), regulation of epidermal cell differentiation (GO:0045604), cellular response to interleukin-4 (GO:0071353), negative regulation of response to oxidative stress (GO:1902883), regulation of DNA-templated transcription (GO:0006355), response to oxidative stress (GO:0006979), protein K48-linked ubiquitination (GO:0070936)

GO Molecular Function (6): identical protein binding (GO:0042802), RNA polymerase II-specific DNA-binding transcription factor binding (GO:0061629), disordered domain specific binding (GO:0097718), transcription regulator inhibitor activity (GO:0140416), ubiquitin-like ligase-substrate adaptor activity (GO:1990756), protein binding (GO:0005515)

GO Cellular Component (11): nucleoplasm (GO:0005654), cytoplasm (GO:0005737), endoplasmic reticulum (GO:0005783), cytosol (GO:0005829), actin filament (GO:0005884), inclusion body (GO:0016234), midbody (GO:0030496), Cul3-RING ubiquitin ligase complex (GO:0031463), centriolar satellite (GO:0034451), nucleus (GO:0005634), protein-containing complex (GO:0032991)

Reactome top-level categories

Rollup of top-14 pathways:

CategoryPathways
Post-translational protein modification2
Deubiquitination1
SARS-CoV Infections1
Cellular response to chemical stress1
KEAP1-NFE2L2 pathway1
Class I MHC mediated antigen processing & presentation1
Immune System1
Cellular responses to stimuli1
Disease1
Metabolism of proteins1
Viral Infection Pathways1
Cellular responses to stress1
Infectious disease1
Adaptive Immune System1

GO top-level categories

Rollup of top GO terms by namespace:

CategoryTerms
cellular anatomical structure5
response to oxidative stress2
regulation of gene expression2
intracellular anatomical structure2
cytoplasm2
intracellular membrane-bounded organelle2
regulation of transcription by RNA polymerase II1
transcription by RNA polymerase II1
negative regulation of DNA-templated transcription1
chordate embryonic development1
protein ubiquitination1
modification-dependent protein catabolic process1
autophagy1
regulation of catabolic process1
protein modification by small protein conjugation1
regulation of proteasomal ubiquitin-dependent protein catabolic process1
proteasome-mediated ubiquitin-dependent protein catabolic process1
positive regulation of proteasomal protein catabolic process1
positive regulation of ubiquitin-dependent protein catabolic process1
cellular response to chemical stress1
ubiquitin-dependent protein catabolic process1
proteasomal protein catabolic process1
epidermal cell differentiation1
regulation of epithelial cell differentiation1
regulation of epidermis development1
response to interleukin-41
cellular response to cytokine stimulus1
negative regulation of response to stimulus1
regulation of response to oxidative stress1
DNA-templated transcription1
regulation of RNA biosynthetic process1
response to stress1
protein polyubiquitination1
protein binding1
DNA-binding transcription factor binding1
protein domain specific binding1
transcription regulator activity1
molecular function inhibitor activity1
enzyme-substrate adaptor activity1
binding1

Protein interactions and networks

STRING

3612 interactions, top by confidence (×1000):

Protein AProtein BPartner UniProtScore
KEAP1NEIL2Q969S2999
KEAP1NFE2L2Q16236999
KEAP1CUL3Q13618998
KEAP1RBX1P62877996
KEAP1SQSTM1Q13501995
KEAP1PGAM5Q96HS1993
KEAP1IKBKBO14920983
KEAP1PALB2Q86YC2962
KEAP1NQO1P15559954
KEAP1PTMAP06454936
KEAP1HMOX1P09601934
KEAP1GCLMP48507932
KEAP1RELAQ04206932
KEAP1DPP3Q9NY33919
KEAP1PARK7Q99497914

IntAct

507 interactions, top by confidence:

ABTypeScore
NFE2L2KEAP1psi-mi:“MI:0915”(physical association)0.980
KEAP1NFE2L2psi-mi:“MI:0915”(physical association)0.980
KEAP1NFE2L2psi-mi:“MI:0407”(direct interaction)0.980
NFE2L2KEAP1psi-mi:“MI:0914”(association)0.980
KEAP1NFE2L2psi-mi:“MI:0914”(association)0.980
SQSTM1KEAP1psi-mi:“MI:0915”(physical association)0.970
KEAP1SQSTM1psi-mi:“MI:0915”(physical association)0.970
KEAP1SQSTM1psi-mi:“MI:0407”(direct interaction)0.970
KEAP1DPP3psi-mi:“MI:0915”(physical association)0.910
DPP3KEAP1psi-mi:“MI:0915”(physical association)0.910
KEAP1ETF1psi-mi:“MI:0915”(physical association)0.830
MAD2L1KEAP1psi-mi:“MI:0915”(physical association)0.830
KEAP1MAD2L1psi-mi:“MI:0915”(physical association)0.830
KEAP1WDR83psi-mi:“MI:0915”(physical association)0.780

BioGRID (903): NFE2L2 (Biochemical Activity), KEAP1 (Affinity Capture-MS), KEAP1 (Two-hybrid), KEAP1 (Two-hybrid), KEAP1 (Two-hybrid), KEAP1 (Two-hybrid), KEAP1 (Two-hybrid), KEAP1 (Two-hybrid), KEAP1 (Two-hybrid), KEAP1 (Two-hybrid), DPP3 (Two-hybrid), NUDT4 (Two-hybrid), MAPKBP1 (Two-hybrid), KLHL3 (Two-hybrid), LSM3 (Two-hybrid)

ESM2 similar proteins: A0A2R8Q1W5, A6QQY2, B0WWP2, B3NDN0, B4HIK1, B4L0G9, B4LIG6, B4PD06, D3Z8N4, E0CZ16, E1B932, E7F6F9, E9Q4F2, F1LZ52, O94889, P28575, P57790, P59280, Q08DK3, Q14145, Q16RL8, Q2T9Z7, Q53G59, Q5R774, Q5R7B8, Q5U374, Q5ZKD9, Q5ZLD3, Q684M4, Q6DFF6, Q6JEL2, Q6JEL3, Q6NRH0, Q6TDP3, Q6TDP4, Q6ZPT1, Q7QGL0, Q80TF4, Q8BZM0, Q8K430

Diamond homologs: A0A2R8Q1W5, A9JRD8, B0WWP2, B3DIV9, B3M9V8, B3NDN0, B4GRJ2, B4HIK1, B4J045, B4L0G9, B4LIG6, B4MXW3, B4PD06, B4QLQ2, D3Z8N4, D3ZUU2, E0CZ16, E1B932, E7F6F9, E9Q4F2, F1LZ52, F1LZF0, F1MBP6, G3X9X1, O15062, O93567, O94889, O95198, P28575, P57790, Q04652, Q08DK3, Q13105, Q14145, Q16RL8, Q1ECZ2, Q2M0J9, Q2TBA0, Q3SWU4, Q3ZB90

SIGNOR signaling

13 interactions.

AEffectBMechanism
KEAP1down-regulatesNFE2L2binding
KEAP1“down-regulates quantity”NFE2L2ubiquitination
DPP3“down-regulates quantity by destabilization”KEAP1cleavage
BAP1“up-regulates quantity by stabilization”KEAP1binding
KEAP1“form complex”CUL3-RBX1-KEAP1binding
“hydrogen peroxide”“up-regulates activity”KEAP1“chemical modification”
SQSTM1“down-regulates quantity by destabilization”KEAP1ubiquitination
KEAP1“up-regulates activity”CUL3binding
KEAP1“down-regulates quantity by destabilization”PGAM5binding
KEAP1“up-regulates activity”“Cullin 3-RBX1-Skp1”binding

Enriched among interaction partners

Reactome pathways and GO biological processes over-represented among this gene’s 117 IntAct physical interaction partners (hypergeometric vs the genome-wide background, BH-FDR, gene-set size 15–500, ranked by fold). A functional readout of the neighbourhood — distinct from this gene’s own memberships above, and biased toward well-studied / hub proteins, so read it as themes rather than proof.

Reactome pathways:

PathwayPartnersFoldFDR
KEAP1-NFE2L2 pathway1117.4×2e-08
Cellular response to chemical stress713.2×3e-04
Neddylation95.6×4e-03
Cellular responses to stress104.8×4e-03
Cellular responses to stimuli114.5×4e-03

GO biological processes:

GO termPartnersFoldFDR
cellular response to amino acid stimulus516.5×5e-03

Disease & clinical

Cancer significance

From intOGen — cancer-driver classification: activating (oncogene-like) across 6 cancer types — CHOL, HCC, LUAD, LUSC, NPC, NSCLC.

Clinical variants and AI predictions

ClinVar

80 variants total. Per-class counts are floors (≥ shown; pagination cap):

ClassificationCount (floor)
Pathogenic0
Likely pathogenic0
Uncertain significance49
Likely benign9
Benign1

Top pathogenic / likely-pathogenic (0)

SpliceAI

838 predictions. Top by Δscore:

VariantEffectΔscore
19:10486814:GCCTC:Gacceptor_gain1.0000
19:10486815:CCTCC:Cacceptor_gain1.0000
19:10486816:CTC:Cacceptor_gain1.0000
19:10486817:TC:Tacceptor_gain1.0000
19:10486818:CC:Cacceptor_gain1.0000
19:10486818:CCT:Cacceptor_loss1.0000
19:10486819:C:CAacceptor_loss1.0000
19:10486819:C:CCacceptor_gain1.0000
19:10486820:T:Gacceptor_loss1.0000
19:10486822:C:CTacceptor_gain1.0000
19:10486823:G:Tacceptor_gain1.0000
19:10489188:TCACC:Tdonor_loss1.0000
19:10489189:CACCA:Cdonor_loss1.0000
19:10489190:ACCAA:Adonor_loss1.0000
19:10489209:C:Adonor_gain1.0000
19:10489366:CGC:Cacceptor_gain1.0000
19:10489367:GCC:Gacceptor_loss1.0000
19:10489368:CCTAA:Cacceptor_loss1.0000
19:10489369:C:Aacceptor_loss1.0000
19:10489369:C:CCacceptor_gain1.0000
19:10489370:T:Gacceptor_loss1.0000
19:10489658:T:TAdonor_gain1.0000
19:10491572:CTCA:Cdonor_loss1.0000
19:10491573:TCACC:Tdonor_loss1.0000
19:10491574:CACCT:Cdonor_loss1.0000
19:10491575:A:ACdonor_gain1.0000
19:10491575:ACCTC:Adonor_loss1.0000
19:10491576:C:CCdonor_gain1.0000
19:10491576:CCT:Cdonor_gain1.0000
19:10499396:T:TAdonor_gain1.0000

AlphaMissense

4110 scored. Top likely-pathogenic:

VariantProtein changeam_pathogenicity
19:10486719:C:TG603E1.000
19:10486756:A:GW591R1.000
19:10486756:A:TW591R1.000
19:10486796:G:CF577L1.000
19:10486796:G:TF577L1.000
19:10486798:A:GF577L1.000
19:10486815:C:AG571V1.000
19:10486815:C:TG571D1.000
19:10486818:C:AG570V1.000
19:10486818:C:TG570E1.000
19:10489332:C:TG523E1.000
19:10489333:C:AG523W1.000
19:10489653:C:TG509E1.000
19:10489688:C:AW497C1.000
19:10489688:C:GW497C1.000
19:10489690:A:GW497R1.000
19:10489690:A:TW497R1.000
19:10489745:A:CF478L1.000
19:10489745:A:TF478L1.000
19:10489747:A:GF478L1.000
19:10489749:C:TG477D1.000
19:10489752:C:TG476E1.000
19:10489753:C:AG476W1.000
19:10489794:C:TG462E1.000
19:10489795:C:AG462W1.000
19:10489799:C:AR460S1.000
19:10489799:C:GR460S1.000
19:10489800:C:AR460M1.000
19:10489800:C:GR460T1.000
19:10489831:A:GW450R1.000

dbSNP variants (sampled 300 via entrez): RS1000081810 (19:10504656 G>C), RS1000115630 (19:10496793 C>G,T), RS1000177106 (19:10504982 T>C), RS1000277602 (19:10498182 G>A), RS1000416320 (19:10485993 C>T), RS1000433910 (19:10490980 A>G), RS1000653506 (19:10493123 G>A,C), RS1000764295 (19:10492600 C>G), RS1000787528 (19:10485773 G>C), RS1000818506 (19:10493061 T>A), RS1000943167 (19:10492800 TAG>T), RS1001151960 (19:10487358 A>G), RS1001155321 (19:10496208 AAAAAAGGCCAGGTGC>A), RS1001332821 (19:10488281 C>G), RS1001397882 (19:10487228 C>A)

Disease associations

OMIM: gene MIM:606016 | disease phenotypes:

GenCC curated gene-disease

DiseaseClassificationInheritance
goiter, multinodular 1, with or without Sertoli-Leydig cell tumorsSupportiveAutosomal dominant

Mondo (1): goiter, multinodular 1, with or without Sertoli-Leydig cell tumors (MONDO:0007681)

Orphanet (0):

HPO phenotypes

14 total (14 of 14 shown, HPO-id order):

HPOTerm
HP:0000836Hyperthyroidism
HP:0002671Basal cell carcinoma
HP:0002890Thyroid carcinoma
HP:0005584Renal cell carcinoma
HP:0005987Multinodular goiter
HP:0006779Alveolar rhabdomyosarcoma
HP:0007129Cerebellar medulloblastoma
HP:0030071Medulloepithelioma
HP:0030434Pilomatrixoma
HP:0100528Pleuropulmonary blastoma
HP:0100615Ovarian neoplasm
HP:0100617Testicular seminoma
HP:0100619Sertoli cell neoplasm
HP:0200063Colorectal polyposis

GWAS associations

6 associations (top):

StudyTraitp-value
GCST004131_88Inflammatory bowel disease2.000000e-11
GCST004132_111Crohn’s disease3.000000e-13
GCST005537_48Chronic inflammatory diseases (ankylosing spondylitis, Crohn’s disease, psoriasis, primary sclerosing cholangitis, ulcerative colitis) (pleiotropy)6.000000e-09
GCST006430_8Body mass index and waist-hip ratio (pleiotropy)5.000000e-06
GCST012190_13Body mass index and diastolic blood pressure (bivariate analysis)5.000000e-06
GCST012191_13Body mass index and systolic blood pressure (bivariate analysis)5.000000e-06

EFO canonical traits (4, from GWAS)

EFO IDTrait name
EFO:0004340body mass index
EFO:0004343waist-hip ratio
EFO:0006336diastolic blood pressure
EFO:0006335systolic blood pressure

MeSH disease descriptors (1)

DescriptorNameTree numbers
C562732Euthyroid Goiter (supp.)

Drugs & pharmacology

Drug and pharmacology data

Is drug target: yes

ChEMBL targets (7): CHEMBL2069156 (SINGLE PROTEIN), CHEMBL3038498 (PROTEIN-PROTEIN INTERACTION), CHEMBL4106129 (PROTEIN-PROTEIN INTERACTION), CHEMBL4296122 (PROTEIN-PROTEIN INTERACTION), CHEMBL4296123 (PROTEIN-PROTEIN INTERACTION), CHEMBL6193832 (PROTEIN-PROTEIN INTERACTION), CHEMBL6193838 (PROTEIN-PROTEIN INTERACTION)

Molecules with ChEMBL bioactivity

9 molecules (phase ≥1), by development phase (incl. off-target/promiscuous compounds). Patent mentions across the top 20 by phase: 391,615 (via chembl_molecule»patent_compound — counts attach to the compound, not the gene–compound relationship, so off-target/promiscuous molecules can dominate).

MoleculeNamePhasePatents
CHEMBL1200969DUTASTERIDE411,156
CHEMBL157101KETOCONAZOLE475,361
CHEMBL1607TOPOTECAN HYDROCHLORIDE456,379
CHEMBL2107333DIMETHYL FUMARATE422,969
CHEMBL359744DOXORUBICIN HYDROCHLORIDE4141,917
CHEMBL603ZAFIRLUKAST423,220
CHEMBL932DIPYRIDAMOLE451,743
CHEMBL48802SULFORAPHANE37,981
CHEMBL1093059BARDOXOLONE2889

Clinical evidence (CIViC)

Drug × variant × indication: 4 predictive associations from 4 curated evidence items.

VariantTherapyIndicationEffectLevelCIViC
KEAP1 MutationDurvalumab Regimen + Chemotherapy + TremelimumabLung Non-small Cell CarcinomaSensitivity/ResponseCIViC AEID12419
KEAP1 MutationCisplatin/Pembrolizumab/Pemetrexed RegimenLung Non-small Cell CarcinomaResistanceCIViC BEID12339
NFE2L2 Mutation OR KEAP1 Mutation OR CUL3 MutationPlatinum DoubletLung Non-small Cell CarcinomaResistanceCIViC BEID12558
KEAP1 MutationNSC84167Lung Non-small Cell CarcinomaSensitivity/ResponseCIViC DEID12575

PharmGKB: 1 entry (VIP=true, CPIC=false)

GtoPdb / IUPHAR curated pharmacology

(IUPHAR/BPS Guide to Pharmacology — expert-curated)

Target class: other protein — Kelch-like proteins

Most potent curated ligand interactions (7 total), top 7:

LigandActionAffinityParameter
biKEAP1Binding8.07pIC50
compound 10 [PMID: 31411465]Binding7.72pKd
Nrf2 peptide [PMID: 23647822]Binding7.48pIC50
CPUY192018Binding7.4pKd
bardoxoloneBinding7.0pIC50
isoquinoline 17 [PMID: 30122040]Binding6.99pKd
RA839Binding5.22pKd

Binding affinities (BindingDB)

372 measured of 462 human assays (462 total across all organisms); most potent 50 below. Values come from heterogeneous assays and are not directly comparable.

LigandMeasureValuePatent
3-[7-[[(4R)-8-chloro-4-ethyl-1,1-dioxo-3,4-dihydropyrido[2,3-b][1,4,5]oxathiazepin-2-yl]methyl]-1-benzothiophen-5-yl]-3-(4-chloro-1-methylbenzotriazol-5-yl)propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
(3R)-3-[7-[[(4R)-8-chloro-4-ethyl-1,1-dioxo-3,4-dihydropyrido[2,3-b][1,4,5]oxathiazepin-2-yl]methyl]-2,3-dihydro-1H-inden-5-yl]-3-(1,4-dimethylbenzotriazol-5-yl)propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
3-(1,4-dimethylbenzotriazol-5-yl)-3-[7-[[(4R)-7-ethoxy-4-ethyl-1,1-dioxo-3,4-dihydropyrido[2,3-b][1,4,5]oxathiazepin-2-yl]methyl]-2,3-dihydro-1H-inden-5-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
3-(1,4-dimethylbenzotriazol-5-yl)-3-[7-[[(4R)-4-ethyl-8-methyl-1,1-dioxo-3,4-dihydropyrido[2,3-b][1,4,5]oxathiazepin-2-yl]methyl]-2,3-dihydro-1H-inden-5-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
3-(1,4-dimethylbenzotriazol-5-yl)-3-[7-[[(4R)-4-ethyl-7-methyl-1,1-dioxo-3,4-dihydropyrido[2,3-b][1,4,5]oxathiazepin-2-yl]methyl]-2,3-dihydro-1H-inden-5-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
3-[7-[[(4R)-8-chloro-4-ethyl-1,1-dioxo-3,4-dihydro-5,1lambda6,2-benzoxathiazepin-2-yl]methyl]-2,3-dihydro-1H-inden-5-yl]-3-(1,4-dimethylbenzotriazol-5-yl)propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
3-(1,4-dimethylbenzotriazol-5-yl)-3-[7-[[(4R)-7-ethoxy-4-ethyl-1,1-dioxo-3,4-dihydropyrido[2,3-b][1,4,5]oxathiazepin-2-yl]methyl]-1-benzothiophen-5-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
(3R)-3-[7-[[(4R)-8-chloro-4-ethyl-7-fluoro-1,1-dioxo-3,4-dihydro-5,1lambda6,2-benzoxathiazepin-2-yl]methyl]-2,3-dihydro-1H-inden-5-yl]-3-(1,4-dimethylbenzotriazol-5-yl)propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
3-(1,4-dimethylbenzotriazol-5-yl)-3-[7-[[(4R)-4-ethyl-1,1-dioxo-3,4-dihydropyrido[2,3-b][1,4,5]oxathiazepin-2-yl]methyl]-2,3-dihydro-1H-inden-5-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
3-(1,4-dimethylbenzotriazol-5-yl)-3-[7-[[(4R)-4-ethyl-1,1-dioxo-8-(trifluoromethyl)-3,4-dihydropyrido[2,3-b][1,4,5]oxathiazepin-2-yl]methyl]-2,3-dihydro-1H-inden-5-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
3-(1,4-dimethylbenzotriazol-5-yl)-3-[7-[(4,4-dimethyl-1,1-dioxo-3H-pyrido[2,3-b][1,4,5]oxathiazepin-2-yl)methyl]-2,3-dihydro-1H-inden-5-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
3-(1,4-dimethylbenzotriazol-5-yl)-3-[7-[(1,1-dioxospiro[3H-pyrido[2,3-b][1,4,5]oxathiazepine-4,1’-cyclobutane]-2-yl)methyl]-2,3-dihydro-1H-inden-5-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
3-(1,4-dimethylbenzotriazol-5-yl)-3-[7-[(1,1-dioxospiro[3H-pyrido[2,3-b][1,4,5]oxathiazepine-4,3’-oxetane]-2-yl)methyl]-2,3-dihydro-1H-inden-5-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
(3R)-3-[7-[[(4R)-8-chloro-4-ethyl-1,1-dioxo-3,4-dihydropyrido[2,3-b][1,4,5]oxathiazepin-2-yl]methyl]-2,3-dihydro-1H-inden-5-yl]-3-(7-cyclopropyl-1,4-dimethylbenzotriazol-5-yl)propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
(3S)-3-[7-[[(4R)-8-chloro-4-ethyl-1,1-dioxo-3,4-dihydropyrido[2,3-b][1,4,5]oxathiazepin-2-yl]methyl]-2,3-dihydro-1H-inden-5-yl]-3-(7-cyclopropyl-1,4-dimethylbenzotriazol-5-yl)propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
(3S)-3-[7-[[(4R)-8-chloro-4-ethyl-1,1-dioxo-3,4-dihydropyrido[2,3-b][1,4,5]oxathiazepin-2-yl]methyl]-1-benzothiophen-5-yl]-3-[1,4-dimethyl-7-(trifluoromethoxy)benzotriazol-5-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
3-[7-[[(4R)-8-chloro-4-ethyl-1,1-dioxo-3,4-dihydropyrido[2,3-b][1,4,5]oxathiazepin-2-yl]methyl]-1-benzothiophen-5-yl]-3-(7-methoxy-1,4-dimethylbenzotriazol-5-yl)propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
3-(7-chloro-1,4-dimethylbenzotriazol-5-yl)-3-[7-[[(4R)-8-chloro-4-ethyl-1,1-dioxo-3,4-dihydropyrido[2,3-b][1,4,5]oxathiazepin-2-yl]methyl]-1-benzothiophen-5-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
3-[7-[[(4R)-8-chloro-4-ethyl-1,1-dioxo-3,4-dihydropyrido[2,3-b][1,4,5]oxathiazepin-2-yl]methyl]-1-benzothiophen-5-yl]-3-(7-cyano-1,4-dimethylbenzotriazol-5-yl)propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
3-[7-[[(4R)-8-chloro-4-ethyl-1,1-dioxo-3,4-dihydropyrido[2,3-b][1,4,5]oxathiazepin-2-yl]methyl]-1-benzothiophen-5-yl]-3-[1,4-dimethyl-7-(trifluoromethyl)benzotriazol-5-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
3-[7-[[(4R)-8-chloro-4-ethyl-1,1-dioxo-3,4-dihydropyrido[2,3-b][1,4,5]oxathiazepin-2-yl]methyl]-1-benzothiophen-5-yl]-3-[1,4-dimethyl-7-(trifluoromethoxy)benzotriazol-5-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
3-[7-[[(4R)-8-chloro-4-ethyl-1,1-dioxo-3,4-dihydropyrido[2,3-b][1,4,5]oxathiazepin-2-yl]methyl]-1-benzothiophen-5-yl]-3-[7-(difluoromethoxy)-1,4-dimethylbenzotriazol-5-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
3-[7-[[(4R)-8-chloro-4-ethyl-1,1-dioxo-3,4-dihydropyrido[2,3-b][1,4,5]oxathiazepin-2-yl]methyl]-1-benzothiophen-5-yl]-3-[1-(2-hydroxy-2-methylpropyl)-4-methylbenzotriazol-5-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
ethyl 3-[7-[[(4R)-8-chloro-4-ethyl-1,1-dioxo-3,4-dihydropyrido[2,3-b][1,4,5]oxathiazepin-2-yl]methyl]-1-benzothiophen-5-yl]-3-[4-methyl-1-(3-methylsulfonylpropyl)benzotriazol-5-yl]propanoateIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
3-[7-[[(4R)-8-chloro-4-ethyl-1,1-dioxo-3,4-dihydropyrido[2,3-b][1,4,5]oxathiazepin-2-yl]methyl]-1-benzothiophen-5-yl]-3-(3,7-dimethyltriazolo[4,5-b]pyridin-6-yl)propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
3-[7-[[(4R)-8-chloro-4-ethyl-1,1-dioxo-3,4-dihydropyrido[2,3-b][1,4,5]oxathiazepin-2-yl]methyl]-1-benzothiophen-5-yl]-3-[4-methyl-1-(2,2,2-trifluoroethyl)benzotriazol-5-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
3-(1,4-dimethylbenzotriazol-5-yl)-3-[7-[[(2R,5S)-2-ethyl-5-methyl-3,5-dihydro-2H-pyrido[2,3-f][1,4]oxazepin-4-yl]methyl]-1-benzothiophen-5-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
(3R)-3-(1,4-dimethylbenzotriazol-5-yl)-3-[7-[[(2R,5S)-2-ethyl-5-methyl-3,5-dihydro-2H-pyrido[2,3-f][1,4]oxazepin-4-yl]methyl]-1-benzothiophen-5-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
(3R)-3-(1,4-dimethylbenzotriazol-5-yl)-3-[7-[[(2R,5S)-2-ethyl-5-methyl-3,5-dihydro-2H-pyrido[2,3-f][1,4]oxazepin-4-yl]methyl]-2,3-dihydro-1H-inden-5-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
ethyl (3S)-3-(1,4-dimethylbenzotriazol-5-yl)-3-[7-[[(2R,5S)-2-ethyl-5-methyl-3,5-dihydro-2H-pyrido[2,3-f][1,4]oxazepin-4-yl]methyl]-2,3-dihydro-1H-inden-5-yl]propanoateIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
(3R)-3-(1,4-dimethyl-2,3-dihydrobenzotriazol-5-yl)-3-[7-[[(2R)-2-ethyl-5-methyl-7-oxo-2,3,5,6-tetrahydropyrido[2,3-f][1,4]oxazepin-4-yl]methyl]-1-benzothiophen-5-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
(3R)-3-(1,4-dimethyl-2,3-dihydrobenzotriazol-5-yl)-3-[7-[[(2R,5S)-2,5-dimethyl-7-oxo-2,3,5,6-tetrahydropyrido[2,3-f][1,4]oxazepin-4-yl]methyl]-1-benzothiophen-5-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
(3R)-3-(3,7-dimethyl-1,2-dihydrotriazolo[4,5-b]pyridin-6-yl)-3-[7-[[(2R)-2-ethyl-5-methyl-7-oxo-2,3,5,6-tetrahydropyrido[2,3-f][1,4]oxazepin-4-yl]methyl]-1-benzothiophen-5-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
ethyl (3R)-3-(1,4-dimethyl-2,3-dihydrobenzotriazol-5-yl)-3-[7-[[(2R,5S)-2,5-dimethyl-7-oxo-2,3,5,6-tetrahydropyrido[2,3-f][1,4]oxazepin-4-yl]methyl]-1-benzothiophen-5-yl]propanoateIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
ethyl (3R)-3-[7-[[(2R)-2-cyclopropyl-5-methyl-7-oxo-2,3,5,6-tetrahydropyrido[2,3-f][1,4]oxazepin-4-yl]methyl]-1-benzothiophen-5-yl]-3-(3,7-dimethyl-1,2-dihydrotriazolo[4,5-b]pyridin-6-yl)propanoateIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
(3R)-3-(1,4-dimethylbenzotriazol-5-yl)-3-[7-[[(5R)-5-methyl-7-oxospiro[5,6-dihydro-3H-pyrido[2,3-f][1,4]oxazepine-2,1’-cyclopropane]-4-yl]methyl]-1-benzothiophen-5-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
3-(1,4-dimethylbenzotriazol-5-yl)-3-[4-[[(4R)-4-methyl-1,1-dioxo-3,4-dihydro-5,1lambda6,2-benzoxathiazepin-2-yl]methyl]-1H-indol-6-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
3-[7-[[(4R)-8-chloro-4-ethyl-1,1-dioxo-3,4-dihydropyrido[2,3-b][1,4,5]oxathiazepin-2-yl]methyl]-2,3-dihydro-1-benzofuran-5-yl]-3-(1,4-dimethylbenzotriazol-5-yl)propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
3-[7-[[(4R)-8-chloro-4-ethyl-1,1-dioxo-3,4-dihydropyrido[2,3-b][1,4,5]oxathiazepin-2-yl]methyl]-1-benzofuran-5-yl]-3-(4-chloro-1-methylbenzotriazol-5-yl)propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
tert-butyl 6-[1-(7-cyclopropyl-1,4-dimethylbenzotriazol-5-yl)-3-ethoxy-3-oxopropyl]-4-[[(2R,5S)-2-ethyl-5-methyl-3,5-dihydro-2H-pyrido[2,3-f][1,4]oxazepin-4-yl]methyl]indole-1-carboxylateIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
(3R)-3-(7-cyclopropyl-1,4-dimethylbenzotriazol-5-yl)-3-[7-[[(2R,5S)-2-ethyl-5-methyl-3,5-dihydro-2H-pyrido[2,3-f][1,4]oxazepin-4-yl]methyl]-2,3-dihydro-1H-inden-5-yl]propanoic acidIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
ethyl 3-(7-cyclopropyl-1,4-dimethylbenzotriazol-5-yl)-3-[7-[[(2R,5S)-2-ethyl-5-methyl-3,5-dihydro-2H-pyrido[2,3-f][1,4]oxazepin-4-yl]methyl]-2,3-dihydro-1-benzofuran-5-yl]propanoateIC5020 nMUS-20250092064: BENZOTRIAZOLE COMPOUND
(3S)-3-(1,4-dimethylbenzotriazol-5-yl)-3-[7-[[(2R)-2-ethyl-7-oxo-2,3,5,6-tetrahydropyrido[2,3-f][1,4]oxazepin-4-yl]methyl]-1-benzothiophen-5-yl]propanoic acidIC5020 nMUS-20250296939: BENZOTHIOPHENE COMPOUND
(3S)-3-(1,4-dimethylbenzotriazol-5-yl)-3-[7-[(7-oxospiro[5,6-dihydro-3H-pyrido[2,3-f][1,4]oxazepine-2,1’-cyclopropane]-4-yl)methyl]-1-benzothiophen-5-yl]propanoic acidIC5020 nMUS-20250296939: BENZOTHIOPHENE COMPOUND
(3R)-3-(1,4-dimethylbenzotriazol-5-yl)-3-[7-[(7-oxospiro[5,6-dihydro-3H-pyrido[2,3-f][1,4]oxazepine-2,1’-cyclopropane]-4-yl)methyl]-1-benzothiophen-5-yl]propanoic acidIC5020 nMUS-20250296939: BENZOTHIOPHENE COMPOUND
(3R)-3-[7-[[(2R)-2-cyclopropyl-7-oxo-2,3,5,6-tetrahydropyrido[2,3-f][1,4]oxazepin-4-yl]methyl]-1-benzothiophen-5-yl]-3-(1,4-dimethylbenzotriazol-5-yl)propanoic acidIC5020 nMUS-20250296939: BENZOTHIOPHENE COMPOUND
(3S)-3-[7-[[(2R)-2-cyclopropyl-7-oxo-2,3,5,6-tetrahydropyrido[2,3-f][1,4]oxazepin-4-yl]methyl]-1-benzothiophen-5-yl]-3-(1,4-dimethylbenzotriazol-5-yl)propanoic acidIC5020 nMUS-20250296939: BENZOTHIOPHENE COMPOUND
(3R)-3-[7-[[(2R)-2-cyclopropyl-7-oxo-2,3,5,6-tetrahydropyrido[2,3-f][1,4]oxazepin-4-yl]methyl]-1-benzothiophen-5-yl]-3-(3,7-dimethyltriazolo[4,5-b]pyridin-6-yl)propanoic acidIC5020 nMUS-20250296939: BENZOTHIOPHENE COMPOUND
(3S)-3-(1,4-dimethylbenzotriazol-5-yl)-2,2-dimethyl-3-[7-[(7-oxospiro[5,6-dihydro-3H-pyrido[2,3-f][1,4]oxazepine-2,1’-cyclopropane]-4-yl)methyl]-1-benzothiophen-5-yl]propanoic acidIC5020 nMUS-20250296939: BENZOTHIOPHENE COMPOUND

ChEMBL bioactivities

1612 potent at pChembl≥5 of 1964 total, top 50 by pChembl (potency: 10 = 0.1 nM, 6 = 1 µM).

pChemblTypeValueUnitMolecule
11.00Kd0.01nMCHEMBL5759229
10.62Kd0.024nMCHEMBL5886931
10.39Kd0.041nMCHEMBL6052915
10.38Kd0.042nMCHEMBL6036229
10.15Kd0.07nMCHEMBL4644995
10.10Kd0.079nMCHEMBL5945962
10.09Kd0.081nMCHEMBL5997320
10.09Kd0.081nMCHEMBL5920236
10.05Kd0.089nMCHEMBL5893955
9.92Kd0.12nMCHEMBL5996288
9.90Kd0.127nMCHEMBL5915606
9.80Kd0.16nMCHEMBL5789875
9.79Kd0.162nMCHEMBL5765112
9.76Kd0.174nMCHEMBL5881215
9.73Kd0.188nMCHEMBL5823251
9.62Kd0.24nMCHEMBL5756145
9.58Kd0.264nMCHEMBL5818820
9.56Kd0.273nMCHEMBL5956739
9.56Kd0.278nMCHEMBL6033425
9.47Kd0.34nMCHEMBL5964184
9.38Kd0.42nMCHEMBL6000717
9.29Kd0.518nMCHEMBL5815214
9.15Kd0.7nMCHEMBL4643134
9.06Kd0.88nMCHEMBL4633657
9.01Kd0.97nMCHEMBL6054981
9.00Ki1nMCHEMBL3819587
9.00Kd1nMCHEMBL5567793
9.00Ki1nMCHEMBL3237245
9.00Kd1nMCHEMBL5998600
8.99Kd1.02nMCHEMBL5904165
8.97Kd1.08nMCHEMBL5966649
8.96Kd1.1nMCHEMBL5824789
8.96Kd1.1nMCHEMBL5864191
8.96Kd1.09nMCHEMBL5966874
8.96Kd1.1nMCHEMBL5838878
8.92Kd1.2nMCHEMBL5748784
8.92Kd1.2nMCHEMBL5783798
8.92Kd1.2nMCHEMBL6025266
8.92Kd1.19nMCHEMBL5776238
8.92Kd1.19nMCHEMBL5948517
8.92Kd1.19nMCHEMBL6022430
8.91Kd1.24nMCHEMBL6065860
8.90Kd1.26nMCHEMBL5859607
8.89Kd1.3nMCHEMBL3819587
8.89Kd1.3nMCHEMBL5757170
8.89Kd1.28nMCHEMBL5900035
8.89Kd1.29nMCHEMBL5747464
8.85Kd1.4nMCHEMBL3819587
8.85Kd1.4nMCHEMBL5806480
8.85Kd1.4nMCHEMBL5971865

PubChem BioAssay actives

989 with measured affinity, of 3094 total; 50 most potent distinct compounds. Largely complementary to BindingDB; screening values are coarse (µM, 4 dp), so sub-nM hits tie at the floor.

CompoundAssayTypeValueUnit
(2S,3S)-3-(1-ethyl-4-methylbenzotriazol-5-yl)-2-methyl-3-[2-(2,3,5,6-tetramethylbenzoyl)-3,4-dihydro-1H-isoquinolin-7-yl]propanoic acid1651179: Binding affinity to Keap1 (unknown origin) by SPR assaykd0.0001uM
2-[[4-[carboxymethyl-(4-methoxyphenyl)sulfonylamino]-3-[3-(1,3-dioxoisoindol-2-yl)propyl]naphthalen-1-yl]-(4-methoxyphenyl)sulfonylamino]acetic acid2008554: Inhibition of Keap1-Nrf2 protein-protein interaction (unknown origin) preincubated for 30 mins followed by Terbium labeled anti-His antibody addition for 30 mins prior to fluorescein labeled-9mer Nrf2 peptide addition and measured after 60 mins by TR-FRET assayki0.0001uM
(3S)-1-[4-[(2,3,5,6-tetramethylphenyl)sulfonylamino]naphthalen-1-yl]pyrrolidine-3-carboxylic acid1676325: Inhibition of FITC-9mer Nrf2 peptide interaction with human recombinant Keap1 Kelch domain (321 to 609 residues) expressed in Escherichia coli DE3 cells incubated for 30 mins by fluorescence polarization assayic500.0001uM
(3S)-3-(1-ethyl-4-methylbenzotriazol-5-yl)-3-[2-(2,3,5,6-tetramethylbenzoyl)-3,4-dihydro-1H-isoquinolin-7-yl]propanoic acid1651179: Binding affinity to Keap1 (unknown origin) by SPR assaykd0.0007uM
(2S)-2-[(S)-(1-ethyl-4-methylbenzotriazol-5-yl)-[2-(2,3,5,6-tetramethylbenzoyl)-3,4-dihydro-1H-isoquinolin-7-yl]methyl]butanoic acid1651179: Binding affinity to Keap1 (unknown origin) by SPR assaykd0.0009uM
(3S)-3-(7-methoxy-1-methylbenzotriazol-5-yl)-3-[4-methyl-3-[[(4R)-4-methyl-1,1-dioxo-3,4-dihydro-5,1lambda6,2-benzoxathiazepin-2-yl]methyl]phenyl]propanoic acid1577815: Inhibition of Cys5-LDEETGEFL-NH2 binding to recombinant human KEAP1 Kelch domain (321 to 609 residues) expressed in Escherichia coli BL21 (DE3) measured after 10 to 15 mins by fluorescence polarization assayki0.0010uM
(2R,3S)-3-[3-[(1-cyanocyclopropyl)methoxy]-4-methylphenyl]-3-[[(2S)-2-fluoro-2-(4-methylphenyl)acetyl]amino]-2-methylpropanoic acid2080975: Binding affinity to biotinylated Keap1 Kelch domain (unknown origin) by SPR methodkd0.0010uM
2-[[4-[carboxymethyl-(4-methoxyphenyl)sulfonylamino]naphthalen-1-yl]-(4-methoxyphenyl)sulfonylamino]acetic acid2088407: Inhibition of Keap1-Nrf2 protein-protein interaction (unknown origin) assessed as inhibition constant by TR-FRET assayki0.0010uM
2-[[4-[carboxymethyl-(4-methoxyphenyl)sulfonylamino]-3-phenylsulfanylnaphthalen-1-yl]-(4-methoxyphenyl)sulfonylamino]acetic acid2008554: Inhibition of Keap1-Nrf2 protein-protein interaction (unknown origin) preincubated for 30 mins followed by Terbium labeled anti-His antibody addition for 30 mins prior to fluorescein labeled-9mer Nrf2 peptide addition and measured after 60 mins by TR-FRET assayki0.0015uM
(2R,3S)-3-(1-ethyl-4-methylbenzotriazol-5-yl)-2-methyl-3-[2-(2,3,5,6-tetramethylbenzoyl)-3,4-dihydro-1H-isoquinolin-7-yl]propanoic acid1651179: Binding affinity to Keap1 (unknown origin) by SPR assaykd0.0019uM
2-[[3-(carboxymethoxy)-4-[carboxymethyl-(4-methylphenyl)sulfonylamino]naphthalen-1-yl]-(4-methylphenyl)sulfonylamino]acetic acid2088408: Inhibition of Keap1-Nrf2 protein-protein interaction (unknown origin) assessed as inhibition constant by fluorescence polarization assayki0.0019uM
3-[5-fluoro-3-(1-methylpyrazol-4-yl)-2-pyridinyl]-3-methoxy-5,5-dimethyl-6-oxocyclohexene-1-carbonitrile1511440: Inhibition of KEAP1 interaction with Nrf2 in human HEK293 cells transfected with ARE-LUC assessed as activation of Nrf2 signaling pathway measured after 18 hrs by steady-Glo luciferase assayec500.0020uM
2-[[4-[carboxymethyl-(4-methoxyphenyl)sulfonylamino]-3-(3-morpholin-4-yl-3-oxopropyl)naphthalen-1-yl]-(4-methoxyphenyl)sulfonylamino]acetic acid2008554: Inhibition of Keap1-Nrf2 protein-protein interaction (unknown origin) preincubated for 30 mins followed by Terbium labeled anti-His antibody addition for 30 mins prior to fluorescein labeled-9mer Nrf2 peptide addition and measured after 60 mins by TR-FRET assayki0.0020uM
(3S)-3-[5-fluoro-3-(1,2-thiazol-5-yl)-2-pyridinyl]-3-methoxy-5,5-dimethyl-6-oxocyclohexene-1-carbonitrile1511440: Inhibition of KEAP1 interaction with Nrf2 in human HEK293 cells transfected with ARE-LUC assessed as activation of Nrf2 signaling pathway measured after 18 hrs by steady-Glo luciferase assayec500.0020uM
1-(4-ethoxyphenyl)sulfonyl-5-[(4-ethoxyphenyl)sulfonylamino]-N-hydroxy-2-methylbenzo[g]indole-3-carboxamide1577815: Inhibition of Cys5-LDEETGEFL-NH2 binding to recombinant human KEAP1 Kelch domain (321 to 609 residues) expressed in Escherichia coli BL21 (DE3) measured after 10 to 15 mins by fluorescence polarization assayki0.0021uM
2-[[4-[carboxymethyl-(4-methoxyphenyl)sulfonylamino]-3-[4-(1,3-dioxoisoindol-2-yl)phenyl]naphthalen-1-yl]-(4-methoxyphenyl)sulfonylamino]acetic acid2008554: Inhibition of Keap1-Nrf2 protein-protein interaction (unknown origin) preincubated for 30 mins followed by Terbium labeled anti-His antibody addition for 30 mins prior to fluorescein labeled-9mer Nrf2 peptide addition and measured after 60 mins by TR-FRET assayki0.0021uM
2-[[4-[carboxymethyl-(4-methoxyphenyl)sulfonylamino]-3-phenylnaphthalen-1-yl]-(4-methoxyphenyl)sulfonylamino]acetic acid2008554: Inhibition of Keap1-Nrf2 protein-protein interaction (unknown origin) preincubated for 30 mins followed by Terbium labeled anti-His antibody addition for 30 mins prior to fluorescein labeled-9mer Nrf2 peptide addition and measured after 60 mins by TR-FRET assayki0.0024uM
8,8-dimethyl-4-(4-methyl-3-pyridinyl)-9-oxo-1-oxa-4-azaspiro[5.5]undec-10-ene-10-carbonitrile1828011: Modulation of Keap1-Nrf2 protein-protein interaction in human U2OS cells assessed as inhibition of Keap1 by PathHunter assayec500.0025uM
(3S)-3-(2-benzoyl-3,4-dihydro-1H-isoquinolin-7-yl)-3-(1-ethyl-4-methylbenzotriazol-5-yl)propanoic acid1651179: Binding affinity to Keap1 (unknown origin) by SPR assaykd0.0027uM
(7R,10S,16S,19S,22S,25S,28S)-28-amino-10,19-bis(2-carboxyethyl)-25-(carboxymethyl)-16-[(1R)-1-hydroxyethyl]-22-(hydroxymethyl)-9,12,15,18,21,24,27-heptaoxo-2-oxa-5-thia-8,11,14,17,20,23,26-heptazabicyclo[28.2.2]tetratriaconta-1(32),30,33-triene-7-carboxylic acid1856128: Inhibition of Keap1 (unknown origin) by ELISA analysisic500.0028uM
6-chloro-5’,5’-dimethyl-6’-oxospiro[1H-2-benzofuran-3,3’-cyclohexene]-1’-carbonitrile1828011: Modulation of Keap1-Nrf2 protein-protein interaction in human U2OS cells assessed as inhibition of Keap1 by PathHunter assayec500.0029uM
3-[(4S,7S,11S,13S,16S,19S,25S,28S)-4-[[(2S)-2-[[(2S)-2-[[(2S)-2-[[(2S)-5-amino-2-[[(2S)-2-[[(2S)-2-[[(2S)-6-amino-2-[[(2S)-6-amino-2-[[(2S)-2-[[2-[[(2S)-2-amino-3-(4-hydroxyphenyl)propanoyl]amino]acetyl]amino]-5-carbamimidamidopentanoyl]amino]hexanoyl]amino]hexanoyl]amino]-5-carbamimidamidopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-5-oxopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-28-carbamoyl-16-(2-carboxyethyl)-7-(carboxymethyl)-11-fluoro-19-[(1R)-1-hydroxyethyl]-5,8,14,17,20,23,26-heptaoxo-1,6,9,15,18,21,24,27,31,32-decazatricyclo[28.2.1.09,13]tritriaconta-30(33),31-dien-25-yl]propanoic acid1677710: Binding affinity to human KEAP1 Kelch domain (322 to 609 residues) incubated for 60 mins by TR-FRET assayic500.0030uM
(2R,3S)-3-[[(2S)-2-[4-[(3-ethoxy-2-pyridinyl)methyl]phenyl]-2-fluoroacetyl]amino]-2-methyl-3-(4-methylphenyl)propanoic acid2080975: Binding affinity to biotinylated Keap1 Kelch domain (unknown origin) by SPR methodkd0.0030uM
2-[[4-[carboxymethyl-(4-methoxyphenyl)sulfonylamino]-3-(4-cyanophenyl)naphthalen-1-yl]-(4-methoxyphenyl)sulfonylamino]acetic acid2008554: Inhibition of Keap1-Nrf2 protein-protein interaction (unknown origin) preincubated for 30 mins followed by Terbium labeled anti-His antibody addition for 30 mins prior to fluorescein labeled-9mer Nrf2 peptide addition and measured after 60 mins by TR-FRET assayki0.0031uM
8,8-dimethyl-9-oxo-4-pyridin-3-yl-1-oxa-4-azaspiro[5.5]undec-10-ene-10-carbonitrile1828011: Modulation of Keap1-Nrf2 protein-protein interaction in human U2OS cells assessed as inhibition of Keap1 by PathHunter assayec500.0031uM
3-[(3S,6S,12S,15S,21S,24S,27S,29S)-6-[[(2S)-2-[[(2S)-2-[[(2S)-2-[[(2S)-5-amino-2-[[(2S)-2-[[(2S)-2-[[(2S)-6-amino-2-[[(2S)-6-amino-2-[[(2S)-2-[[2-[[(2S)-2-amino-3-(4-hydroxyphenyl)propanoyl]amino]acetyl]amino]-5-carbamimidamidopentanoyl]amino]hexanoyl]amino]hexanoyl]amino]-5-carbamimidamidopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-5-oxopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-12-carbamoyl-24-(2-carboxyethyl)-3-(carboxymethyl)-29-fluoro-21-[(1R)-1-hydroxyethyl]-2,5,9,14,17,20,23,26-octaoxo-1,4,10,13,16,19,22,25-octazabicyclo[25.3.0]triacontan-15-yl]propanoic acid1677710: Binding affinity to human KEAP1 Kelch domain (322 to 609 residues) incubated for 60 mins by TR-FRET assayic500.0031uM
4-(3-fluoro-4-pyridinyl)-8,8-dimethyl-9-oxo-1-oxa-4-azaspiro[5.5]undec-10-ene-10-carbonitrile1828011: Modulation of Keap1-Nrf2 protein-protein interaction in human U2OS cells assessed as inhibition of Keap1 by PathHunter assayec500.0032uM
3-[(3S,6S,12S,15S,21S,24S,27S,29S)-6-[[(2S)-2-[[(2S)-2-[[(2S)-2-[[(2S)-2-[[(2S)-2-[[(2S)-2-[[(2S)-2-[[(2S)-2-[[(2S)-2-amino-5-carbamimidamidopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-12-carbamoyl-24-(2-carboxyethyl)-3-(carboxymethyl)-29-fluoro-21-[(1R)-1-hydroxyethyl]-2,5,9,14,17,20,23,26-octaoxo-1,4,10,13,16,19,22,25-octazabicyclo[25.3.0]triacontan-15-yl]propanoic acid1677710: Binding affinity to human KEAP1 Kelch domain (322 to 609 residues) incubated for 60 mins by TR-FRET assayic500.0034uM
3-[(4S,7S,11S,13S,16S,19S,25S,28S)-4-[[2-[[(2S)-2-[[(2S)-1-[(2S)-2-[[(2S)-2-[[(2R)-2-[[(2S)-2-amino-4-methylpentanoyl]amino]-3-sulfanylpropanoyl]amino]-4-methylpentanoyl]amino]-5-(diaminomethylideneamino)pentanoyl]pyrrolidine-2-carbonyl]amino]-3-methylbutanoyl]amino]acetyl]amino]-28-carbamoyl-16-(2-carboxyethyl)-7-(carboxymethyl)-11-fluoro-19-[(1R)-1-hydroxyethyl]-5,8,14,17,20,23,26-heptaoxo-1,6,9,15,18,21,24,27,31,32-decazatricyclo[28.2.1.09,13]tritriaconta-30(33),31-dien-25-yl]propanoic acid1677710: Binding affinity to human KEAP1 Kelch domain (322 to 609 residues) incubated for 60 mins by TR-FRET assayic500.0036uM
2-[[3-(3-amino-3-oxopropyl)-4-[carboxymethyl-(4-methoxyphenyl)sulfonylamino]naphthalen-1-yl]-(4-methoxyphenyl)sulfonylamino]acetic acid2008554: Inhibition of Keap1-Nrf2 protein-protein interaction (unknown origin) preincubated for 30 mins followed by Terbium labeled anti-His antibody addition for 30 mins prior to fluorescein labeled-9mer Nrf2 peptide addition and measured after 60 mins by TR-FRET assayki0.0042uM
3-[(4S,7S,11S,13S,16S,19S,25S,28S)-4-[[(2S)-6-amino-2-[[(2S)-6-amino-2-[[(2S)-2-[[(2S)-6-amino-2-[[(2S)-2-[[(2S)-2-[[(2S)-2-[[(2S)-4-amino-2-[[(2S)-5-amino-2-[[(2S)-2-[[(2S)-2-[[(2S,3S)-2-[[(2S)-6-amino-2-[[(2S,3S)-2-[[(2S)-5-amino-2-[[(2S)-2-amino-5-carbamimidamidopentanoyl]amino]-5-oxopentanoyl]amino]-3-methylpentanoyl]amino]hexanoyl]amino]-3-methylpentanoyl]amino]-3-(1H-indol-3-yl)propanoyl]amino]-3-phenylpropanoyl]amino]-5-oxopentanoyl]amino]-4-oxobutanoyl]amino]-5-carbamimidamidopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-4-methylsulfanylbutanoyl]amino]hexanoyl]amino]-3-(1H-indol-3-yl)propanoyl]amino]hexanoyl]amino]hexanoyl]amino]-28-carbamoyl-16-(2-carboxyethyl)-7-(carboxymethyl)-11-fluoro-19-[(1R)-1-hydroxyethyl]-5,8,14,17,20,23,26-heptaoxo-1,6,9,15,18,21,24,27,31,32-decazatricyclo[28.2.1.09,13]tritriaconta-30(33),31-dien-25-yl]propanoic acid1677710: Binding affinity to human KEAP1 Kelch domain (322 to 609 residues) incubated for 60 mins by TR-FRET assayic500.0042uM
8,8-dimethyl-4-(5-methyl-3-pyridinyl)-9-oxo-1-oxa-4-azaspiro[5.5]undec-10-ene-10-carbonitrile1828011: Modulation of Keap1-Nrf2 protein-protein interaction in human U2OS cells assessed as inhibition of Keap1 by PathHunter assayec500.0045uM
2-[[4-[carboxymethyl-(4-methoxyphenyl)sulfonylamino]-3-[3-oxo-3-(propan-2-ylamino)propyl]naphthalen-1-yl]-(4-methoxyphenyl)sulfonylamino]acetic acid2008554: Inhibition of Keap1-Nrf2 protein-protein interaction (unknown origin) preincubated for 30 mins followed by Terbium labeled anti-His antibody addition for 30 mins prior to fluorescein labeled-9mer Nrf2 peptide addition and measured after 60 mins by TR-FRET assayki0.0046uM
3-[(4S,7S,11S,13S,16S,19S,25S,28S)-4-[[(2S)-2-[[(2S)-5-amino-2-[[2-[[(2S)-5-carbamimidamido-2-[[(2S)-2-[[(2S)-2-[[(2S)-5-carbamimidamido-2-[[(2S)-2-[[(2S)-2-[[(2S,3S)-2-[[(2S)-2-[(2S)-2-carbamoylpyrrolidin-1-yl]-4-methylpentanoyl]amino]-3-methylpentanoyl]amino]-4-methylpentanoyl]amino]-4-methylpentanoyl]amino]pentanoyl]amino]-4-methylpentanoyl]amino]-4-methylpentanoyl]amino]pentanoyl]amino]acetyl]amino]-5-oxopentanoyl]amino]-3-phenylpropanoyl]amino]-28-carbamoyl-16-(2-carboxyethyl)-7-(carboxymethyl)-11-fluoro-19-[(1R)-1-hydroxyethyl]-5,8,14,17,20,23,26-heptaoxo-1,6,9,15,18,21,24,27,31,32-decazatricyclo[28.2.1.09,13]tritriaconta-30(33),31-dien-25-yl]propanoic acid1677710: Binding affinity to human KEAP1 Kelch domain (322 to 609 residues) incubated for 60 mins by TR-FRET assayic500.0047uM
3-[(3S,6S,12S,15S,21S,24S,27S,29S)-6-[[(2S)-6-amino-2-[[(2S)-6-amino-2-[[(2S)-2-[[(2S)-6-amino-2-[[(2S)-2-[[(2S)-2-[[(2S)-2-[[(2S)-4-amino-2-[[(2S)-5-amino-2-[[(2S)-2-[[(2S)-2-[[(2S,3S)-2-[[(2S)-6-amino-2-[[(2S,3S)-2-[[(2S)-5-amino-2-[[(2S)-2-amino-5-carbamimidamidopentanoyl]amino]-5-oxopentanoyl]amino]-3-methylpentanoyl]amino]hexanoyl]amino]-3-methylpentanoyl]amino]-3-(1H-indol-3-yl)propanoyl]amino]-3-phenylpropanoyl]amino]-5-oxopentanoyl]amino]-4-oxobutanoyl]amino]-5-carbamimidamidopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-4-methylsulfanylbutanoyl]amino]hexanoyl]amino]-3-(1H-indol-3-yl)propanoyl]amino]hexanoyl]amino]hexanoyl]amino]-12-carbamoyl-24-(2-carboxyethyl)-3-(carboxymethyl)-29-fluoro-21-[(1R)-1-hydroxyethyl]-2,5,9,14,17,20,23,26-octaoxo-1,4,10,13,16,19,22,25-octazabicyclo[25.3.0]triacontan-15-yl]propanoic acid1677710: Binding affinity to human KEAP1 Kelch domain (322 to 609 residues) incubated for 60 mins by TR-FRET assayic500.0048uM
3-[(3S,6S,12S,15S,21S,24S,27S,29S)-6-[[2-[[(2S)-2-[[(2S)-1-[(2S)-2-[[(2S)-2-[[(2R)-2-[[(2S)-2-amino-4-methylpentanoyl]amino]-3-sulfanylpropanoyl]amino]-4-methylpentanoyl]amino]-5-(diaminomethylideneamino)pentanoyl]pyrrolidine-2-carbonyl]amino]-3-methylbutanoyl]amino]acetyl]amino]-12-carbamoyl-24-(2-carboxyethyl)-3-(carboxymethyl)-29-fluoro-21-[(1R)-1-hydroxyethyl]-2,5,9,14,17,20,23,26-octaoxo-1,4,10,13,16,19,22,25-octazabicyclo[25.3.0]triacontan-15-yl]propanoic acid1677710: Binding affinity to human KEAP1 Kelch domain (322 to 609 residues) incubated for 60 mins by TR-FRET assayic500.0048uM
2-[[4-[carboxymethyl-(4-methoxyphenyl)sulfonylamino]-3-(4-methoxyphenyl)naphthalen-1-yl]-(4-methoxyphenyl)sulfonylamino]acetic acid2008554: Inhibition of Keap1-Nrf2 protein-protein interaction (unknown origin) preincubated for 30 mins followed by Terbium labeled anti-His antibody addition for 30 mins prior to fluorescein labeled-9mer Nrf2 peptide addition and measured after 60 mins by TR-FRET assayki0.0049uM
(2S)-2-[[2-[[(2S,3R)-2-[[(2S)-2-amino-4-carboxybutanoyl]amino]-3-hydroxybutanoyl]amino]acetyl]amino]pentanedioic acid1204220: Binding affinity to Keap1/Nrf2 complex (unknown origin) by ITC methodkd0.0050uM
8,8-dimethyl-9-oxo-4-pyridin-2-yl-1-oxa-4-azaspiro[5.5]undec-10-ene-10-carbonitrile1828011: Modulation of Keap1-Nrf2 protein-protein interaction in human U2OS cells assessed as inhibition of Keap1 by PathHunter assayec500.0058uM
3-(3-bromo-5-fluoro-2-pyridinyl)-3-methoxy-5,5-dimethyl-6-oxocyclohexene-1-carbonitrile1511440: Inhibition of KEAP1 interaction with Nrf2 in human HEK293 cells transfected with ARE-LUC assessed as activation of Nrf2 signaling pathway measured after 18 hrs by steady-Glo luciferase assayec500.0060uM
(2R,3S)-3-[[(2S)-2-(4-chlorophenyl)-2-fluoroacetyl]amino]-3-[3-(2-cyano-2-methylpropoxy)-4-methylphenyl]-2-methylpropanoic acid2080975: Binding affinity to biotinylated Keap1 Kelch domain (unknown origin) by SPR methodkd0.0060uM
3-[(4R,7S,13S,16S,19S,25S,28R)-4-acetamido-28-carbamoyl-16-(2-carboxyethyl)-7-(carboxymethyl)-32,33,34,35-tetrafluoro-19-[(1R)-1-hydroxyethyl]-5,8,14,17,20,23,26-heptaoxo-2,30-dithia-6,9,15,18,21,24,27-heptazatricyclo[29.2.2.09,13]pentatriaconta-1(34),31(35),32-trien-25-yl]propanoic acid1394077: Displacement of F-14 from Keap1 Kelch domain (unknown origin) expressed in Escherichia coli BL21 (DE3) after 15 mins by fluorescence polarization assayki0.0061uM
3-[(3S,6S,9S,15S,18S,21S,27S,30S,33S)-9-(3-amino-3-oxopropyl)-18-benzyl-30-(2-carboxyethyl)-3-(carboxymethyl)-27-[(1R)-1-hydroxyethyl]-6,15-bis(2-methylpropyl)-2,5,8,11,14,17,20,23,26,29,32-undecaoxo-1,4,7,10,13,16,19,22,25,28,31-undecazabicyclo[31.3.0]hexatriacontan-21-yl]propanoic acid1493694: Binding affinity to human biotin-labeled Keap1 Kelch domain (322 to 609 residues) expressed in Escherichia coli (DE3) by biolayer interferometrykd0.0062uM
2-[3-(carboxymethoxy)-4-[carboxymethyl-(4-methylphenyl)sulfonylamino]-N-(4-methylphenyl)sulfonylanilino]acetic acid2088408: Inhibition of Keap1-Nrf2 protein-protein interaction (unknown origin) assessed as inhibition constant by fluorescence polarization assayki0.0064uM
3-[5-fluoro-3-(2-methyl-1,3-thiazol-5-yl)-2-pyridinyl]-3-methoxy-5,5-dimethyl-6-oxocyclohexene-1-carbonitrile1511440: Inhibition of KEAP1 interaction with Nrf2 in human HEK293 cells transfected with ARE-LUC assessed as activation of Nrf2 signaling pathway measured after 18 hrs by steady-Glo luciferase assayec500.0070uM
N-[4-[2,3-dihydro-1,4-benzodioxin-6-ylsulfonyl(2H-tetrazol-5-ylmethyl)amino]naphthalen-1-yl]-N-(2H-tetrazol-5-ylmethyl)-2,3-dihydro-1,4-benzodioxine-6-sulfonamide1755203: Inhibition of recombinant human KEAP1 Kelch domain (321 to 609 residues) expressed in Escherichia coli BL21 (DE3)pLysS cells interaction with FITC-9mer Nrf2 (unknown origin) incubated for 30 mins under shaking condition by TR-FRET assayic500.0072uM
3-[(4S,7S,11S,13S,16S,19S,25S,28S)-4-[[(2S)-2-[[(2S)-2-[[(2S)-2-[[(2S)-2-[[(2S)-2-[[(2S)-2-[[(2S)-2-[[(2S)-2-[[(2S)-2-amino-5-carbamimidamidopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-5-carbamimidamidopentanoyl]amino]-28-carbamoyl-16-(2-carboxyethyl)-7-(carboxymethyl)-11-fluoro-19-[(1R)-1-hydroxyethyl]-5,8,14,17,20,23,26-heptaoxo-1,6,9,15,18,21,24,27,31,32-decazatricyclo[28.2.1.09,13]tritriaconta-30(33),31-dien-25-yl]propanoic acid1677710: Binding affinity to human KEAP1 Kelch domain (322 to 609 residues) incubated for 60 mins by TR-FRET assayic500.0074uM
3-[(3S,6S,12S,15S,21S,24S,27S,29S)-6-[[(2S)-2-[[(2S)-5-amino-2-[[2-[[(2S)-5-carbamimidamido-2-[[(2S)-2-[[(2S)-2-[[(2S)-5-carbamimidamido-2-[[(2S)-2-[[(2S)-2-[[(2S,3S)-2-[[(2S)-2-[[(2S)-2-carbamoylpyrrolidine-1-carbonyl]amino]-4-methylpentanoyl]amino]-3-methylpentanoyl]amino]-4-methylpentanoyl]amino]-4-methylpentanoyl]amino]pentanoyl]amino]-4-methylpentanoyl]amino]-4-methylpentanoyl]amino]pentanoyl]amino]acetyl]amino]-5-oxopentanoyl]amino]-3-phenylpropanoyl]amino]-12-carbamoyl-24-(2-carboxyethyl)-3-(carboxymethyl)-29-fluoro-21-[(1R)-1-hydroxyethyl]-2,5,9,14,17,20,23,26-octaoxo-1,4,10,13,16,19,22,25-octazabicyclo[25.3.0]triacontan-15-yl]propanoic acid1677710: Binding affinity to human KEAP1 Kelch domain (322 to 609 residues) incubated for 60 mins by TR-FRET assayic500.0075uM
3-[3-(difluoromethoxy)-5-fluoro-2-pyridinyl]-3-methoxy-5,5-dimethyl-6-oxocyclohexene-1-carbonitrile1511440: Inhibition of KEAP1 interaction with Nrf2 in human HEK293 cells transfected with ARE-LUC assessed as activation of Nrf2 signaling pathway measured after 18 hrs by steady-Glo luciferase assayec500.0080uM
2-[[4-[carboxymethyl-[(6-chloro-2,3-dihydro-1,4-benzodioxin-7-yl)sulfonyl]amino]naphthalen-1-yl]-[(6-chloro-2,3-dihydro-1,4-benzodioxin-7-yl)sulfonyl]amino]acetic acid1755203: Inhibition of recombinant human KEAP1 Kelch domain (321 to 609 residues) expressed in Escherichia coli BL21 (DE3)pLysS cells interaction with FITC-9mer Nrf2 (unknown origin) incubated for 30 mins under shaking condition by TR-FRET assayic500.0089uM

CTD chemical–gene interactions

193 total (human), top 30 by PubMed support.

ChemicalActions (top 5)PubMed papers
Benzo(a)pyrenedecreases expression, increases expression7
Hydrogen Peroxideincreases expression, affects reaction, affects binding, increases activity, decreases reaction (+3 more)7
sulforaphaneincreases metabolic processing, increases activity, increases expression, increases reaction, increases response to substance (+5 more)6
sodium arseniteincreases reaction, decreases reaction, increases methylation, increases activity, affects localization (+4 more)6
Arsenic Trioxideaffects reaction, increases expression, decreases expression, affects response to substance, decreases reaction (+4 more)6
Cisplatinaffects cotreatment, increases expression, decreases stability, increases response to substance, affects reaction6
(+)-JQ1 compounddecreases expression, increases reaction4
Doxorubicindecreases response to substance, increases expression, increases response to substance, decreases export4
Oxygendecreases reaction, increases degradation, increases expression, decreases expression, affects cotreatment4
decabromobiphenyl etherincreases expression, decreases reaction, decreases expression3
diethyl maleateincreases expression3
2-tert-butylhydroquinonedecreases reaction, increases ubiquitination, increases reaction, increases expression, increases activity (+2 more)3
Resveratrolaffects cotreatment, decreases reaction, increases expression, affects binding, affects expression3
Decitabineincreases expression, affects expression, affects methylation, decreases expression, decreases reaction (+1 more)3
Ascorbic Acidaffects binding, affects cotreatment, decreases expression, decreases reaction, increases abundance (+1 more)3
Curcumindecreases expression, decreases reaction, increases ubiquitination, increases expression3
Etoposideincreases response to substance, affects reaction, increases expression, decreases response to substance3
Plant Extractsdecreases reaction, increases expression, decreases expression, increases chemical synthesis, affects cotreatment3
Quercetindecreases expression, decreases reaction, increases abundance, increases expression, increases degradation (+1 more)3
Valproic Acidaffects expression, decreases expression3
Cadmium Chloridedecreases expression, increases abundance, decreases reaction3
triptolidedecreases expression2
arseniteaffects response to substance, decreases reaction, decreases expression, increases expression, increases reaction (+1 more)2
solanesoldecreases reaction, increases expression, affects binding2
brusatolincreases expression, decreases expression, decreases response to substance, decreases reaction2
andrographolidedecreases expression2
mercuric bromideincreases expression, affects cotreatment2
epigallocatechin gallateincreases expression, increases reaction2
bardoxolone methyldecreases expression, increases expression2
Rosiglitazoneincreases reaction, increases response to substance, increases expression2

ChEMBL screening assays

537 unique, capped per target: 535 binding, 1 functional, 1 admet

Representative assays (with source publication via chembl_document):

Assay IDTypeDescriptionSource paper
CHEMBL2072790BindingInhibition of Keap1 interaction with Nrf2 in human THP1 cells assessed as increase in HO-1 mRNA expression at 100 uM from 3 to 6 hrs by qRT-PCR analysisAnti-inflammatory Effect of a Cell-Penetrating Peptide Targeting the Nrf2/Keap1 Interaction. — ACS Med Chem Lett
CHEMBL2114882FunctionalPubChem BioAssay. Thermal shift assay to identify compound binding to Kelch domain of Keap1 protein Measured in Biochemical System Using RT-PCR - 2119-02_Inhibitor_Dose_CherryPick_Activity. (Class of assay: confirmatory)PubChem BioAssay data set
CHEMBL4140689ADMETActivation of keap1 in human TIG3 cells assessed as Nrf2 nuclear accumulation at 0.6 uM by immuno staining analysis2,3-Dihydro-3β-methoxy Withaferin-A Protects Normal Cells against Stress: Molecular Evidence of Its Potent Cytoprotective Activity. — J Nat Prod

Cellosaurus cell lines

25 cell lines: 23 cancer cell line, 1 transformed cell line, 1 embryonic stem cell

First 10 cell lines (id-ordered, not curated):

CellosaurusNameCategorySex
CVCL_1537NCI-H2172Cancer cell lineFemale
CVCL_4972MHCC97-HCancer cell lineMale
CVCL_6832HCCLM3Cancer cell lineMale
CVCL_7680HCCLM6Cancer cell lineMale
CVCL_A5CUHCCLM9Cancer cell lineMale
CVCL_A9LYMHCC97H-GFP-LC3Cancer cell lineMale
CVCL_B8J9Abcam HCT 116 KEAP1 KOCancer cell lineMale
CVCL_B8Y3Abcam MCF-7 KEAP1 KOCancer cell lineFemale
CVCL_B9LJAbcam A-549 KEAP1 KOCancer cell lineMale
CVCL_D1TCAbcam U-87MG KEAP1 KOCancer cell lineMale

Clinical trials (associated diseases)

2 trials via MONDO — disease-level, not drug-specific.

TrialPhaseStatusTitle
NCT04411290Not specifiedUNKNOWNMalignancy Predictors, Bethesda and TI-RADS Scores Correlated With Final Histopathology in Thyroid Diseases
NCT05774535Not specifiedWITHDRAWNProspective, Observational Study on the Carotid Intima-media Thickness in Patients Undergoing Thyroid Surgery