METTL3

gene
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Also known as Spo8M6AMT-A70

Summary

METTL3 (methyltransferase 3, N6-adenosine-methyltransferase complex catalytic subunit, HGNC:17563) is a protein-coding gene on chromosome 14q11.2, encoding N(6)-adenosine-methyltransferase catalytic subunit METTL3 (Q86U44). The METTL3-METTL14 heterodimer forms a N6-methyltransferase complex that methylates adenosine residues at the N(6) position of some RNAs and regulates various processes such as the circadian clock, differentiation of embryonic and hematopoietic stem cells, cortical neurogenesis,…. It is a selective cancer dependency (DepMap: 79.5% of cell lines).

This gene encodes the 70 kDa subunit of MT-A which is part of N6-adenosine-methyltransferase. This enzyme is involved in the posttranscriptional methylation of internal adenosine residues in eukaryotic mRNAs, forming N6-methyladenosine.

Source: NCBI Gene 56339 — RefSeq curated summary.

At a glance

  • GWAS associations: 1
  • Clinical variants (ClinVar): 53 total
  • Druggable target: yes — 6 molecules with ChEMBL bioactivity
  • Cancer dependency (DepMap): dependent in 79.5% of screened cell lines
  • MANE Select transcript: NM_019852

Identifiers

Gene identifiers

FieldValue
HGNC IDHGNC:17563
Approved symbolMETTL3
Namemethyltransferase 3, N6-adenosine-methyltransferase complex catalytic subunit
Location14q11.2
Locus typegene with protein product
StatusApproved
AliasesSpo8, M6A, MT-A70
Ensembl geneENSG00000165819
Ensembl biotypeprotein_coding
OMIM612472
Entrez56339

Gene structure

Transcript identifiers

Ensembl transcripts: 28 — 16 protein_coding, 5 retained_intron, 4 nonsense_mediated_decay, 3 protein_coding_CDS_not_defined

ENST00000298717, ENST00000367606, ENST00000377543, ENST00000396522, ENST00000536201, ENST00000537163, ENST00000538267, ENST00000539760, ENST00000542054, ENST00000543235, ENST00000544248, ENST00000544500, ENST00000545319, ENST00000545788, ENST00000894032, ENST00000894034, ENST00000894036, ENST00000912042, ENST00000912043, ENST00000912044, ENST00000912045, ENST00000912046, ENST00000912047, ENST00000912048, ENST00000912049, ENST00000967377, ENST00000967378, ENST00000967379

RefSeq mRNA: 1 — MANE Select: NM_019852 NM_019852

CCDS: CCDS32044

Canonical transcript exons

ENST00000298717 — 11 exons

ExonStartEnd
ENSE000012637952149813821498369
ENSE000034746562150172821501903
ENSE000034868072149949221499600
ENSE000035032702151112421511340
ENSE000035239562149902521499137
ENSE000035412792149976421499802
ENSE000035421002150049521500682
ENSE000035820412150317321503577
ENSE000035947892150091321501129
ENSE000036219322150366421503881
ENSE000036719202149930621499371

Expression profiles

Bgee: expression breadth ubiquitous, 297 present calls, max score 97.47.

FANTOM5 (CAGE): breadth ubiquitous, TPM avg 19.1088 / max 165.0067, expressed in 1811 samples.

FANTOM5 promoters (3 alternative TSS)

Promoter IDTPM avgSamples expressed
14218114.31651802
1421804.71381537
1421790.078513

Top tissues by expression

300 total, by Bgee expression score (0-100, higher = more expressed):

TissueAnatomy IDExpression scoreQuality
right uterine tubeUBERON:000130297.47gold quality
right hemisphere of cerebellumUBERON:001489096.17gold quality
cerebellar hemisphereUBERON:000224595.98gold quality
body of uterusUBERON:000985395.96gold quality
cerebellar cortexUBERON:000212995.93gold quality
left lobe of thyroid glandUBERON:000112095.89gold quality
left ovaryUBERON:000211995.84gold quality
right ovaryUBERON:000211895.81gold quality
right lobe of thyroid glandUBERON:000111995.79gold quality
thyroid glandUBERON:000204695.52gold quality
body of pancreasUBERON:000115095.40gold quality
tibial nerveUBERON:000132395.28gold quality
endocervixUBERON:000045895.18gold quality
endothelial cellCL:000011595.16gold quality
cerebellumUBERON:000203795.14gold quality
middle temporal gyrusUBERON:000277195.14gold quality
left uterine tubeUBERON:000130395.09gold quality
ectocervixUBERON:001224994.95gold quality
lymph nodeUBERON:000002994.83gold quality
minor salivary glandUBERON:000183094.82gold quality
right lungUBERON:000216794.72gold quality
spleenUBERON:000210694.70gold quality
ovaryUBERON:000099294.42gold quality
small intestine Peyer’s patchUBERON:000345494.41gold quality
metanephros cortexUBERON:001053394.30gold quality
skin of abdomenUBERON:000141694.24gold quality
granulocyteCL:000009494.17gold quality
saliva-secreting glandUBERON:000104494.16gold quality
mucosa of stomachUBERON:000119994.12gold quality
vaginaUBERON:000099694.09gold quality

Single-cell (SCXA)

Detected in 1 experiment(s), a significant marker in 1.

ExperimentMarker?Max mean expression
E-ANND-3yes8.78

Regulation

Is transcription factor: yes

Downstream targets (CollecTRI)

2 targets.

TargetRegulation
EGFRActivation
WWTR1Activation

Upstream regulators (CollecTRI, top): E2F4

miRNA regulators (miRDB)

6 targeting METTL3, top 30 by miRDB confidence (max_score; target_count = how many genes the miRNA targets in total — lower means more specific):

miRNAMax scoreAvg scoremiRNA target_count
HSA-LET-7C-3P99.9573.422862
HSA-MIR-338-5P99.9272.342951
HSA-MIR-432099.7565.80793
HSA-MIR-33A-3P99.7070.273362
HSA-MIR-124698.5466.21959
HSA-MIR-5579-3P97.0068.811111

Functional genomics

DepMap (CRISPR cell-line fitness): dependent in 79.5% of screened cell lines.

Literature-anchored findings (GeneRIF, showing 40)

  • Study identifies m(6)A methylation sites on many clock gene transcripts and shows that specific inhibition of m(6)A methylation by silencing of the m(6)A methylase Mettl3 is sufficient to elicit circadian period elongation and RNA processing delay. (PMID:24209618)
  • genetic inactivation impaired embryonic stem cells differentiation (PMID:25456834)
  • METTL3 enhances mRNA translation through an interaction with the translation initiation machinery in lung adenocarcinoma cells. (PMID:27117702)
  • Structure of the METTL3-METTL14 complex (PMID:27281194)
  • For the methylation of adenosine in RNA, Mettl3 is the catalytically active subunit, while Mettl14 plays a structural role critical for substrate recognition. (PMID:27373337)
  • The structure reveals the heterodimeric architecture of the complex and donor substrate binding by METTL3. Structure-guided mutagenesis indicates that METTL3 is the catalytic subunit of the complex, whereas METTL14 has a degenerate active site and plays non-catalytic roles in maintaining complex integrity and substrate RNA binding. (PMID:27627798)
  • miR-33a can attenuatenon-small-cell lung carcinoma cells proliferation via targeting to the 3’-untranslated region of METTL3 mRNA. (PMID:27856248)
  • methylation at m6A by METTL3/METTL14 facilitates the methylation of m5C by NSUN2, and vice versa. NSUN2-mediated m5C and METTL3/METTL14-mediated m6A methylation synergistically enhance p21 expression at the translational level (PMID:28247949)
  • loss of METTL3 leads to increased levels of phosphorylated AKT, which contributes to the differentiation-promoting effects of METTL3 depletion. Overall, these results provide a rationale for the therapeutic targeting of METTL3 in myeloid leukemia (PMID:28920958)
  • Study reports the importance of m6A modification in glioma stem-like cells and uncovers METTL3 as a potential molecular target in glioblastoma therapy. (PMID:28991227)
  • Data show that hepatitis B X-interacting protein (HBXIP) modulated Methyltransferase-like 3 (METTL3) by inhibiting miRNA let-7g, which down-regulated the expression of METTL3 by targeting its 3’UTR. (PMID:29174803)
  • data define METTL3 as a regulator of a chromatin-based pathway that is necessary for maintenance of the leukaemic state and identify this enzyme as a potential therapeutic target for acute myeloid leukaemia (PMID:29186125)
  • The role of METTL3 in pancreatic cancer cells drug resistance and radiation resistance.METTL3 was associated with mitogen-activated protein kinase cascades, ubiquitin-dependent process and RNA splicing and regulation of cellular process. (PMID:29345285)
  • METTL3 is soluble and inactive while the catalytic center of METTL14 is degenerated and thus also inactive. In addition, the C-terminal RGG repeats of METTL14 are required for METTL3/14 activity by contributing to RNA substrate binding. (PMID:29348140)
  • METTL3 might inhibit the LPS-induced inflammatory response of human dental pulp cells by regulating alternative splicing of MyD88. (PMID:29502358)
  • An important mechanism for SUMOylation of METTL3 regulating its m6A RNA methyltransferase activity.Lysine 177/211/212/215 are major SUMOylation-sites of METTL3.METTL3 role in tumorigenesis. (PMID:29506078)
  • METTL3 acts as an oncogene in myeloid leukemia MOLM13 cells by upregulating MYC expression. (PMID:29978784)
  • METTL3-eIF3h interaction is required for enhanced translation, formation of densely packed polyribosomes and oncogenic transformation; findings uncover a mechanism of translation control that is based on mRNA looping and identify METTL3-eIF3h as a potential therapeutic target for patients with cancer (PMID:30232453)
  • Findings suggest that METTL3 plays very important oncogenic roles in ovarian carcinoma development and/or aggressiveness by stimulating AXL translation and EMT. (PMID:30249526)
  • we found that METTL3 interacted with viral RNA-dependent RNA polymerase 3D and induced enhanced sumoylation and ubiquitination of the 3D polymerase that boosted viral replication. Taken together, our findings demonstrated that the host m6A modification complex interacts with viral proteins to modulate EV71 replication. (PMID:30364964)
  • METTL3 expression is elevated in bladder cancer.MYC, AFF4, RELA and IKBKB are functionally important target genes of METTL3 in bladder cancer. (PMID:30659266)
  • Results for the first time uncover the role of mA modification in melanoma cell biology. Authors show that METTL3 is upregulated in human melanoma and plays a role in invasion/migration through MMP2. (PMID:30762711)
  • We conclude that METTL3 plays a vital role in many steps of RNA processing and orchestrates successful execution of oncogenic pathways in glioma stem-like cells . (PMID:30781903)
  • Genetic alterations of m6A regulatory genes in clear cell renal cell carcinoma indicate a relationship between the alterations resulting in decreased m6A level and worse clinical characteristics including survival. (PMID:30877265)
  • main writer of N6-methyladenosine modification in germ cell tumors (PMID:30903744)
  • Study demonstrates that oncogenic miR-25 in pancreatic duct epithelial cells can be excessively maturated by cigarette smoke condensate (CSC) via enhanced N(6)-methyladenosine modification that is mediated by NF-kappaB associated protein NKAP. This modification is catalyzed by overexpressed METTL3 due to hypomethylation of the METTL3 promoter also caused by CSC. (PMID:31015415)
  • The m(6)A methyltransferase METTL3 is a critical gene in regulating tumorigeneis of cutaneous squamous cell carcinoma. METTL3 knock down decreased m(6)A levels and the expression of DeltaNp63. (PMID:31153635)
  • Results showed that METTL3 was significantly increased in bladder cancer and correlated with poor prognosis of bladder cancer patients. Moreover, METTL3 could promote cell proliferation both in vitro and tumorigenesis in vivo. (PMID:31228940)
  • Higher METTL3 expression was found in colorectal carcinoma (CRC) metastatic tissues and was associated with a poor prognosis. Further data revealed that METTL3, acting as an oncogene, maintained SOX2 expression through an m6A-IGF2BP2-dependent mechanism in CRC cells, and indicated a potential biomarker panel for prognostic prediction in CRC. (PMID:31230592)
  • Dysregulated N6-methyladenosine methylation writer METTL3 contributes to the proliferation and migration of gastric cancer. (PMID:31232471)
  • In silico genome-wide identification of m6A-associated SNPs as potential functional variants for periodontitis. (PMID:31245852)
  • Data suggest that methyltransferase-like protein 3 (METTL3) silence decreased the m6A methylation and total mRNA level of lymphoid enhancer-binding factor 1 (LEF1), followed by inhibited the activity of Wnt/beta-catenin signaling pathway. (PMID:31253399)
  • miR-4429 prevented gastric cancer progression through targeting METTL3 to inhibit m(6)A-caused stabilization of SEC62 (PMID:31395342)
  • METTL3 inhibits hepatic insulin sensitivity via N6-methylation of Fasn mRNA and promoting fatty acid metabolism. (PMID:31405565)
  • METTL3 was up-regulated in the breast cancer tissue and cells. And, METTL3 could install the methylation of Bcl-2 and promote its translation, thereby regulating the proliferation and apoptosis of breast cancer. (PMID:31454538)
  • our findings provide a totally different mechanism of METTL3 in regulating CRC metastasis. Our study demonstrates that aberrant m6A modification in CRC was responsible for the upregulated METTL3 expression, which marks pri-miR-1246 for further processing (PMID:31492150)
  • METTL3-mediated m(6)A modification of HDGF mRNA promotes gastric cancer progression and has prognostic significance. (PMID:31582403)
  • A Mass Spectrometric Assay of METTL3/METTL14 Methyltransferase Activity. (PMID:31585521)
  • METTL3 is probably involved in pancreatic carcinogenesis (PMID:31606241)
  • our findings indicate the critical role of m6A modification in GC and uncover METTL3/ZMYM1/E-cadherin signaling as a potential therapeutic target in anti-metastatic strategy against GC. (PMID:31607270)

Cross-species orthologs

4 orthologs

OrganismSymbolGene ID
danio_reriomettl3ENSDARG00000012827
mus_musculusMettl3ENSMUSG00000022160
rattus_norvegicusMettl3ENSRNOG00000013111
drosophila_melanogasterMettl3FBGN0039139

Protein

Protein identifiers

N(6)-adenosine-methyltransferase catalytic subunit METTL3Q86U44 (reviewed: Q86U44)

Alternative names: Methyltransferase-like protein 3, N(6)-adenosine-methyltransferase 70 kDa subunit

All UniProt accessions (6): A0A0G2JH39, B4DTN4, B4E349, Q86U44, F5H6D8, H0YFV6

UniProt curated annotations — full annotation on UniProt →

Function. The METTL3-METTL14 heterodimer forms a N6-methyltransferase complex that methylates adenosine residues at the N(6) position of some RNAs and regulates various processes such as the circadian clock, differentiation of embryonic and hematopoietic stem cells, cortical neurogenesis, response to DNA damage, differentiation of T-cells and primary miRNA processing. In the heterodimer formed with METTL14, METTL3 constitutes the catalytic core. N6-methyladenosine (m6A), which takes place at the 5’-[AG]GAC-3’ consensus sites of some mRNAs, plays a role in mRNA stability, processing, translation efficiency and editing. M6A acts as a key regulator of mRNA stability: methylation is completed upon the release of mRNA into the nucleoplasm and promotes mRNA destabilization and degradation. In embryonic stem cells (ESCs), m6A methylation of mRNAs encoding key naive pluripotency-promoting transcripts results in transcript destabilization, promoting differentiation of ESCs. M6A regulates the length of the circadian clock: acts as an early pace-setter in the circadian loop by putting mRNA production on a fast-track for facilitating nuclear processing, thereby providing an early point of control in setting the dynamics of the feedback loop. M6A also regulates circadian regulation of hepatic lipid metabolism. M6A regulates spermatogonial differentiation and meiosis and is essential for male fertility and spermatogenesis. Also required for oogenesis. Involved in the response to DNA damage: in response to ultraviolet irradiation, METTL3 rapidly catalyzes the formation of m6A on poly(A) transcripts at DNA damage sites, leading to the recruitment of POLK to DNA damage sites. M6A is also required for T-cell homeostasis and differentiation: m6A methylation of transcripts of SOCS family members (SOCS1, SOCS3 and CISH) in naive T-cells promotes mRNA destabilization and degradation, promoting T-cell differentiation. Inhibits the type I interferon response by mediating m6A methylation of IFNB. M6A also takes place in other RNA molecules, such as primary miRNA (pri-miRNAs). Mediates m6A methylation of Xist RNA, thereby participating in random X inactivation: m6A methylation of Xist leads to target YTHDC1 reader on Xist and promote transcription repression activity of Xist. M6A also regulates cortical neurogenesis: m6A methylation of transcripts related to transcription factors, neural stem cells, the cell cycle and neuronal differentiation during brain development promotes their destabilization and decay, promoting differentiation of radial glial cells. METTL3 mediates methylation of pri-miRNAs, marking them for recognition and processing by DGCR8. Acts as a positive regulator of mRNA translation independently of the methyltransferase activity: promotes translation by interacting with the translation initiation machinery in the cytoplasm. Its overexpression in a number of cancer cells suggests that it may participate in cancer cell proliferation by promoting mRNA translation. During human coronavirus SARS-CoV-2 infection, adds m6A modifications in SARS-CoV-2 RNA leading to decreased RIGI binding and subsequently dampening the sensing and activation of innate immune responses.

Subunit / interactions. Heterodimer; heterodimerizes with METTL14 to form an antiparallel heterodimer that constitutes an active methyltransferase. Component of the WMM complex, a N6-methyltransferase complex composed of a catalytic subcomplex, named MAC, and of an associated subcomplex, named MACOM. The MAC subcomplex is composed of METTL3 and METTL14. The MACOM subcomplex is composed of WTAP, ZC3H13, CBLL1/HAKAI, VIRMA, and, in some cases of RBM15 (RBM15 or RBM15B). Interacts with NCBP1/CBP80. Interacts with EIF4E. Interacts with EIF3B.

Subcellular location. Nucleus. Nucleus speckle. Cytoplasm.

Tissue specificity. Widely expressed at low level. Expressed in spleen, thymus, prostate, testis, ovary, small intestine, colon and peripheral blood leukocytes.

Post-translational modifications. Sumoylation inhibits the N6-adenosine-methyltransferase activity. Sumoylation does not affect subcellular location or interaction with METTL14. Desumoylated by SENP1.

Activity regulation. Methyltransferase activity is regulated by miRNAs via a sequence pairing mechanism. Methyltransferase activity is inhibited by sumoylation.

Domain organisation. Gate loop 1 and gate loop 2 regions are adjacent to the S-adenosyl-L-homocysteine-binding site and display large conformational changes upon ligand-binding. They may play an important role in adenosine recognition. The interface loop contributes to the heterodimer interaction.

Induction. Overexpressed in a number of cancer tissues, such as lung adenocarcinoma and colon adenocarcinoma.

Similarity. Belongs to the MT-A70-like family.

Isoforms (2)

UniProt IDNamesCanonical?
Q86U44-11yes
Q86U44-22

RefSeq proteins (1): NP_062826* (*=MANE)

Domains & families (InterPro)

IDNameType
IPR007757MT-A70-likeFamily
IPR025848MT-A70Family
IPR029063SAM-dependent_MTases_sfHomologous_superfamily

Pfam: PF05063

Enzyme classification (BRENDA):

  • EC 2.1.1.348 — mRNA m6A methyltransferase (BRENDA: 11 organisms, 24 substrates, 64 inhibitors, 6 Km, 5 kcat entries)

Substrate kinetics (BRENDA)

3 substrates with measured Km, best-characterized 3. Km ranges are aggregated across organisms/conditions.

SubstrateKm (mM)Measurements
ADENINE IN DOUBLE-STRANDED DNA CONTAINING A CYCL0.0006–0.00072
ADENINE IN MRNA0.0002–0.0232
ADENINE IN SINGLE-STRANDED DNA0.0032–0.00772

Catalyzed reactions (Rhea), 1 shown:

  • an adenosine in mRNA + S-adenosyl-L-methionine = an N(6)-methyladenosine in mRNA + S-adenosyl-L-homocysteine + H(+) (RHEA:55584)

UniProt features (87 total): mutagenesis site 25, strand 13, modified residue 9, region of interest 8, helix 7, binding site 5, cross-link 4, sequence conflict 4, splice variant 3, site 2, turn 2, initiator methionine 1, chain 1, short sequence motif 1, compositionally biased region 1, sequence variant 1

Structure

Experimental structures (PDB)

77 structures, top 30 by resolution.

PDBMethodResolution (Å)
5IL2X-RAY DIFFRACTION1.61
7RX7X-RAY DIFFRACTION1.65
5K7MX-RAY DIFFRACTION1.65
5K7WX-RAY DIFFRACTION1.65
5K7UX-RAY DIFFRACTION1.7
5IL1X-RAY DIFFRACTION1.71
9RS1X-RAY DIFFRACTION1.73
9Q8GX-RAY DIFFRACTION1.75
5TEYX-RAY DIFFRACTION1.8
7O09X-RAY DIFFRACTION1.8
7RX6X-RAY DIFFRACTION1.8
7NHIX-RAY DIFFRACTION1.85
7RX8X-RAY DIFFRACTION1.85
9G4WX-RAY DIFFRACTION1.85
9IH5X-RAY DIFFRACTION1.85
5L6DX-RAY DIFFRACTION1.85
7OELX-RAY DIFFRACTION1.86
5IL0X-RAY DIFFRACTION1.88
6Y4GX-RAY DIFFRACTION1.9
7O0LX-RAY DIFFRACTION1.9
7OEKX-RAY DIFFRACTION1.9
5L6EX-RAY DIFFRACTION1.9
7NHVX-RAY DIFFRACTION1.91
9RRYX-RAY DIFFRACTION1.91
9G4SX-RAY DIFFRACTION1.95
9Q89X-RAY DIFFRACTION1.95
9Q8AX-RAY DIFFRACTION1.95
6TTPX-RAY DIFFRACTION2
6TTXX-RAY DIFFRACTION2
7O08X-RAY DIFFRACTION2

Predicted structure (AlphaFold)

ModelpLDDTFraction very-high
AF-Q86U44-F176.710.35

Functional residue map

Curated UniProt residues grouped by drug-discovery relevance — catalytic, ligand-binding, modification, and mutation-validated positions. Source: UniProtKB sequence features.

Catalytic / active sites (2): 438 (interaction with mettl14); 441 (interaction with mettl14)

Ligand- & substrate-binding residues (5): 377–378; 395; 513; 536–539; 549–550

Post-translational modifications (13): 2, 2, 43, 48, 50, 219, 243, 348, 350, 177, 211, 212, 215

Mutagenesis-validated functional residues (25):

PositionPhenotype
2does not affect nuclear localization, interaction with mettl14 or wtap or catalytic activity; when associated with a-43;
43does not affect nuclear localization, interaction with mettl14 or wtap or catalytic activity; when associated with a-2;
48does not affect nuclear localization, interaction with mettl14 or wtap or catalytic activity; when associated with a-2;
50does not affect nuclear localization, interaction with mettl14 or wtap or catalytic activity; when associated with a-2;
177in 4kr; strongly decreased sumoylation; when associated with 211-r–r-215.
211–215abolishes localization to the nucleus.
211–215in 3kr; decreased sumoylation. in 4kr; strongly decreased sumoylation; when associated with r-177.
219does not affect nuclear localization, interaction with mettl14 or wtap or catalytic activity; when associated with a-243
243does not affect nuclear localization, interaction with mettl14 or wtap or catalytic activity; when associated with a-219
294abolishes methyltransferase activity.
326abolishes methyltransferase activity.
348–350does not affect nuclear localization, interaction with mettl14 or wtap or catalytic activity; when associated with a-219
377abolishes methyltransferase activity.
395–398loss of function. abolishes ability to regulate primary mirna processing. does not affect ability to promote mrna transl
395abolishes methyltransferase activity.
406strong reduction in methyltransferase activity.
462–479impaired rna-binding and methyltransferase activities.
475decreased methyltransferase activity.
477decreased methyltransferase activity.
532abolishes methyltransferase activity.
536slight reduction in methyltransferase activity.
538slight reduction in methyltransferase activity.
539abolishes methyltransferase activity.
549slight reduction in methyltransferase activity. strong reduction in methyltransferase activity; when associated with a-5
550slight reduction in methyltransferase activity. strong reduction in methyltransferase activity; when associated with a-5

Function

Pathways and Gene Ontology

Reactome pathways

2 pathways

IDPathway
R-HSA-72203Processing of Capped Intron-Containing Pre-mRNA
R-HSA-8953854Metabolism of RNA

MSigDB gene sets: 331 (showing top): GSE45365_NK_CELL_VS_CD8_TCELL_DN, GOBP_CIRCADIAN_RHYTHM, GOBP_CYTOPLASMIC_TRANSLATION, GOBP_REGULATION_OF_CELL_ACTIVATION, GOBP_HEMATOPOIETIC_PROGENITOR_CELL_DIFFERENTIATION, GOBP_REGULATION_OF_MRNA_CATABOLIC_PROCESS, GOBP_CELLULAR_RESPONSE_TO_UV, GOMF_OXIDOREDUCTASE_ACTIVITY_ACTING_ON_PAIRED_DONORS_WITH_INCORPORATION_OR_REDUCTION_OF_MOLECULAR_OXYGEN, GOBP_RESPONSE_TO_PEPTIDE, GOBP_CELLULAR_RESPONSE_TO_LIGHT_STIMULUS, GOBP_OOGENESIS, GOBP_RESPONSE_TO_TYPE_I_INTERFERON, GOBP_MACROMOLECULE_CATABOLIC_PROCESS, GOBP_NEGATIVE_REGULATION_OF_INNATE_IMMUNE_RESPONSE, GOBP_NEGATIVE_REGULATION_OF_CELL_CELL_ADHESION

GO Biological Process (29): mRNA splicing, via spliceosome (GO:0000398), RNA methylation (GO:0001510), mRNA processing (GO:0006397), DNA damage response (GO:0006974), spermatogenesis (GO:0007283), circadian rhythm (GO:0007623), dosage compensation by inactivation of X chromosome (GO:0009048), mRNA modification (GO:0016556), stem cell population maintenance (GO:0019827), forebrain radial glial cell differentiation (GO:0021861), primary miRNA processing (GO:0031053), cellular response to UV (GO:0034644), gliogenesis (GO:0042063), innate immune response (GO:0045087), regulation of T cell differentiation (GO:0045580), positive regulation of translation (GO:0045727), negative regulation of Notch signaling pathway (GO:0045746), oogenesis (GO:0048477), regulation of meiotic cell cycle (GO:0051445), negative regulation of type I interferon-mediated signaling pathway (GO:0060339), mRNA destabilization (GO:0061157), endothelial to hematopoietic transition (GO:0098508), regulation of hematopoietic stem cell differentiation (GO:1902036), positive regulation of cap-independent translational initiation (GO:1903679), immune system process (GO:0002376), mRNA export from nucleus (GO:0006406), cell differentiation (GO:0030154), methylation (GO:0032259), rhythmic process (GO:0048511)

GO Molecular Function (11): mRNA m(6)A methyltransferase activity (GO:0001734), mRNA binding (GO:0003729), RNA methyltransferase activity (GO:0008173), protein heterodimerization activity (GO:0046982), S-adenosyl-L-methionine binding (GO:1904047), RNA binding (GO:0003723), protein binding (GO:0005515), methyltransferase activity (GO:0008168), S-adenosylmethionine-dependent methyltransferase activity (GO:0008757), transferase activity (GO:0016740), catalytic activity, acting on a nucleic acid (GO:0140640)

GO Cellular Component (9): nucleus (GO:0005634), nucleoplasm (GO:0005654), Golgi apparatus (GO:0005794), cytosol (GO:0005829), nuclear body (GO:0016604), nuclear speck (GO:0016607), RNA N6-methyladenosine methyltransferase complex (GO:0036396), cytoplasm (GO:0005737), oxidoreductase complex (GO:1990204)

Reactome top-level categories

Rollup of top-1 pathways:

CategoryPathways
Metabolism of RNA1

GO top-level categories

Rollup of top GO terms by namespace:

CategoryTerms
cellular anatomical structure3
RNA modification2
mRNA metabolic process2
methyltransferase activity2
catalytic activity2
intracellular membrane-bounded organelle2
cytoplasm2
RNA splicing, via transesterification reactions with bulged adenosine as nucleophile1
mRNA processing1
macromolecule methylation1
RNA processing1
cellular response to stress1
developmental process involved in reproduction1
male gamete generation1
rhythmic process1
sex-chromosome dosage compensation1
heterochromatin formation1
multicellular organismal process1
maintenance of cell number1
forebrain generation of neurons1
radial glial cell differentiation1
miRNA processing1
response to UV1
cellular response to light stimulus1
neurogenesis1
immune response1
defense response to symbiont1
T cell differentiation1
regulation of lymphocyte differentiation1
regulation of T cell activation1
translation1
regulation of translation1
positive regulation of gene expression1
positive regulation of protein metabolic process1
Notch signaling pathway1
regulation of Notch signaling pathway1
negative regulation of signal transduction1
germ cell development1
female gamete generation1
meiotic cell cycle1

Protein interactions and networks

STRING

2399 interactions, top by confidence (×1000):

Protein AProtein BPartner UniProtScore
METTL3VIRMAQ69YN4999
METTL3WTAPQ15007999
METTL3METTL4Q8N3J2999
METTL3ZC3H13Q5T200998
METTL3RBM15Q96T37998
METTL3RBM15BQ8NDT2997
METTL3CBLL1Q75N03996
METTL3METTL14Q9HCE5993
METTL3METTL16Q86W50990
METTL3EIF3HO15372989
METTL3YTHDF3Q7Z739988
METTL3DGCR8Q8WYQ5985
METTL3SMAD2Q15796969
METTL3YTHDC1Q96MU7951
METTL3YTHDC2Q9H6S0942

IntAct

68 interactions, top by confidence:

ABTypeScore
METTL14METTL3psi-mi:“MI:0915”(physical association)0.940
METTL14METTL3psi-mi:“MI:0407”(direct interaction)0.940
METTL3METTL14psi-mi:“MI:0407”(direct interaction)0.940
METTL3METTL14psi-mi:“MI:0914”(association)0.940
BMAL2CLOCKpsi-mi:“MI:0914”(association)0.670
EIF3HMETTL3psi-mi:“MI:0407”(direct interaction)0.560
EIF3HMETTL3psi-mi:“MI:2364”(proximity)0.560
METTL3EIF3Hpsi-mi:“MI:0914”(association)0.560
METTL3WTAPpsi-mi:“MI:0914”(association)0.530
METTL14WTAPpsi-mi:“MI:0914”(association)0.530
NSUN2LIN7Apsi-mi:“MI:0914”(association)0.530
CPSF6DDX39Apsi-mi:“MI:0914”(association)0.480
METTL14psi-mi:“MI:0213”(methylation reaction)0.440
MYCTARS3psi-mi:“MI:0914”(association)0.350
MPP1DKC1psi-mi:“MI:0914”(association)0.350

BioGRID (337): METTL3 (Affinity Capture-RNA), METTL3 (Co-fractionation), METTL3 (Proximity Label-MS), METTL3 (Proximity Label-MS), ROBO2 (Affinity Capture-MS), SEL1L (Affinity Capture-MS), WTAP (Affinity Capture-MS), RBM15B (Affinity Capture-MS), PPHLN1 (Affinity Capture-MS), METTL3 (Affinity Capture-MS), METTL3 (Affinity Capture-MS), METTL3 (Affinity Capture-MS), METTL14 (Affinity Capture-MS), METTL3 (Affinity Capture-MS), METTL3 (Affinity Capture-MS)

ESM2 similar proteins: A0A1D3PCM3, A0A1U8QYZ5, A0A2Z4HPY5, A0A384JRP0, A1D3I1, A2QCJ2, A4RE47, A7EYY7, B0XQ16, C0SGJ2, C1G2I2, C1GP85, C5P635, C8V9Y5, C8VP37, E3QDT3, E9CS37, E9ESM3, G9NNY7, J4W6X9, O42783, O47881, O94069, P08465, P0CQ16, P0CQ17, P12917, P21334, P38092, Q06736, Q10341, Q2QXY1, Q2RAP0, Q2U9G3, Q2URJ0, Q4HVQ9, Q4IQC1, Q4WTK9, Q4X0Z7, Q5AT15

Diamond homologs: A4IFD8, F1R777, O82486, Q3UIK4, Q5R5N4, Q5ZK35, Q66KJ9, Q6EU10, Q6NU56, Q6NZ22, Q86U44, Q8C3P7, Q94AI4, Q9HCE5, Q9VCE6, Q9VLP7, P41833, Q2HVD6

SIGNOR signaling

16 interactions.

AEffectBMechanism
MAPK3“up-regulates quantity by stabilization”METTL3phosphorylation
MAPK1“up-regulates quantity by stabilization”METTL3phosphorylation
ERK1/2“up-regulates quantity by stabilization”METTL3phosphorylation
METTL3“up-regulates quantity by expression”EGFR“transcriptional regulation”
METTL3“up-regulates quantity by expression”WWTR1“transcriptional regulation”
Gbeta“up-regulates quantity by stabilization”METTL3phosphorylation
TBK1“up-regulates activity”METTL3phosphorylation
ATM“up-regulates activity”METTL3phosphorylation
METTL3“up-regulates quantity by stabilization”USP13“post transcriptional regulation”
METTL3“up-regulates quantity by stabilization”UCK2“post transcriptional regulation”

Enriched among interaction partners

Reactome pathways and GO biological processes over-represented among this gene’s 67 IntAct physical interaction partners (hypergeometric vs the genome-wide background, BH-FDR, gene-set size 15–500, ranked by fold). A functional readout of the neighbourhood — distinct from this gene’s own memberships above, and biased toward well-studied / hub proteins, so read it as themes rather than proof.

GO biological processes:

GO termPartnersFoldFDR
circadian regulation of gene expression518.9×4e-03

Disease & clinical

Clinical variants and AI predictions

ClinVar

53 variants total. Per-class counts are floors (≥ shown; pagination cap):

ClassificationCount (floor)
Pathogenic0
Likely pathogenic0
Uncertain significance38
Likely benign0
Benign0

Top pathogenic / likely-pathogenic (0)

SpliceAI

1786 predictions. Top by Δscore:

VariantEffectΔscore
14:21499506:T:TAdonor_gain1.0000
14:21499507:C:Adonor_gain1.0000
14:21499596:CATAC:Cacceptor_gain1.0000
14:21499598:TAC:Tacceptor_gain1.0000
14:21499599:AC:Aacceptor_gain1.0000
14:21499599:ACC:Aacceptor_loss1.0000
14:21499600:CC:Cacceptor_gain1.0000
14:21499601:C:CAacceptor_loss1.0000
14:21500528:T:TAdonor_gain1.0000
14:21500681:CA:Cacceptor_gain1.0000
14:21500683:C:CCacceptor_gain1.0000
14:21501059:CCA:Cacceptor_gain1.0000
14:21501060:CA:Cacceptor_gain1.0000
14:21501061:A:Cacceptor_gain1.0000
14:21501071:A:Cacceptor_gain1.0000
14:21501125:TTCGT:Tacceptor_gain1.0000
14:21501126:TCGT:Tacceptor_gain1.0000
14:21501127:CGT:Cacceptor_gain1.0000
14:21501127:CGTC:Cacceptor_gain1.0000
14:21501130:C:CCacceptor_gain1.0000
14:21503167:CCCTA:Cdonor_loss1.0000
14:21503168:CCTAC:Cdonor_loss1.0000
14:21503169:CTAC:Cdonor_loss1.0000
14:21503170:TAC:Tdonor_loss1.0000
14:21503172:CCTT:Cdonor_gain1.0000
14:21511138:T:TAdonor_gain1.0000
14:21511145:T:TAdonor_gain1.0000
14:21499019:CCTTA:Cdonor_loss0.9900
14:21499020:CTTA:Cdonor_loss0.9900
14:21499022:TACC:Tdonor_loss0.9900

AlphaMissense

3807 scored. Top likely-pathogenic:

VariantProtein changeam_pathogenicity
14:21498358:C:AG548V1.000
14:21498358:C:TG548E1.000
14:21498359:C:GG548R1.000
14:21498359:C:TG548R1.000
14:21499026:A:GW544R1.000
14:21499026:A:TW544R1.000
14:21499039:A:CN539K1.000
14:21499039:A:TN539K1.000
14:21499052:C:TG535E1.000
14:21499053:C:GG535R1.000
14:21499053:C:TG535R1.000
14:21499054:A:CF534L1.000
14:21499054:A:TF534L1.000
14:21499056:A:GF534L1.000
14:21499056:A:TF534I1.000
14:21499085:A:GL524P1.000
14:21499115:G:CP514R1.000
14:21499115:G:TP514Q1.000
14:21499116:G:AP514S1.000
14:21499117:T:AK513N1.000
14:21499117:T:GK513N1.000
14:21499118:T:AK513I1.000
14:21499119:T:CK513E1.000
14:21499119:T:GK513Q1.000
14:21499123:A:CS511R1.000
14:21499123:A:TS511R1.000
14:21499124:C:AS511I1.000
14:21499124:C:TS511N1.000
14:21499125:T:GS511R1.000
14:21499311:C:GA505P1.000

dbSNP variants (sampled 300 via entrez): RS1000165463 (14:21505465 A>G), RS1000174585 (14:21510483 A>T), RS1000219668 (14:21505790 C>T), RS1000373945 (14:21498838 C>T), RS1000723956 (14:21498492 G>A,C), RS1000769783 (14:21508665 C>A,T), RS1000781812 (14:21511591 T>C), RS1001119760 (14:21506508 C>G,T), RS1001130481 (14:21511423 G>C), RS1001280877 (14:21507930 AAG>A), RS1001311875 (14:21507501 C>T), RS1001732096 (14:21512720 C>T), RS1001775762 (14:21500765 C>T), RS1002119876 (14:21503431 G>A), RS1002184492 (14:21512976 C>A)

Disease associations

OMIM: gene MIM:612472 | disease phenotypes:

GenCC curated gene-disease

Mondo (0):

Orphanet (0):

HPO phenotypes

0 total (0 of 0 shown, HPO-id order):

GWAS associations

1 associations (top):

StudyTraitp-value
GCST002749_4Response to Homoharringtonine (cytotoxicity)9.000000e-07

EFO canonical traits (1, from GWAS)

EFO IDTrait name
EFO:0006952cytotoxicity measurement

Drugs & pharmacology

Drug and pharmacology data

Is drug target: yes

ChEMBL targets (4): CHEMBL4106140 (PROTEIN COMPLEX), CHEMBL4739695 (SINGLE PROTEIN), CHEMBL6195524 (PROTEIN-PROTEIN INTERACTION), CHEMBL6195539 (PROTEIN-PROTEIN INTERACTION)

Molecules with ChEMBL bioactivity

6 molecules (phase ≥1), by development phase (incl. off-target/promiscuous compounds). Patent mentions across the top 20 by phase: 149,602 (via chembl_molecule»patent_compound — counts attach to the compound, not the gene–compound relationship, so off-target/promiscuous molecules can dominate).

MoleculeNamePhasePatents
CHEMBL461101ELTROMBOPAG4602
CHEMBL204656ELVITEGRAVIR42,944
CHEMBL509MECLOFENAMIC ACID445,809
CHEMBL50QUERCETIN374,559
CHEMBL1214186SINEFUNGIN22,165
CHEMBL151LUTEOLIN223,523

PharmGKB: 1 entry (VIP=true, CPIC=false)

PharmGKB variants

1 variants.

VariantGenesLevelScore#Clin annotsDrugs
rs1139130METTL30.000

GtoPdb / IUPHAR curated pharmacology

(IUPHAR/BPS Guide to Pharmacology — expert-curated)

Target class: enzyme — 2.1.1.- Methyltransferases

Most potent curated ligand interactions (3 total), top 3:

LigandActionAffinityParameter
UZH2Inhibition8.3pIC50
STC-15Inhibition8.21pIC50
STM2457Inhibition7.77pIC50

Binding affinities (BindingDB)

135 measured of 135 human assays (135 total across all organisms); most potent 50 below. Values come from heterogeneous assays and are not directly comparable.

LigandMeasureValuePatent
US20250214997, Compound E-57IC500.1 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
5-[1-[[2-[(4,4-dimethylpiperidin-1-yl)methyl]-1H-indol-6-yl]methyl]triazol-4-yl]-N,N-dimethylpyridin-3-amineIC500.12 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
N-[(3-fluoro-1-bicyclo[1.1.1]pentanyl)methyl]-1-[2-[[4-[5-(3-fluoropyrrolidin-1-yl)-3-pyridinyl]triazol-1-yl]methyl]imidazo[1,2-a]pyridin-6-yl]methanamineIC500.16 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
N-[(3-fluoro-1-bicyclo[1.1.1]pentanyl)methyl]-1-[2-[[4-(5-pyrrolidin-1-yl-3-pyridinyl)triazol-1-yl]methyl]imidazo[1,2-a]pyridin-6-yl]methanamineIC500.16 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
US20250214997, Compound E-140IC500.17 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
US20250214997, Compound E-87IC500.19 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
US20250214997, Compound E-121IC500.2 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
US20250214997, Compound E-84IC500.24 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
US20250214997, Compound E-111IC500.24 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
US20250214997, Compound E-51IC500.25 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
US20250214997, Compound E-101IC500.25 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
US20250214997, Compound E-50IC500.29 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
US20250214997, Compound E-105IC500.29 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
US20250214997, Compound E-119IC500.29 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
8-bromo-2-[(1-methylpiperidin-4-yl)amino]-4-(4-phenylmethoxyanilino)-6H-pyrido[4,3-d]pyrimidin-5-one;hydrochlorideIC500.3 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
N-[(3-fluoro-1-bicyclo[1.1.1]pentanyl)methyl]-1-[2-[[4-(5-pyrrol-1-yl-3-pyridinyl)triazol-1-yl]methyl]imidazo[1,2-a]pyridin-6-yl]methanamineIC500.31 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
US20250214997, Compound E-56IC500.32 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
N-[[6-[[(3-fluoro-1-bicyclo[1.1.1]pentanyl)methylamino]methyl]imidazo[1,2-a]pyridin-2-yl]methyl]-5-pyrrolidin-1-ylpyridine-3-carboxamideIC500.33 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
2-[[4-[5-(2,5-dihydropyrrol-1-yl)-3-pyridinyl]triazol-1-yl]methyl]-6-[(4,4-dimethylpiperidin-1-yl)methyl]imidazo[1,2-a]pyridineIC500.36 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
US20250214997, Compound E-49IC500.37 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
6-(aziridin-1-yl)-4-[1-[[2-[(4,4-dimethylpiperidin-1-yl)methyl]-1H-indol-6-yl]methyl]triazol-4-yl]-1H-indazoleIC500.4 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
6-[(4,4-dimethylpiperidin-1-yl)methyl]-2-[[4-(5-pyrrolidin-1-yl-3-pyridinyl)triazol-1-yl]methyl]imidazo[1,2-a]pyridineIC500.4 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
1-[2-[[4-[5-(3,3-difluoropyrrolidin-1-yl)-3-pyridinyl]triazol-1-yl]methyl]imidazo[1,2-a]pyridin-6-yl]-N-[(3-fluoro-1-bicyclo[1.1.1]pentanyl)methyl]methanamineIC500.41 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
US20250214997, Compound E-106IC500.48 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
6-[[4-[5-(azetidin-1-yl)-3-pyridinyl]triazol-1-yl]methyl]-2-[(4,4-dimethylpiperidin-1-yl)methyl]-1H-indoleIC500.5 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
US20250214997, Compound E-79IC500.54 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
8-bromo-4-(4-phenoxyanilino)-2-[[1-(2,2,2-trifluoroethyl)piperidin-4-yl]amino]-6H-pyrido[4,3-d]pyrimidin-5-oneIC500.56 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
US20250214997, Compound E-59IC500.59 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
8-bromo-4-(3-methyl-4-phenoxyanilino)-2-[(1-methylpiperidin-4-yl)amino]-6H-pyrido[4,3-d]pyrimidin-5-one;hydrochlorideIC500.61 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
8-bromo-4-[4-(3,5-difluorophenoxy)anilino]-2-[(1-methylpiperidin-4-yl)amino]-6H-pyrido[4,3-d]pyrimidin-5-one;hydrochlorideIC500.69 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
1-cyclobutyl-N-[[2-[[4-[5-(2,5-dihydropyrrol-1-yl)-3-pyridinyl]triazol-1-yl]methyl]imidazo[1,2-a]pyridin-6-yl]methyl]methanamineIC500.74 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
8-bromo-2-[(1-methylpiperidin-4-yl)amino]-4-(4-phenylmethoxyanilino)-6H-pyrido[4,3-d]pyrimidin-5-oneIC500.75 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
US20250214997, Compound E-39IC500.75 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
US20250214997, Compound E-143IC500.77 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
8-bromo-4-[3-fluoro-4-(3-fluorophenoxy)anilino]-2-[(1-methylpiperidin-4-yl)amino]-6H-pyrido[4,3-d]pyrimidin-5-one;hydrochlorideIC500.8 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
8-chloro-4-(4-cyclohexyloxyanilino)-2-[(1-methylpiperidin-4-yl)amino]-6H-pyrido[4,3-d]pyrimidin-5-oneIC500.9 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
1-cyclobutyl-N-[[2-[[4-(5-pyrrolidin-1-yl-3-pyridinyl)triazol-1-yl]methyl]imidazo[1,2-a]pyridin-6-yl]methyl]methanamineIC501.02 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
5-[1-[[6-[(4,4-dimethylpiperidin-1-yl)methyl]imidazo[1,2-a]pyridin-2-yl]methyl]triazol-4-yl]-N,N-dimethylpyridin-3-amineIC501.06 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
5-(dimethylamino)-N-[[2-[(4,4-dimethylpiperidin-1-yl)methyl]-1H-indol-6-yl]methyl]pyridine-3-carboxamideIC501.09 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
2-[[(3S)-pyrrolidin-3-yl]amino]-4-[4-(trifluoromethyl)anilino]-6H-pyrido[4,3-d]pyrimidin-5-oneIC501.17 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
US20250214997, Compound E-58IC501.18 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
8-bromo-4-[4-(4-chlorophenoxy)anilino]-2-[(1-methylpiperidin-4-yl)amino]-6H-pyrido[4,3-d]pyrimidin-5-one;hydrochlorideIC501.21 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
4-[1-[[2-(6-azaspiro[3.4]octan-6-ylmethyl)-1H-indol-6-yl]methyl]triazol-4-yl]-6-methylsulfanyl-1H-indazoleIC501.23 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
1-cyclobutyl-N-[[2-[[4-(6-iodo-1H-indazol-4-yl)triazol-1-yl]methyl]imidazo[1,2-a]pyridin-6-yl]methyl]methanamineIC501.24 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
US20250214997, Compound E-112IC501.25 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
4-[1-[[2-(6-azaspiro[3.4]octan-6-ylmethyl)-1H-indol-6-yl]methyl]triazol-4-yl]-6-bromo-1H-indazoleIC501.37 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
8-bromo-4-[4-(4-methylphenoxy)anilino]-2-[(1-methylpiperidin-4-yl)amino]-6H-pyrido[4,3-d]pyrimidin-5-oneIC501.38 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
US20250214997, Compound E-60IC501.39 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
8-bromo-4-[4-(3-fluorophenoxy)-3-methylanilino]-2-[(1-methylpiperidin-4-yl)amino]-6H-pyrido[4,3-d]pyrimidin-5-one;hydrochlorideIC501.4 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF
US20250214997, Compound E-43IC501.4 nMUS-20250214997: METTL3 INHIBITOR COMPOUND, AND PHARMACEUTICAL COMPOSITION AND USE THEREOF

ChEMBL bioactivities

1054 potent at pChembl≥5 of 1076 total, top 50 by pChembl (potency: 10 = 0.1 nM, 6 = 1 µM).

pChemblTypeValueUnitMolecule
9.00IC501nMCHEMBL6171683
9.00IC501nMCHEMBL6161197
9.00IC501nMCHEMBL6174416
9.00IC501nMCHEMBL6162072
8.85Kd1.4nMCHEMBL5291234
8.70IC502nMCHEMBL6149057
8.70IC502nMCHEMBL6169196
8.70IC502nMCHEMBL6164629
8.70IC502nMCHEMBL6145109
8.70IC502nMCHEMBL6167143
8.70IC502nMCHEMBL6162084
8.70IC502nMCHEMBL6170438
8.70IC502nMCHEMBL6174400
8.70IC502nMCHEMBL6175655
8.70IC502nMCHEMBL6173476
8.70IC502nMCHEMBL6161072
8.52IC503nMCHEMBL6167275
8.52IC503nMCHEMBL6169100
8.52IC503nMCHEMBL6147720
8.52IC503nMCHEMBL6148888
8.52IC503nMCHEMBL6163833
8.52IC503nMCHEMBL6161072
8.44IC503.63nMCHEMBL5396973
8.42IC503.84nMCHEMBL5076382
8.41IC503.85nMCHEMBL5077332
8.40IC504nMCHEMBL6171694
8.40IC504nMCHEMBL6143768
8.40IC504nMCHEMBL6170155
8.40IC504nMCHEMBL6165737
8.40IC504nMCHEMBL6165967
8.40IC504nMCHEMBL5075067
8.37IC504.23nMCHEMBL5438009
8.36IC504.35nMCHEMBL5405105
8.35IC504.44nMCHEMBL5425488
8.32Kd4.79nMELVITEGRAVIR
8.32Kd4.8nMELVITEGRAVIR
8.30IC505nMCHEMBL5077332
8.30IC505nMCHEMBL6152127
8.30IC505nMCHEMBL6166439
8.30IC505nMCHEMBL6161960
8.30IC505nMCHEMBL6161072
8.25IC505.67nMCHEMBL5412324
8.24IC505.76nMCHEMBL5423596
8.24IC505.8nMCHEMBL5433144
8.24IC505.776nMCHEMBL5433927
8.23IC505.92nMCHEMBL5407802
8.22IC506.009nMCHEMBL5404800
8.22IC506nMCHEMBL6162091
8.22IC506nMCHEMBL6147934
8.22IC506nMCHEMBL6151936

PubChem BioAssay actives

144 with measured affinity, of 267 total; 50 most potent distinct compounds. Largely complementary to BindingDB; screening values are coarse (µM, 4 dp), so sub-nM hits tie at the floor.

CompoundAssayTypeValueUnit
N-[[6-[(cyclohexylmethylamino)methyl]imidazo[1,2-a]pyridin-2-yl]methyl]-4-oxopyrido[1,2-a]pyrimidine-2-carboxamide1939568: Binding affinity to METTL3 (unknown origin) by SPR methodkd0.0014uM
1-cyclobutyl-N-[[2-[[4-(6-methoxy-1H-indazol-4-yl)triazol-1-yl]methyl]imidazo[1,2-a]pyridin-6-yl]methyl]methanamine1986899: Inhibition of full-length His-tagged METTL3/full length FLAG-tagged METTL14 (unknown origin) expressed in baculovirus expression system using synthetic RNA 5’P-uacacucgaucuggacuaaagcugcuc-3’ and SAM as substrate preincubated for 10 mins followed by substrate addition and measured after 60 mins by RF/MS analysisic500.0036uM
N-[[2-[[4-(6-chloro-1H-indazol-4-yl)triazol-1-yl]methyl]imidazo[1,2-a]pyridin-6-yl]methyl]-1-cyclobutylmethanamine1986899: Inhibition of full-length His-tagged METTL3/full length FLAG-tagged METTL14 (unknown origin) expressed in baculovirus expression system using synthetic RNA 5’P-uacacucgaucuggacuaaagcugcuc-3’ and SAM as substrate preincubated for 10 mins followed by substrate addition and measured after 60 mins by RF/MS analysisic500.0043uM
6-[(3-chloro-2-fluorophenyl)methyl]-1-[(2S)-1-hydroxy-3-methylbutan-2-yl]-7-methoxy-4-oxoquinoline-3-carboxylic acid1986930: Binding affinity to METTL3 (unknown origin) assessed as dissociation constant by SPR analysiskd0.0048uM
4-[4-[(4,4-dimethylpiperidin-1-yl)methyl]-2,5-difluorophenyl]-9-[6-(methylamino)pyrimidin-4-yl]-1,4,9-triazaspiro[5.5]undecan-2-one1829989: Inhibition of recombinant METLL3 (unknown origin) expressed in baculovirus infected Sf9 cells assessed as decrease in N6-methyladenosine level in oligonucleotide substrate by TR-FRET assayic500.0050uM
(1R,2S,3R,5R)-3-(4-amino-5-bromopyrrolo[2,3-d]pyrimidin-7-yl)-5-[2-[[(3-fluoro-1-bicyclo[1.1.1]pentanyl)methylamino]methyl]-1H-indol-6-yl]cyclopentane-1,2-diol1986899: Inhibition of full-length His-tagged METTL3/full length FLAG-tagged METTL14 (unknown origin) expressed in baculovirus expression system using synthetic RNA 5’P-uacacucgaucuggacuaaagcugcuc-3’ and SAM as substrate preincubated for 10 mins followed by substrate addition and measured after 60 mins by RF/MS analysisic500.0060uM
N-[(1-methoxycyclobutyl)methyl]-1-[2-[[4-(6-methoxy-1H-indazol-4-yl)triazol-1-yl]methyl]imidazo[1,2-a]pyridin-6-yl]methanamine1986899: Inhibition of full-length His-tagged METTL3/full length FLAG-tagged METTL14 (unknown origin) expressed in baculovirus expression system using synthetic RNA 5’P-uacacucgaucuggacuaaagcugcuc-3’ and SAM as substrate preincubated for 10 mins followed by substrate addition and measured after 60 mins by RF/MS analysisic500.0061uM
N-[[6-[[(3-fluoro-1-bicyclo[1.1.1]pentanyl)methylamino]methyl]imidazo[1,2-a]pyridin-2-yl]methyl]-4-oxopyrido[1,2-a]pyrimidine-2-carboxamide1986899: Inhibition of full-length His-tagged METTL3/full length FLAG-tagged METTL14 (unknown origin) expressed in baculovirus expression system using synthetic RNA 5’P-uacacucgaucuggacuaaagcugcuc-3’ and SAM as substrate preincubated for 10 mins followed by substrate addition and measured after 60 mins by RF/MS analysisic500.0061uM
1-cyclobutyl-N-[[2-[[4-(5-methoxy-1H-indazol-4-yl)triazol-1-yl]methyl]imidazo[1,2-a]pyridin-6-yl]methyl]methanamine1986899: Inhibition of full-length His-tagged METTL3/full length FLAG-tagged METTL14 (unknown origin) expressed in baculovirus expression system using synthetic RNA 5’P-uacacucgaucuggacuaaagcugcuc-3’ and SAM as substrate preincubated for 10 mins followed by substrate addition and measured after 60 mins by RF/MS analysisic500.0061uM
N-[[2-[[(3-fluoro-1-bicyclo[1.1.1]pentanyl)methylamino]methyl]-1H-indol-6-yl]methyl]-5-oxo-[1,3]thiazolo[3,2-a]pyrimidine-7-carboxamide1986899: Inhibition of full-length His-tagged METTL3/full length FLAG-tagged METTL14 (unknown origin) expressed in baculovirus expression system using synthetic RNA 5’P-uacacucgaucuggacuaaagcugcuc-3’ and SAM as substrate preincubated for 10 mins followed by substrate addition and measured after 60 mins by RF/MS analysisic500.0061uM
N-[[2-[(cyclobutylmethylamino)methyl]-1H-indol-6-yl]methyl]-4-oxopyrido[1,2-a]pyrimidine-2-carboxamide1986899: Inhibition of full-length His-tagged METTL3/full length FLAG-tagged METTL14 (unknown origin) expressed in baculovirus expression system using synthetic RNA 5’P-uacacucgaucuggacuaaagcugcuc-3’ and SAM as substrate preincubated for 10 mins followed by substrate addition and measured after 60 mins by RF/MS analysisic500.0061uM
N-[[2-[(cyclobutylmethylamino)methyl]-5-fluoro-1H-indol-6-yl]methyl]-4-oxopyrido[1,2-a]pyrimidine-2-carboxamide1986899: Inhibition of full-length His-tagged METTL3/full length FLAG-tagged METTL14 (unknown origin) expressed in baculovirus expression system using synthetic RNA 5’P-uacacucgaucuggacuaaagcugcuc-3’ and SAM as substrate preincubated for 10 mins followed by substrate addition and measured after 60 mins by RF/MS analysisic500.0061uM
N-[[2-[[(3-fluoro-1-bicyclo[1.1.1]pentanyl)methylamino]methyl]-1H-indol-6-yl]methyl]-4-oxopyrido[1,2-a]pyrimidine-2-carboxamide1986899: Inhibition of full-length His-tagged METTL3/full length FLAG-tagged METTL14 (unknown origin) expressed in baculovirus expression system using synthetic RNA 5’P-uacacucgaucuggacuaaagcugcuc-3’ and SAM as substrate preincubated for 10 mins followed by substrate addition and measured after 60 mins by RF/MS analysisic500.0061uM
N-[[2-[(3,3-diethylazetidin-1-yl)methyl]-1H-indol-6-yl]methyl]-4-oxopyrido[1,2-a]pyrimidine-2-carboxamide1986899: Inhibition of full-length His-tagged METTL3/full length FLAG-tagged METTL14 (unknown origin) expressed in baculovirus expression system using synthetic RNA 5’P-uacacucgaucuggacuaaagcugcuc-3’ and SAM as substrate preincubated for 10 mins followed by substrate addition and measured after 60 mins by RF/MS analysisic500.0061uM
N-[[2-[[1-(3-fluoro-1-bicyclo[1.1.1]pentanyl)ethylamino]methyl]-1H-indol-6-yl]methyl]-4-oxopyrido[1,2-a]pyrimidine-2-carboxamide1986899: Inhibition of full-length His-tagged METTL3/full length FLAG-tagged METTL14 (unknown origin) expressed in baculovirus expression system using synthetic RNA 5’P-uacacucgaucuggacuaaagcugcuc-3’ and SAM as substrate preincubated for 10 mins followed by substrate addition and measured after 60 mins by RF/MS analysisic500.0061uM
N-(1-bicyclo[1.1.1]pentanylmethyl)-1-[2-[[4-(6-methoxyimidazo[1,5-a]pyridin-8-yl)triazol-1-yl]methyl]imidazo[1,2-a]pyridin-6-yl]methanamine1986899: Inhibition of full-length His-tagged METTL3/full length FLAG-tagged METTL14 (unknown origin) expressed in baculovirus expression system using synthetic RNA 5’P-uacacucgaucuggacuaaagcugcuc-3’ and SAM as substrate preincubated for 10 mins followed by substrate addition and measured after 60 mins by RF/MS analysisic500.0061uM
1-cyclobutyl-N-[[2-[[4-(6-methoxyimidazo[1,5-a]pyridin-8-yl)triazol-1-yl]methyl]imidazo[1,2-a]pyridin-6-yl]methyl]methanamine1986899: Inhibition of full-length His-tagged METTL3/full length FLAG-tagged METTL14 (unknown origin) expressed in baculovirus expression system using synthetic RNA 5’P-uacacucgaucuggacuaaagcugcuc-3’ and SAM as substrate preincubated for 10 mins followed by substrate addition and measured after 60 mins by RF/MS analysisic500.0061uM
N-[(3-fluoro-1-bicyclo[1.1.1]pentanyl)methyl]-1-[2-[[4-(6-methoxyimidazo[1,5-a]pyridin-8-yl)triazol-1-yl]methyl]imidazo[1,2-a]pyridin-6-yl]methanamine1986899: Inhibition of full-length His-tagged METTL3/full length FLAG-tagged METTL14 (unknown origin) expressed in baculovirus expression system using synthetic RNA 5’P-uacacucgaucuggacuaaagcugcuc-3’ and SAM as substrate preincubated for 10 mins followed by substrate addition and measured after 60 mins by RF/MS analysisic500.0061uM
(1R,2S,3R,5R)-3-(4-amino-5-bromopyrrolo[2,3-d]pyrimidin-7-yl)-5-[2-(6-azaspiro[3.4]octan-6-ylmethyl)-1H-indol-6-yl]cyclopentane-1,2-diol1986899: Inhibition of full-length His-tagged METTL3/full length FLAG-tagged METTL14 (unknown origin) expressed in baculovirus expression system using synthetic RNA 5’P-uacacucgaucuggacuaaagcugcuc-3’ and SAM as substrate preincubated for 10 mins followed by substrate addition and measured after 60 mins by RF/MS analysisic500.0061uM
(1R,2S,3R,5R)-3-(4-amino-5-chloropyrrolo[2,3-d]pyrimidin-7-yl)-5-[2-[[(3-fluoro-1-bicyclo[1.1.1]pentanyl)methylamino]methyl]-1H-indol-6-yl]cyclopentane-1,2-diol1986899: Inhibition of full-length His-tagged METTL3/full length FLAG-tagged METTL14 (unknown origin) expressed in baculovirus expression system using synthetic RNA 5’P-uacacucgaucuggacuaaagcugcuc-3’ and SAM as substrate preincubated for 10 mins followed by substrate addition and measured after 60 mins by RF/MS analysisic500.0061uM
(1R,2S,3R,5R)-3-(4-aminopyrrolo[2,3-d]pyrimidin-7-yl)-5-[2-(6-azaspiro[3.4]octan-6-ylmethyl)-1H-indol-6-yl]cyclopentane-1,2-diol1986899: Inhibition of full-length His-tagged METTL3/full length FLAG-tagged METTL14 (unknown origin) expressed in baculovirus expression system using synthetic RNA 5’P-uacacucgaucuggacuaaagcugcuc-3’ and SAM as substrate preincubated for 10 mins followed by substrate addition and measured after 60 mins by RF/MS analysisic500.0062uM
2-[[4-(6-bromo-1H-indazol-4-yl)triazol-1-yl]methyl]-6-[(4,4-dimethylpiperidin-1-yl)methyl]imidazo[1,2-a]pyridine1986899: Inhibition of full-length His-tagged METTL3/full length FLAG-tagged METTL14 (unknown origin) expressed in baculovirus expression system using synthetic RNA 5’P-uacacucgaucuggacuaaagcugcuc-3’ and SAM as substrate preincubated for 10 mins followed by substrate addition and measured after 60 mins by RF/MS analysisic500.0079uM
7-[2-[5-[4-amino-5-(1-methylpyrazol-3-yl)pyrrolo[2,3-d]pyrimidin-7-yl]-3-pyridinyl]ethyl]-N-methylquinolin-2-amine1986933: Inhibition of METTL3/METTL14 (unknown origin) using biotinylated RNA (UCUGGACUAAA-biotin) and 3H-SAM as substrate preincubated for 5 mins followed by substrate addition and measured after 30 mins by scintillation counter analysisic500.0100uM
7-[2-[5-[4-amino-5-(1-methylpyrazol-3-yl)pyrrolo[2,3-d]pyrimidin-7-yl]-3-pyridinyl]ethyl]-2-ethyl-N-(2-methoxyethyl)-N-methylquinolin-4-amine1986933: Inhibition of METTL3/METTL14 (unknown origin) using biotinylated RNA (UCUGGACUAAA-biotin) and 3H-SAM as substrate preincubated for 5 mins followed by substrate addition and measured after 30 mins by scintillation counter analysisic500.0100uM
7-[2-[5-[4-amino-5-(1-methylpyrazol-3-yl)pyrrolo[2,3-d]pyrimidin-7-yl]-3-pyridinyl]ethyl]-2-ethyl-N-(2-methoxyethyl)quinolin-4-amine1986933: Inhibition of METTL3/METTL14 (unknown origin) using biotinylated RNA (UCUGGACUAAA-biotin) and 3H-SAM as substrate preincubated for 5 mins followed by substrate addition and measured after 30 mins by scintillation counter analysisic500.0100uM
7-[[5-[4-amino-5-[(2S)-oxolan-2-yl]pyrrolo[2,3-d]pyrimidin-7-yl]-3-pyridinyl]methoxy]-N-methylquinolin-2-amine1986933: Inhibition of METTL3/METTL14 (unknown origin) using biotinylated RNA (UCUGGACUAAA-biotin) and 3H-SAM as substrate preincubated for 5 mins followed by substrate addition and measured after 30 mins by scintillation counter analysisic500.0100uM
(2R,3R,4S,5R)-5-[4-amino-5-(5-methylfuran-2-yl)pyrrolo[2,3-d]pyrimidin-7-yl]-4-fluoro-2-[[2-(methylamino)quinolin-7-yl]oxymethyl]oxolan-3-ol1768407: Inhibition of METTL3 (unknown origin) using 3’ biotinylated RNA (UCUGGACUAAA) and 3H-SAM as a substrate preincubated for 5 mins followed by substrate addition incubated for 30 mins by scintillation counting methodic500.0100uM
(2R,3R,4S,5R)-5-[4-amino-5-(1-methylpyrazol-3-yl)pyrrolo[2,3-d]pyrimidin-7-yl]-2-[[2-(azetidin-1-yl)quinolin-7-yl]oxymethyl]-4-fluorooxolan-3-ol1986933: Inhibition of METTL3/METTL14 (unknown origin) using biotinylated RNA (UCUGGACUAAA-biotin) and 3H-SAM as substrate preincubated for 5 mins followed by substrate addition and measured after 30 mins by scintillation counter analysisic500.0100uM
(2R,3S,5R)-5-[4-amino-5-(1-methylpyrazol-3-yl)pyrrolo[2,3-d]pyrimidin-7-yl]-2-[[2-(azetidin-1-yl)quinolin-7-yl]oxymethyl]oxolan-3-ol1986933: Inhibition of METTL3/METTL14 (unknown origin) using biotinylated RNA (UCUGGACUAAA-biotin) and 3H-SAM as substrate preincubated for 5 mins followed by substrate addition and measured after 30 mins by scintillation counter analysisic500.0100uM
(2R,3R,4S,5R)-5-(4-amino-5-pyrazol-1-ylpyrrolo[2,3-d]pyrimidin-7-yl)-2-[[2-(azetidin-1-yl)quinolin-7-yl]oxymethyl]-4-fluorooxolan-3-ol1768407: Inhibition of METTL3 (unknown origin) using 3’ biotinylated RNA (UCUGGACUAAA) and 3H-SAM as a substrate preincubated for 5 mins followed by substrate addition incubated for 30 mins by scintillation counting methodic500.0100uM
(2R,3R,4S,5R)-5-[4-amino-5-(4-methyl-1,3-oxazol-2-yl)pyrrolo[2,3-d]pyrimidin-7-yl]-2-[[2-(azetidin-1-yl)quinolin-7-yl]oxymethyl]-4-fluorooxolan-3-ol1768407: Inhibition of METTL3 (unknown origin) using 3’ biotinylated RNA (UCUGGACUAAA) and 3H-SAM as a substrate preincubated for 5 mins followed by substrate addition incubated for 30 mins by scintillation counting methodic500.0100uM
(2R,3R,4S,5R)-5-[4-amino-5-(1-methylpyrazol-3-yl)pyrrolo[2,3-d]pyrimidin-7-yl]-2-[[2-(cyclobutylamino)quinolin-7-yl]oxymethyl]-4-fluorooxolan-3-ol1986933: Inhibition of METTL3/METTL14 (unknown origin) using biotinylated RNA (UCUGGACUAAA-biotin) and 3H-SAM as substrate preincubated for 5 mins followed by substrate addition and measured after 30 mins by scintillation counter analysisic500.0100uM
(2R,3R,4S,5R)-2-(4-amino-5-cyclopentylpyrrolo[2,3-d]pyrimidin-7-yl)-5-[[2-(ethylamino)quinolin-7-yl]oxymethyl]oxolane-3,4-diol1768407: Inhibition of METTL3 (unknown origin) using 3’ biotinylated RNA (UCUGGACUAAA) and 3H-SAM as a substrate preincubated for 5 mins followed by substrate addition incubated for 30 mins by scintillation counting methodic500.0100uM
(2R,3R,4S,5R)-2-[4-amino-5-(1-methylpyrazol-3-yl)pyrrolo[2,3-d]pyrimidin-7-yl]-5-[[2-(methylamino)quinolin-7-yl]oxymethyl]oxolane-3,4-diol1768407: Inhibition of METTL3 (unknown origin) using 3’ biotinylated RNA (UCUGGACUAAA) and 3H-SAM as a substrate preincubated for 5 mins followed by substrate addition incubated for 30 mins by scintillation counting methodic500.0100uM
(2R,3S,5R)-5-(4-amino-5-pyrazol-1-ylpyrrolo[2,3-d]pyrimidin-7-yl)-2-[[2-(azetidin-1-yl)quinolin-7-yl]oxymethyl]oxolan-3-ol1986933: Inhibition of METTL3/METTL14 (unknown origin) using biotinylated RNA (UCUGGACUAAA-biotin) and 3H-SAM as substrate preincubated for 5 mins followed by substrate addition and measured after 30 mins by scintillation counter analysisic500.0100uM
(2R,3R,4S,5R)-5-[4-amino-5-[(2S)-oxolan-2-yl]pyrrolo[2,3-d]pyrimidin-7-yl]-2-[[2-(azetidin-1-yl)quinolin-7-yl]oxymethyl]-4-fluorooxolan-3-ol1768407: Inhibition of METTL3 (unknown origin) using 3’ biotinylated RNA (UCUGGACUAAA) and 3H-SAM as a substrate preincubated for 5 mins followed by substrate addition incubated for 30 mins by scintillation counting methodic500.0100uM
(2R,3R,4S,5R)-5-[4-amino-5-[(2S)-oxolan-2-yl]pyrrolo[2,3-d]pyrimidin-7-yl]-4-fluoro-2-[[2-(methylamino)quinolin-7-yl]oxymethyl]oxolan-3-ol1986933: Inhibition of METTL3/METTL14 (unknown origin) using biotinylated RNA (UCUGGACUAAA-biotin) and 3H-SAM as substrate preincubated for 5 mins followed by substrate addition and measured after 30 mins by scintillation counter analysisic500.0100uM
(2S,3R,4S,5R)-5-[4-amino-5-(1-methylpyrazol-3-yl)pyrrolo[2,3-d]pyrimidin-7-yl]-N-[2-(azetidin-1-yl)quinolin-7-yl]-4-fluoro-3-hydroxyoxolane-2-carboxamide1986933: Inhibition of METTL3/METTL14 (unknown origin) using biotinylated RNA (UCUGGACUAAA-biotin) and 3H-SAM as substrate preincubated for 5 mins followed by substrate addition and measured after 30 mins by scintillation counter analysisic500.0100uM
N-[[(3R)-1-[6-(benzylamino)pyrimidin-4-yl]-3-hydroxypiperidin-3-yl]methyl]-4-[(4,4-dimethylpiperidin-1-yl)methyl]-2-hydroxybenzamide1939567: Inhibition of METTL3 in human MOLM-13 cells assessed as reduction in m6A methylation levelic500.0170uM
9-(2-chloro-7H-pyrrolo[2,3-d]pyrimidin-4-yl)-4-[4-[(4,4-dimethylpiperidin-1-yl)methyl]phenyl]-1,4,9-triazaspiro[5.5]undecan-2-one1829989: Inhibition of recombinant METLL3 (unknown origin) expressed in baculovirus infected Sf9 cells assessed as decrease in N6-methyladenosine level in oligonucleotide substrate by TR-FRET assayic500.0240uM
9-[6-(benzylamino)pyrimidin-4-yl]-4-[4-[(4,4-dimethylpiperidin-1-yl)methyl]phenyl]-1,4,9-triazaspiro[5.5]undecan-2-one1829989: Inhibition of recombinant METLL3 (unknown origin) expressed in baculovirus infected Sf9 cells assessed as decrease in N6-methyladenosine level in oligonucleotide substrate by TR-FRET assayic500.0260uM
4-[4-[(4,4-dimethylpiperidin-1-yl)methyl]-3-fluorophenyl]-9-[6-(methylamino)pyrimidin-4-yl]-1,4,9-triazaspiro[5.5]undecan-2-one1829989: Inhibition of recombinant METLL3 (unknown origin) expressed in baculovirus infected Sf9 cells assessed as decrease in N6-methyladenosine level in oligonucleotide substrate by TR-FRET assayic500.0320uM
9-[6-(benzylamino)pyrimidin-4-yl]-4-[6-[(4,4-dimethylpiperidin-1-yl)methyl]-3-pyridinyl]-1,4,9-triazaspiro[5.5]undecan-2-one1829989: Inhibition of recombinant METLL3 (unknown origin) expressed in baculovirus infected Sf9 cells assessed as decrease in N6-methyladenosine level in oligonucleotide substrate by TR-FRET assayic500.0370uM
4-[4-[(4,4-dimethylpiperidin-1-yl)methyl]-2-fluorophenyl]-9-[6-(methylamino)pyrimidin-4-yl]-1,4,9-triazaspiro[5.5]undecan-2-one1829989: Inhibition of recombinant METLL3 (unknown origin) expressed in baculovirus infected Sf9 cells assessed as decrease in N6-methyladenosine level in oligonucleotide substrate by TR-FRET assayic500.0380uM
4-[4-[(4,4-dimethylpiperidin-1-yl)methyl]phenyl]-9-(7H-pyrrolo[2,3-d]pyrimidin-4-yl)-1,4,9-triazaspiro[5.5]undecan-2-one1829989: Inhibition of recombinant METLL3 (unknown origin) expressed in baculovirus infected Sf9 cells assessed as decrease in N6-methyladenosine level in oligonucleotide substrate by TR-FRET assayic500.0610uM
9-[6-(cyclopropylamino)pyrimidin-4-yl]-4-[4-[(4,4-dimethylpiperidin-1-yl)methyl]phenyl]-1,4,9-triazaspiro[5.5]undecan-2-one1829989: Inhibition of recombinant METLL3 (unknown origin) expressed in baculovirus infected Sf9 cells assessed as decrease in N6-methyladenosine level in oligonucleotide substrate by TR-FRET assayic500.0840uM
4-[4-[(4,4-dimethylpiperidin-1-yl)methyl]phenyl]-9-[6-(methylamino)pyrimidin-4-yl]-1,4,9-triazaspiro[5.5]undecan-2-one1829989: Inhibition of recombinant METLL3 (unknown origin) expressed in baculovirus infected Sf9 cells assessed as decrease in N6-methyladenosine level in oligonucleotide substrate by TR-FRET assayic500.0890uM
N-benzyl-6-[4-[6-[(4,4-dimethylpiperidin-1-yl)methyl]-3-pyridinyl]-1-oxa-4,9-diazaspiro[5.5]undecan-9-yl]pyrimidin-4-amine1829989: Inhibition of recombinant METLL3 (unknown origin) expressed in baculovirus infected Sf9 cells assessed as decrease in N6-methyladenosine level in oligonucleotide substrate by TR-FRET assayic500.2800uM
4-[4-[(4,4-dimethylpiperidin-1-yl)methyl]phenyl]-9-[6-(propan-2-ylamino)pyrimidin-4-yl]-1,4,9-triazaspiro[5.5]undecan-2-one1829989: Inhibition of recombinant METLL3 (unknown origin) expressed in baculovirus infected Sf9 cells assessed as decrease in N6-methyladenosine level in oligonucleotide substrate by TR-FRET assayic500.3300uM
8-[4-[(4,4-dimethylpiperidin-1-yl)methyl]phenyl]-2-[6-(methylamino)pyrimidin-4-yl]-2,5,8-triazaspiro[3.5]nonan-6-one1829989: Inhibition of recombinant METLL3 (unknown origin) expressed in baculovirus infected Sf9 cells assessed as decrease in N6-methyladenosine level in oligonucleotide substrate by TR-FRET assayic500.4400uM

CTD chemical–gene interactions

49 total (human), top 30 by PubMed support.

ChemicalActions (top 5)PubMed papers
sodium arsenitedecreases expression, increases methylation, increases reaction, affects expression, decreases reaction (+7 more)13
Benzo(a)pyrenedecreases methylation, increases expression, affects cotreatment, increases reaction3
Valproic Acidaffects cotreatment, increases expression, decreases expression3
bisphenol Adecreases expression2
N-methyladenosineaffects abundance, affects expression, affects methylation, increases abundance, increases methylation2
Arsenic Trioxidedecreases expression2
Arsenicaffects methylation, increases expression2
Tobacco Smoke Pollutionaffects reaction, increases expression, affects binding, increases reaction, decreases expression2
Cadmium Chloridedecreases expression, increases abundance, increases expression2
FR900359affects phosphorylation1
quinoneincreases expression1
sodium arsenatedecreases expression, increases reaction, increases expression1
trichostatin Aaffects expression1
beta-lapachonedecreases expression1
3-deazaadenosinedecreases reaction, increases expression1
2,4,6-trichlorophenolincreases expression1
thallium acetatedecreases reaction, increases expression, affects reaction1
coumarinincreases phosphorylation1
cadmium sulfateincreases expression1
beta-methylcholineaffects expression1
fumonisin B1increases expression1
di-n-butylphosphoric acidaffects expression1
chromium hexavalent ionincreases expression, increases response to substance1
ICG 001decreases expression1
statticdecreases reaction, increases expression, increases reaction, increases phosphorylation1
jinfukangincreases expression1
Sunitinibincreases expression1
Leflunomidedecreases expression1
Acetaminophendecreases expression1
Acetylcysteinedecreases reaction, increases expression1

ChEMBL screening assays

141 unique, capped per target: 139 binding, 2 functional

Representative assays (with source publication via chembl_document):

Assay IDTypeDescriptionSource paper
CHEMBL4005637BindingInhibition of METTL3-14 (unknown origin) at 10 uM using biotin-labeled peptide as substrate and [3H]-SAM measured after 1 hr by scintillation proximity assayDiscovery of Potent and Selective Inhibitors for G9a-Like Protein (GLP) Lysine Methyltransferase. — J Med Chem
CHEMBL5723072FunctionalAffinity Biochemical interaction: (METTL3/14 RF/MS methyltransferase assay) EUB0001097a METTL3Affinity Biochemical Literature for EUbOPEN Chemogenomic Library

Clinical trials (associated diseases)

0 trials via MONDO — disease-level, not drug-specific.

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.